EXSC1600 Exam 2 Study Guide
Soft Tissue Response to Injury
3 phases
Continuum
Overlap
No definitive beginning or end
Clinician goals
Recognize what physiological events are taking place during the sequence
Don't interfere with the process
Phases of healing
Inflammatory response
24-36 hours
Begins immediatly after injrury
Chemicals are released causing a cascade of effects
Most critical to healing process
Repair (fibroblastic repair phase)
4-6 weeks
maturation-remodeling
Can take months – years
3 layers of lacerations
Skin
Fat
Muscle
4 stages of healing lacerations
Bleeding
Blood clot
Holds and closes wound
Macrophages
Fibroblast
Neutrophils
Clotted
Scabs
Phase 1
Chemical mediators released
Histamines
Leukotrienes & prostaglandins
Cytokines
Vascular Response
Vasodilation
Increased cell permeability
Exudate
Edema
Amount of edema/swelling is directly related to extent of vessel damage
Secondary injury due to hypoxia
Cell death due to lack of oxygen
Cell permeability is the ability to allow things to go in and out of cells
Physiological Goals of phase 1
Localize the extent of area injured
Rid area of waste products
PREPARE FOR PHASE 2
5 cardinal Sx
Redness
Swelling
Pain
Increased temperature
Loss of function
Phase 2
Scar tissue formation
Replaces torn tissue sites
General timeline: acute phase is about 3-6 weeks
Begins when area has been cleaned by removal of cellular debris
Fibroplasia: SCAR TISSUE formation
Nutrients delivered for tissue regeneration
Increase blood flow by movement: ROM
Granulation tissue fills gaps in tissue
Fibroblast begin producing COLLAGEN
Randomly deposited throughout forming scar
Occurs at about the 6th or 7th day
Strength of the scar tissue increases
Length of phase 2 is dependent on:
Extent of the injury
Proper immediate care and progression of care
Type of tissue
Fine line between enough stress and too much!
Phase 3
Scar tissue begins strengthening and realigns to tissue fibers
Maximal efficiency of scar tissue
Appropriate stress and strain is required to produce this effect
Long term process (6 months to years)
Scar tissue is RARELY as strong as the normal uninjured tissue
Factors that impede healing
Extent of the injury
Edema/hemorrhage
Separates tissue, inhibits NM control, hypoxia
Muscle spasm
Atophy
Immobilization?
Infection
Health, age, nutrition
Soft tissue healing
Cartilage
Articular cartilage
Ligament
Instability
Muscle
Active contraction
Lengthy rehab – paitience
Tendon
Adhesions
Bone healing
Immobilization
Remodeling
“wolff’s law” - adaptations to stress & strain
As a muscle becomes stressed and strained, it will grow and adapt
Stress Fx: d/c activity, eliminate factors
Therapeutic Injury Management
POLICE (RICE)
Rest, Ice, compression, elevation
NSAIDs
Nonsteroidal Antiinflammatory drugs
Indications
Minimize pain and swelling
Contraindictions
Allergies, adverse GI tract rxn, prolonged clotting times, alcohol use, risk of adverse CV events
Ibuprofen
Aspirin
Superficial thermal agents
Cryotherapy – therapeutic use of cold
Vasoconstriction
Metabolic rate goes down
Free nerve ending excitability goes down
Nerve conduction velocity goes down
Initial 48-72 hours: ICE
CBAN – four stages of icing
Cold
Burn
Ache
Numb
Ice, Heat
Use ice in the IPE
Use heat in the FPE and MPE
Therapeutic modalities
Modality – something you use in addition
Therapeutic ultrasound
K tape
cupping
Mobilization
Exercise and rehabilitation
Nutrition and hydration
Mental health
Sleep
Protect
Cast, sling, brace
Optimal Loading
Progress to mechanically loading tissue to promote healing
Early safe functional loading = early recovery
Ice
45-60 minutes initially, then 20-30 minutes every 1.5-2 hours
Compression
Mechanically reducing the space available for swelling and hemorrhage
Secondary Hypoxic Injury – not controlling the swelling of an injury
Elevation
Eliminate the effects of gravity on blood and edema pooling
Cryotherapy techniques
Ice massage
Ice water immersion: 50-60 degrees F
Ice pack vs. Ice bag
Spray coolant
Thermal Agents
Thermotherapy – therapeutic use of heat
Vasodilation
Increased metabolic rate
Increased extensibility of collagen
Capillary permeability
Analgesic
Decreases joint stiffness
Relieves muscle spasm
Can be used to decrease swelling/edema
Electrical Stimulating Currents
Reduces pain, M reeducation and strengthening, retard M atrophy
