Diuretics, ACE
Diuretics Overview
Diuretics are used for various medical conditions such as high blood pressure (HBP), edema, heart failure, and ascites.
They aid in the elimination of toxins from the body.
Use of Diuretics in Hypertension
First-Line Therapy: Used as the first-line drug therapy for hypertension unless contraindicated.
Benefits:
Safe and inexpensive.
Effective in preventing serious cardiovascular events such as stroke and myocardial infarction.
Recent evidence shows they are superior to β-blockers in treating hypertension in older adults.
Mechanism of Action
Relieve pulmonary congestion and peripheral edema.
Reduce symptoms of volume overload (e.g., orthopnea).
Decrease plasma volume leading to reduced venous return (preload) to the heart, thus reducing cardiac workload and oxygen demand.
May decrease blood pressure by reducing afterload.
Sites of Action of Diuretics
Proximal Tubule:
Osmotic diuretics, Carbonic anhydrase inhibitors.
Loop of Henle:
Loop diuretics.
Distal Tubule:
Thiazide diuretics.
Collecting Duct:
Potassium-sparing diuretics, Aldosterone antagonists.
Types of Diuretics and Their Mechanisms
Osmotic Diuretics
Example: Mannitol; acts at the proximal tubule, enhancing Na+ reabsorption via osmotic action.
Carbonic Anhydrase Inhibitors
Example: Acetazolamide; inhibits carbonic anhydrase, increasing bicarbonate excretion.
Thiazide Diuretics
Example: Hydrochlorothiazide; decreases sodium chloride reabsorption in early distal tubule.
Loop Diuretics
Examples: Furosemide, Ethacrynic acid; work at the cortical and medullary TALH to inhibit Na+/K+/2Cl-symporter.
Potassium-Sparing Diuretics
Examples: Spironolactone, Amiloride; reduce potassium loss and may prevent cardiac remodeling.
Thiazide Diuretics: Mechanism of Action
Initially lower blood pressure by increasing sodium and water excretion.
Decrease extracellular volume, affecting cardiac output, renal blood flow, and peripheral resistance.
Often combined with potassium-sparing diuretics to alleviate potassium loss.
Therapeutic Uses of Thiazide Diuretics
Effectively lower blood pressure in supine and standing positions.
Useful in combination therapy with various antihypertensive agents.
Not effective in patients with low kidney function (creatinine clearance <50 mL/min).
Adverse Effects of Thiazide Diuretics
Common Side Effects:
Hypokalemia, hyperuricemia, hyperglycemia, acute gout attacks, hypomagnesemia.
Monitor potassium levels in susceptible patients.
Dose reductions required when used with lithium and digoxin.
Avoid use with quinidine and quinine.
Loop Diuretics
Fast-acting even in patients with poor renal function.
Lower renal vascular resistance and increase renal blood flow.
Cause increased calcium content in urine, unlike thiazides which decrease calcium.
Side Effects of Loop Diuretics
Common Side Effects:
Hypokalemia, hypomagnesemia, hypocalcemia, hyperuricemia.
Ototoxicity and hypotension may occur.
Osmotic Diuretics: Mannitol
Increase urinary excretion of electrolytes, decrease blood viscosity, and inhibit renin release.
Used in acute renal failure, brain edema, and transient effects on the blood-brain barrier.
Side effects include pulmonary edema and dehydration risks.
Potassium-Sparing Diuretics
Include Amiloride, Triamterene, Spironolactone.
Reduce potassium loss and may help in cardiac remodeling in heart failure.
Aldosterone Antagonists
Spironolactone reduces salt retention and myocardial hypertrophy. Used in advanced heart failure cases.
Eplerenone has less endocrine side effects and reduces mortality post-myocardial infarction.
Renin-Angiotensin System Overview
ACE Inhibitors (ACEI): First-line agents when diuretics or β-blockers are not viable.
Mechanism: Reduce vascular resistance without increasing cardiac output.
Indications: Beneficial for diabetic nephropathy, post-myocardial infarction, chronic heart failure.
Adverse Effects of ACE Inhibitors
Potential side effects include dizziness, hypotension, GI distress, and cough.
Angiotensin II Receptor Blockers (ARBs)
Block AT1 receptors, leading to arterial and venous dilation.
Favorable for hypertensive patients with low tolerance for ACE inhibitors.
Lower risk of cough and angioedema compared to ACE inhibitors.
Renin Inhibitors: Aliskiren
Directly inhibits renin, lowers blood pressure effectively.
Can be combined with other antihypertensives.
Adverse effects include diarrhea, cough, and hyperkalemia, especially with ARBs.
Common Fixed-Combination Medications
Examples include:
Hydrochlorothiazide with Lisinopril or Losartan, among others.