16 - Retina & Optic nerve
Know how to recognize persistent hyperplastic primary vitreous, asteroid hyalosis and vitreal hemorrhage.
persistent hyperplastic primary vitreous
primary vitreous proliferates to form vessels and fibrous throughout vitreous, around lens, and into lens substance
can cause spontaneous hemorrhage
asteroid hyalosis
calcium or lipid deposits suspended in vitreous
move when eye moves → bounce back to original position when eye stops moving
clinically insignificant
secondary to inflammatory or intraocular neoplasia
Know the layers of the retina and the vascular patterns of common species.
retinal pigmented epithelium
photoreceptors (rods and cones)
external limiting membrane
outer nuclear layer
outer plexiform layer
inner nuclear layer
inner plexiform layer
ganglion cells
nerve fiber layer
internal limiting membrane
holangiotic
dogs, cats, cattle, sheep, pigs
blood vessels scattered throughout retina
paurangiotic
horses
vessels surround optic disc
merangiotic
rabbit
vessels run east and west
anangiotic
birds and reptiles
no blood vessels
What are the lesions seen in the condition known as collie eye anomaly? Which of these lesions interfere with vision? Is this a progressive disease? What do we recommend concerning breeding these animals?
choroidal hypoplasia
coloboma of optic disc → interfere
retinal detachment → interfere
intraocular hemorrhage → interfere with vision
excessive tortuosity of retinal vessels
vermiform streaks
CEA is not a progressive disease
recommend not to breed
Be able to describe/recognize the clinical presentation (history, clinical signs) associated with progressive retinal atrophy (PRA) and sudden acquired retinal degeneration (SARD). Be able to differentiate and diagnose PRA and SARD.
PRA
autosomal recessive
always bilateral
loss of vision → night vision first → rods
diminished PLRs
attenuation of vessels
tapetal hyperreflectivity
pale optic disk
depigmentation of the non-tapetum
cataract formation
may have some activity on ERG
SARD
sudden onset of day and night blindness
absent/sluggish PLRs
PU/PD PP → systemic illness
must have ERG to determine rods and cones are nonfunctional
differentiation
slow vs fast onset
presence of systemic illness
presence of other retinal changes
presence of activity on ERG or not
Describe the lesion(s) seen in feline central retinal degeneration (FCRD). What is one known cause of FCRD?
round to cigar shaped lesion in central retina
area within lesion is hyperreflective → retinal degeneration
associated with lack of taurine
may be associated with systemic disease
Know how to recognize a retinal detachment. What are the common causes of a retinal detachment? What are possible therapies for retinal detachment depending on the original cause?
hypertension
exudative/serous
neoplasia
congenital
trauma
idiopathic/immune-mediated/steroid responsive
inflammatory/infectious
medical: treat medical condition
surgical: retinopexy, scleral buckle, intravitreal reattachment
Know how to recognize retinal hemorrhage. What are the common causes of a retinal hemorrhage? What are possible therapies for retinal hemorrhage?
hypertension
thrombocytopenia
coagulopathy
DIC
hyperviscosity syndrome
radiation
senile
trauma
tx: treat systemic cause, corticosteroids, laser therapy
Be able to describe/recognize the clinical presentation (history/clinical signs) that may be associated with optic neuritis. What is an appropriate work-up for a patient with suspected optic neuritis? What are some possible causes of optic neuritis? What diagnostic test can you use to differentiate between optic neuritis and sudden acquired retinal degeneration (SARD)?
no PLRs
visual deficit
swollen optic disc
dilated retinal vessels
CSF analysis
ERG → use to R/O SARD
EEG
infectious disease testing
meningoencephalitis
tx: steroids, tx underlying disease
Know how to differentiate between optic neuritis and papilledema? What are some common causes of papilledema?
optic neuritis
no PLRs
visual deficit
papilledema
PLRs present
no visual deficit
edema of optic disc
elevation of disc surface above retina
caused by space-occupying mass → neoplasia, hemorrhage, hydrocephalus
Know how to differentiate active from inactive retinal/choroidal lesions (hyper-reflective vs. hyporeflective)
active
hyporeflective
borders are indistinct
perivascular sheathing/engorgement of vessels
retinal changes → hemorrhage, detachment, enlargement of lesion
inactive
hyperreflective
sharply defined borders
decreased vessel size
depigmentation