Iontophoresis
IASTM – instrument assisted Soft Tissue Massage
Recognize what physiological events are taking place during the sequence
Don't interfere with the process
Create an environment conductive to healing
Knock knees is valgus
Bowlegged is verus
Week 7
Foot and ankle
26 bones in each foot
7 tarsals
1-5 metatarsals
Long bones of the foot
14 phalanges
Form determines function & function determines form
Free floating bones are sesamoid bones
2 in your feet just for leverage
Pes Planus
“low arch”
Decreased efficiency
Associated with excessive pronation
Pes Cavus
“High Arch”
Tight Heel Cord
Associated with excessive supination
Decreased shock absorption
Kinetic chain is the direct effect on distal and prostomial segments
Everything in your body is connected
Algia – injury to nerve
Metatarsalgia
Orthotics – can help correct biomechanical problem
Using things to help fix the arch of your foot
Plantar fascia is a super think band of tissue that goes from your heel to your toes
Assists in maintaining stability
Lbracing long. Arch
Good for shock absorption
Flat feet and high arches can causeplantar fasciitis
Increased tension
Tight heel cord
Pain at medial heel
Sharp pain wt-bearing in morning or after rest
Pain with dorsiflexion
Lasts around 8-12 weeks
Orthodics and arch tape can help heal
Stress fracture
Running, jumping, marching
If they’re not treated tehy can lead to full fractures
Usually at the 2nd metatarsal
Point tender, throbbing, aching
Relives with rest
Underlying casuses are high arch/low arch
2-4 weeks complete rest
Gradual introduction back into activity
Bunions: hallux valgus
You can heal bunions by changnig your shoe selection
Surgery is more of a “last resort” for bunions
Tibia – medial malleolus – big bone in lower leg – inside of leg
Fibula – lateral malleolus – small bone in lower leg – outside of leg
Talus – what goes in and out: allowing dorsi/plantarflexion
Calcaneus – heel
Your distal tibiofiblar is between tibia and fibula
Talocrural is your “ankle joint”
3 main lateral ligaments
Anterior talofibular ligament (ATFL)
Calcaneofibular ligament (CFL)
Posterior talofibular ligament (PTFL)
Limits inversion
Lateral ankle sprains
5 cardinal Sx – redness, ecchymosis(redness), pain, swelling, loss of range of motion
POLICE/RICE crutches
Same cardinal symptoms and process as lateral sprains for medial sprains
Knee
4 ligaments in the knee
MCL (Medial Collateral Ligament - INSIDE)
Prevents anterior movement of tibia on femur
ACL - Anterior crucial ligament
PCL (posterior cruciate ligament)
LCL (lateral collateral ligament)
Valgus stress/force
Direct blow to lateral knee
Taking a blow from the outside of the knee pushing inwards
Grade one tear is less than 50% tear on a ligament
Grade two tear is more than 50% tear on a ligament, but not a complete tear
Grade three is a complete tear on a ligament
Ways to tear ACL
Foot planted and pivot in a different direction
Deceleration/landing
Rotary force
Hyperextenxion of knee
More than 50% of ACL tears are non-contact
Females are at higher risk of tearing their ACL
More likely to tear ACL if a family member has
Sx of sprain
A pop or a snap
Often pain immediately, then it subsides
+ Lachmans test
Athlete senses that “something is wrong”
PCL sprain
Mx – posterior translation of the tibia
Hyperflexion in the knee and plantarflexion in the foot
Rx – rarely operative
IT band (iliotibial band ITB) - lateral thigh muscle and tendon
Outside of leg
Patellar tendon
Patella is a sesmoid bone
More than 15 bursas in the knee
IT band
Helps with ABduction
Can get tight
Repition and overuse can cause injury
Sx – pain at the lateral femoral epicondyle
Rx – foam rolling, hip strengthening
Patella Tendon
Excessive jumping, kicking, or repitition can cause injury or strain
Sx – pain at the patella tendon
Rx – no sudden explosive movements
Thigh, Hip and Pelvis
Femur
Femoral Head – large top of bone
Femoral neck – part connecting
Pelvis is made up of three parts
Ilium – Right/left side
Pubis – Bottom part
Sacrum – middle at the base of the spine
Femoral head is sunk into the pelvis to help with weight bearing
Sacrum is formed of 5 different bones fused together
6 degrees of freedom in the hip
Abduction and Adduction
Flexion extension
Internal & external rotation
Flexibility
Hip flexors
Hamstrings
Groin
IT band
