Milk Synthesis & Energy for Lactation
Milk Composition
- Complete food; only low in Fe & vitamin D
- Gross composition (cow): 87.3\%\,H_2O, 3.9\%\,fat, 3.2\%\,protein (casein + whey), 4.6\%\,lactose, 0.7\%\,ash
- Isosmotic with blood → balance between diffusible ions (proteins) & lactose; ↑protein ⇄ ↓lactose
- Fat content varies widely across species; reflects neonatal needs (insulation, energy, brain growth, etc.)
Precursors Delivered by Blood
- Glucose, AA, NEFA/TG, acetate, butyrate, minerals, water
- Determined by blood flow rate & nutrient concentration (diet, tissue mobilisation)
Secretory Cell Organelles & Roles
- Cell membrane (GLUT-1 glucose transport)
- Cytosol (glycolysis, FA chain elongation)
- RER (protein translation; de-novo FA)
- Golgi (casein micelle assembly, lactose synthesis, packaging)
- Mitochondria (ATP)
- Secretion routes: exocytosis (protein/lactose), milk-fat globule budding, transcytosis (Ig), paracellular (pathological)
Lactose Synthesis
- Glucose → G6P \xrightarrow{phosphoglucomutase} G1P
- G1P+UTP \rightarrow UDP!\text{-}glucose
- Epimerase → UDP-galactose
- Golgi lactose synthase (UDP-galactose + glucose) → lactose
- Glucose trapped as G6P; three fates: lactose (galactose), glycolysis/ATP, pentose-P (NADPH)
Protein Synthesis
- AA from blood/muscle → ribosome (mRNA blueprint) → RER
- Caseins assembled in Golgi → micelles
- Whey proteins (α-lactalbumin, β-lactoglobulin) largely synthesised similarly
Milk Fat Synthesis
- Precursors: acetate, \beta-hydroxybutyrate, glucose (glycerol)
- Triglycerides formed in ER; globules bud from apical membrane
- Readily altered by diet lipid profile
- Limited glucose pool: ↑lactose ↓fat/protein (and vice-versa)
- NADPH from pentose-P & isocitrate pathways drives FA synthesis
Energy Requirements (MJ ME)
- To produce 1\,\text{kg} milksolids (MS): 62!\text{–}!65\,\text{MJ ME} (breed dependent)
- Daily ME example: maintenance 64 + 2\,\text{kg MS} \times 65 – weight loss 0.25\,\text{kg}\times32 ≈ 186\,\text{MJ ME}
Maintenance Energy
- Basal heat: 0.32\times BW^{0.75} (MJ d⁻¹)
- Major costs: ion transport (30–40 %), protein turnover (9–12 %), organ work (heart 9–11 %, kidney 6–7 %, resp. 6–7 %)
- Activity increments: standing ↑10 kJ/d, walking level 2.6 kJ/km, vertical 28 kJ/km
Lactation Energy
- Energy for synthesising & retaining fat, protein, lactose
- Efficiency declines as lactation progresses
- Factors altering ME cost: stage of lactation & feed energy density
Negative Energy Balance & Ketosis
- Early lactation: DMI limited → NEB; mobilisation of adipose (NEFA) & muscle (AA)
- Lipolysis products → acetate, \beta-hydroxybutyrate; excess → ketosis (↓pH, acetone odour, ↓appetite)
- Prevention: high-energy ration, glucose precursors, optimal BCS pre-calving
Key Review Points
- List precursors & cell locations for synthesis of lactose (glucose → Golgi), protein (AA → RER/Golgi), fat (acetate/NEFA → ER)
- GLUT-1 + phosphorylation traps glucose
- Fat is the milk component most easily altered by diet
- Energy functions: maintenance, activity, milk, pregnancy, body condition change
- Maintenance affected by BW, activity, environment
- NEB highest in early lactation; ketosis = excess ketone bodies due to low glucose