Unit 04 Pt3
Unit Overview
Unit 04: Intracellular Trafficking & Coats
Primary Reference: Molecular Biology of the Cell, 6th edition, by Alberts B, Johnson A, Lewis J, et al. (Garland Science; 2022)
Covers topics from Chapter 13 (Pages 749-807)
Outline of Topics
SNAREs Mediate Membrane Fusion
Interaction of SNAREs and the need for disassembly before reuse
Membrane Fusion Proteins from Viruses for Cell Entry
Transport Mechanisms from the Endoplasmic Reticulum (ER) through the Golgi Apparatus
COPII-coated Transport Vesicles
Proper folding and Assembly for ER Exit
Vesicular Tubular Clusters Role
Retrieval Pathway using Sorting Signals
Selective retention of proteins in their functional compartments
Structure of the Golgi Apparatus as a sorting station
SNAREs: Mechanism & Structure
SNAREs (Soluble NSF Attachment Protein Receptors) facilitate membrane fusion:
Transport vesicles must be tethered and then fuse with target membranes.
Require close proximity (approx. 1.5 nm) and exclusion of water for fusion.
v-SNAREs (vesicular SNAREs) and t-SNAREs (target SNAREs):
35 different organelle-specific SNAREs
v-SNAREs: Made of a single polypeptide on vesicles
t-SNAREs: Composed of 2-3 proteins on target membranes
These proteins have helical domains that form a trans-SNARE complex during interaction.
Fusion Process
Energy from helices interaction drives membrane fusion:
Lipids flow between membranes to create a fusion stalk.
Completion of fusion leads to the formation of a new bilayer.
Regulatory aspects of fusion include:
Delayed fusion during regulated exocytosis requiring signaling processes.
Neurotransmitter Release
In neurons, neurotransmitter release occurs via synaptic vesicle fusion:
SNAREs play a critical role in vesicle fusion at synaptic junctions.
Toxins from bacteria like tetanus/botulism can interfere with SNARE function, leading to motor reflex issues.
SNARE Reuse and Regulation
Post-fusion disassembly of SNARE complexes requires:
ATP, NSF protein, and accessory proteins.
In Drosophila, NSF mutants (like comatose) affect vesicle transport.
Specificity in vesicle transport relies on correct Rabs and SNAREs, regulation mechanisms are still under investigation.
Cellular Fusion Reactions
Fusion is required in several biological processes:
Examples include fertilization and muscle fiber formation.
Viruses utilize fusion mechanisms:
Enveloped viruses (e.g., HIV) fuse with cell membranes after receptor binding.
Influenza enters via receptor-mediated endocytosis, utilizing low pH for fusion activation.
Transport from ER to Golgi
Golgi apparatus functions:
Major site for carbohydrate synthesis and enzymatic modifications (protein/lipid glycosylation).
Sorts cargo from the ER for further processing.
ER exit is selective and involves COPII-coated vesicles targeting correctly folded proteins.
Mechanisms of Exit from the ER
Proteins sorted for exit must have recognized exit signals:
Selectivity based on COPII coat components and folding state of proteins.
Specific receptors exist for key proteins like Factor V, related to blood clotting.
Proteins lacking exit signals can result in slow leakage to the Golgi.
Protein Quality Control
Misfolded/incompletely-folded proteins are retained in the ER, with chaperones masking exit signals.
Degradation pathways for defective proteins are critical:
Quality control ensures functional throughput and can impact disease (e.g., CFTR involvement in cystic fibrosis).
Vesicular Tubular Clusters
Homotypic fusion of transport vesicles from the ER leads to the formation of tubular clusters.
Requires matching v-SNAREs and t-SNAREs.
Retrograde Transport Mechanism
Vesicular tubular clusters facilitate COPI-coated vesicles for retrieval of ER proteins:
This process is known as retrograde transport, performing continual maturation as they approach the Golgi.
Sorting Signals in Retrieval to the ER
ER retrieval involves specific signal sequences binding to COPI coats:
C-terminal signals (e.g., KKXX for membrane proteins, KDEL for soluble proteins) manage packaging for retrograde transport.
Golgi Apparatus Structure
Comprised of stacked cisternae organized next to the nucleus:
Structures can be disrupted by microtubule alterations, showcasing dependence on cytoskeleton.
Golgi Functional Regions
The Golgi exhibits 'sided-ness' with distinct entry (cis) and exit (trans) faces for molecular processing:
Transport through the Golgi is associated with modification events crucial for function.
Research Questions
How do vesicles fuse with target membranes?
Compare intracellular vesicle transport and viral entry.
Discuss mechanisms returning ER-bound proteins to the ER, including coated vesicle interactions.
Distinguish Golgi complex regions and their unique functions.