Stability
Gluteus medius
“core” muscles
Biomechanics
Leg length discrepancy
Muscular imbalances
Maintain strength
Squats, lunges
Leg Injuries
Quad contusions
Sx – discoloration, Loss of ROM, M. Weakness
Rx – Early is key:
POLICE/RICE 48-72 hours
Position/wrap knee in fully flexed position
NO heat or deep massage for minimum 72 hours
Hip point
Contusion to Iliac crest
Site of numerous m. attachments
No protection
Mx – direct blow
Sx – very dibilitating, painful, ROM decreased, difficulty walking
Rx – protective padding for RTP
Spine
Bones in the spine are called vertebrae
7 cervical
12 thoracic – where your ribs are
5 lumbar
Sacrum and coccyx make up tailbone
Displacement in spine allows certain movements to not put strain on your spine
Axial load
In between the vertebrae there are intervertebral discs
Made of fibrocartilage
Nucleus pulposus – middle of disc
Annulus fibrosus – outside of disc
Spine anatomy - Made of nerve roots and a spinal cord
Change in spincal cord can result in poor posture
Kyphosis
Forward head
Swayback
Flatback
Lordosis
Scoliosis
Strength & flexibility weakness or imbalances
Tight hamstrings
Tight hip flexors
Weak abdominals
Burners are when you stretch out the region between your neck and shoulders
Stingers are neural injuries to your neck area
Stretch or compression of the brachial plexus
Sx – burning, tingling, numbenss down arm. Transient and unilateral
Before RTP, the athlete needs to have full muslce strength
Sx Free
Acute Cervical injuries
Mx – axial load, All other neck movements
Sx – paresthesia or no sensation, weak AROM or Paralysis, is the injury unilateral or bilateral
Rx – Spinal motion restriction(manual), ABCs, Call EMS.
Chronic lower back pain
Mx
Poor mechanics – lifting
Malalignments
Muscle imbalance/weakness
Poor posture
Obesity
Congenital abnormalities
Rx
Read chapter 20, it explains it
Disk Herniation
Etiology
Trauma or degeneration
Ususally L4-L5
Mx
Forward bending and twisting
Sx
Radiation neuro sx
Pain with flexion
Manage with rest
Stabilization ex!, surgery possibly
“spondy” - spondylolisthesis
Degeneration, defect or stress fx fo the vertebrae
Common with repeated extension (L5-S1)
Most of the trunk flexion occurs in the lumbar region of the spine
75% between L5-S1
Review – Soft Tissue Response to Injury
3 stages of healing
Continuum, overlap, no definitive beginning or ending
Clinican goals
Recognize what physiological events are taking place
Dont interfere
Create an environment conducive to healing
“pain is good” - letting you know your injuried
Phase 1 – inflammatory response
Phase 2 – repair
Phase 3 – maturation-remodeling
5 signs of inflammation
Pain
Ecchymosis (swelling)
Redness
Loss or ROM
Increase in temperature
Amount of edema/swelling is directly related to extent of vessel damage
Secondary injury due to hypoxia
Hypoxia is cell death due to low oxygen
Phagocytosis is histamines trying to heal damaged tissues
Fibroplasia: scar tissue formation
Scar tissue is almost never as strong as the original tissue
Broken bones – heal by a spongy-bone covering the broken area
Wolffs law is adaptations to stress & strain
POLICE is the process of decreasing swelling
RICE is the same – rest, ice, compression, elevation
Cryotherapy is the theraputic use of cold
Decrease pain
Decrease muscle spasms
CBAN – Cold, Burn, Ache, Numb
Heat does the opposite
Thermotherapy is the theraputic use of heat
Basic goals of rehab
First aid and control swelling
Decrease pain
Restore ROM
Talus – 4th bone in foot ot know
Outside of ankle is lateral maleolis
Muscles going on the outside of the malleoli correspond to eversion
In front of the malleoli corresponds to dorsiflexion
Behind of malleoli corresponds to plantarflexion
Pes planus is low arch
Pes cavus is high arch
Over-pronation is when your feet lean into the midline causing your ankles to cave inwards
Over-supination is the exact opposite
Stance phase is 60% of total gait
Orthodics helps correct biomechanical issues in the foot
Medial malleolus is on the tibia?
Lateral malleolus is on the fibula?
70-80% of all ankle sprains are lateral movements
Functional testing is “return to play” stuff
Muscle in the front by the tibia is the tibialis anterior
Decelerates your foot
Patella is a sesamoid bone
Sesamoid means floating
MCL has a blood supply so it can heal on its own