Disruptive, Impulse-Control, and Conduct Disorders
Covered Disorders
Oppositional Defiant Disorder (ODD)
Intermittent Explosive Disorder (IED)
Conduct Disorder (CD)
Pyromania
Kleptomania
Study Pointer:
Focus on age of onset, behavioral patterns, motivation behind acts (e.g., impulsivity vs. planning), and comorbidities.
Typical Development of Aggression
Newborn Crying = Early communication
Anger appears by 2–6 months
Aggression = Self-defense and communication tool in toddlers
Normally declines by adolescence
Study Pointer:
Recognize when aggression is part of normal development vs. pathological.
Oppositional Defiant Disorder (ODD)
Diagnostic Criteria (≥4 for ≥6 months):
Angry/Irritable Mood:
Loses temper
Easily annoyed
Angry and resentful
Argumentative/Defiant Behavior:
Argues with authority
Defies rules
Deliberately annoys
Blames others
Vindictiveness:
Spiteful or vindictive ≥2× in past 6 months
Causes significant impairment
Not better explained by mood, psychotic, or conduct disorder
Severity:
Mild: One setting
Moderate: Two settings
Severe: Three or more settings
Gender Differences:
Boys: More overt aggression (yelling, arguing)
Girls: More covert (lying, passive resistance)
Study Pointer:
ODD is non-delinquent and often comorbid with mood disorders or ADHD.
ODD Etiology
Biological:
Family history of ADHD, ODD, CD, mood disorders, substance use
Brain impairments (judgment, impulse control)
Neurochemical imbalances
Psychological:
Poor parental attachment
Difficulty forming social relationships
Social:
Poverty, abuse, neglect, lack of supervision, chaotic homes
Study Pointer:
Use a biopsychosocial model when assessing cause and planning treatment.
ODD Treatment
Parent Management Training: Teaches discipline and communication
CBT: Teaches coping, emotion regulation
Social Skills Training: For peer interactions
Medication: Only for comorbidities (e.g., ADHD)
Alpha-2 agonists (Clonidine, Guanfacine)
SSRIs, stimulants, or atypical antipsychotics (Risperdal)
Prognosis:
67% improve with treatment in 3 years
30% progress to Conduct Disorder
Greater risk if diagnosed in preschool
10% develop Antisocial Personality Disorder
Study Pointer:
Early intervention and family-based therapy prevent escalation to CD or ASPD.
Intermittent Explosive Disorder (IED)
Core Features:
Repeated impulsive aggressive outbursts (verbal/physical)
Outbursts disproportionate to provocation
Not premeditated or goal-directed
Causes impairment or distress
Criteria:
Verbal/physical outbursts ≥2×/week for 3 months
Destruction/assault events within 12 months
Must be ≥6 years old
Symptoms:
Sudden episodes lasting <30 minutes
Regret, embarrassment often follow
Risk Factors:
Childhood trauma
Genetic predisposition
Serotonin abnormalities in limbic areas (e.g., amygdala, orbitofrontal cortex)
Comorbidities:
Depression, anxiety, substance use, ASPD, BPD
ADHD, CD, ODD
Study Pointer:
IED is impulsive and emotion-based. Aggression isn’t preplanned or for gain.
IED Treatment
CBT: Restructure thoughts, teach coping
Group Therapy: Reduces symptoms and aggression
School-Based Programs: Early prevention in teens
Pharmacotherapy:
SSRIs (e.g., Prozac): Early aggression reduction
Mood stabilizers (Lithium, Trileptal)
Antipsychotics (if severe)
Challenge:
Many seek treatment late or only after violence has occurred.
Conduct Disorder (CD)
Core Feature:
Persistent violation of rights of others or societal norms
Diagnostic Criteria (≥3 in 12 months; 1 in last 6 months):
Aggression:
Bullying, fights, weapons, cruelty, forced sex
Property Destruction:
Fire setting, vandalism
Deceit/Theft:
Breaking in, lying, stealing
Rule Violations:
Running away, truancy, staying out late before age 13
Types:
Childhood-Onset: ≥1 symptom before age 10
Adolescent-Onset: No symptom before age 10
Unspecified
Specifier: "With Limited Prosocial Emotions"
Lacks guilt, empathy, shallow affect
Severity:
Mild, moderate, severe based on harm and impact
Study Pointer:
CD has the highest risk of transitioning to Antisocial Personality Disorder in adulthood.
CD Risk Factors
Temperament: Difficult infant temperament, low verbal IQ
Environment: Abuse, neglect, inconsistent discipline
Genetic: Parental criminality, psychiatric disorders
Prevalence:
2–10% (4:1 male-to-female ratio)
Worldwide: 3.2%
25–50% develop ASPD/BPD
CD Treatment
CBT: Behavior management and emotion regulation
Family Therapy: Improves communication and parenting
Peer Group Therapy: Builds empathy and social skills
Medication: Only for comorbid conditions (e.g., ADHD)
Comorbidities:
ADHD, ODD, SLD, Anxiety, Depression, Bipolar, Substance Use
Study Pointer:
Early-onset CD is more severe and long-lasting. Family intervention is critical.
Pyromania
Key Features:
Deliberate fire setting on >1 occasion
Arousal before, pleasure/relief after fire
Fascination with fire-related objects
Not done for money, revenge, or psychosis
Risk Factors:
Male, ≥18 years old
Learning/social skill deficits
Etiology:
Unknown; linked to brain chemistry, genetics, trauma
Kleptomania
Key Features:
Recurrent impulse to steal unneeded items
Tension before theft; relief after
Not for anger, vengeance, or psychosis
Other Features:
Items are unused or returned
Occurs alone, spontaneously
Causes:
Low serotonin, dopamine release, opioid imbalance
Risk Factors:
Family history, other mental illnesses (e.g., Bipolar, OCD, EDs)
Treatment for Pyromania & Kleptomania
CBT: Core intervention
Pharmacotherapy: Limited data
Naltrexone (opioid antagonist)
Antiepileptics, lithium, antipsychotics (proposed)
Study Pointer:
Differentiate impulse disorders from planned behaviors. Motivation is tension relief, not gain.
Final Study Tips
Know diagnostic criteria, duration, severity levels for all 5 disorders
Distinguish impulsive vs. premeditated acts
Learn the trajectory from ODD → CD → ASPD
Understand comorbidities and family/environmental risks
Remember CBT is central to treatment across disorders