Detailed Notes on Plant Responses to Internal & External Signals

Goals: Understand plant responses to stimuli and mechanisms of immunity.
Outcomes:

  • Explain auxin's role in cell elongation and development.

  • Describe key plant hormones: cytokinins, gibberellins, ethylene.

  • Define photomorphogenesis and photoperiodism's impact on flowering.

  • Explain plant immune responses: PAMP-triggered and effector-triggered immunity.

Stimuli and Stationary Life:

  • Plants alter growth in response to stimuli (unlike animal movement).

  • Example: Dodder seedlings bend towards host plants.

Plant Hormones:

  • Auxin (IAA): Stimulates stem elongation, root formation, regulates fruit development.

  • Cytokinins: Promote cell division, lateral bud growth, and delay leaf senescence.

  • Gibberellins (GA): Stimulate stem elongation, fruit growth, seed germination.

  • Abscisic Acid (ABA): Inhibits growth, promotes stomatal closure, and seed dormancy.

  • Ethylene: Regulates fruit ripening, leaf abscission, and stress responses.

  • Brassinosteroids: Promote cell expansion and division, inhibit phloem differentiation.

  • Jasmonates: Involved in responses to herbivory; regulates fruit, pollen, and root growth.

  • Strigolactones: Control apical dominance and mycorrhizal interaction.

Phototropism and Hormone Discovery:

  • Phototropism: Growth towards light via auxin.

  • Acid Growth Hypothesis: Explains auxin-induced cell elongation through cell wall loosening and turgor pressure increase.

Responses to Light:

  • Photomorphogenesis: Light affects growth; red/blue light are key.

  • Phytochromes: Regulate responses like seed germination and flowering.

Flowering Control:

  • Photoperiodism: Relative length of light/dark influences flowering.

  • Short-day and long-day plants rely on specific night lengths for flowering.

Plant Immunity:

  • First defense: Epidermis barrier.

  • PAMP-triggered Immunity: Recognizes pathogen patterns leading to antimicrobial responses.

  • Effector-triggered Immunity: Response to pathogen effectors; involves resistance genes and systemic acquired resistance for widespread defense.