Host defense and Immunity Part 1 and 2
• Immunology:
• The study of all features of the body’s second and third lines of defense
• Study of the body’s response to infectious agents
• Study of allergies and cancer
INTRODUCTION
• A healthy functioning immune system is responsible for the following:
• Surveillance of the body
• Recognition of foreign material
• Destruction of entities deemed to be foreign
• White blood cells constantly move throughout the body, searching for potential pathogens:
• Recognize body cells (self)
• Differentiate them from any foreign material in the body (nonself)
• The ability to evaluate macromolecules as self or nonself is central to the functioning of the immune system
• Many autoimmune disorders are a result of the immune system mistakenly attacking the body’s own tissues and organs
• The immune system evaluates cells by examining molecules on cell surfaces called antigens or markers:
• Consist of proteins and/or sugars
• Allow cells of the immune system to identify whether a newly discovered cell poses a threat and should be marked for destruction:
• Most common method of destruction is phagocytosis
• Pathogen-associated molecular patterns (PAMPs):
• Markers that many different kinds microbes have in common
• Serve as “red flags” for phagocytes and other cells of innate immunity
• Bacterial PAMPs: peptidoglycan and lipopolysaccharide
• Viral PAMPs: double-stranded RNA
• Pattern recognition receptors (PRRs):
• Used by host cells with important roles in the innate immunity of the second line of defense
• Recognize PAMPs
• Nonself proteins that are not harmful are generally recognized as such and the immune system is signaled not to react or to react differently.
• For effective immune responsiveness, the activities of one compartment must be communicated to other compartments
• Mononuclear phagocyte system (MPS):
• Phagocytic cells enmeshed in this network are collectively called the mononuclear phagocyte system (MPS)
• Provides a passageway between tissues and organs
• In direct contact with tissue cells and the extracellular fluid (ECF)
• Blood and lymphatic capillaries penetrate into these tissues
• Allows cells and chemicals that originate in the MPS and ECF to diffuse into the blood and lymphatics
• Found in:
• Thymus: site of white blood cell maturation
• Lymph nodes
• Tonsils
• Spleen
• Lymphoid tissue of the mucosa of the gut and respiratory tract
• The MPS is loaded with white blood cells called macrophages waiting to attack passing foreign intruders as they arrive in these locations
HOST DEFENSES:
• innate (born with) protections: general defenses; protects against many pathogens
• adaptive (develop through life): more specific; carried out by lymphocytes
• Divided into first, second and third lines of defense*
*Most defenses overlap and are redundant in some of their effects
This assault force makes the survival of invading microbes unlikely
First line of defense:
• Any barrier that blocks invasion at the portal of entry
• Skin and mucous membranes: physical barrier and traps pathogens
• Fluid secretions: tears (lysozyme), saliva, urine—flushing action to remove pathogens
• Chemicals: gastric acid
• Limits access to the internal tissues of the body
Human Microbiome
• Forms a type of structural barrier
• Can block the access of pathogens to epithelial surfaces
• Creates an unfavorable environment for pathogens by competing for limited
nutrients and by altering the local pH
• The first line of defense alone is not sufficient protection: inflammation, phagocytosis, and specific immune responses are also necessary
Second line of defense: Acts rapidly at both the local and systemic levels once the first line of defense has been overcome.
• Internalized system of protective cells (phagocytes) and fluids
• Types of phagocytes: Neutrophils, Monocytes and Macrophages
• Survey the tissue compartments and discover microbes, particulate matter, and injured or dead cells
• Ingest and eliminate these materials
• Includes inflammation, fever and phagocytosis
Inflammation
classic series of signs and symptoms:
• Rubor:redness caused by increased circulation and vasodilation in the injured tissue
• Calor:warmth caused by the heat given off by the increased flow of blood
• Tumor:swelling caused by fluid escaping into the tissues
• Dolor:pain caused by the stimulation of nerve endings
• Loss of function
• Factors that elicit inflammation:
• Trauma from infection
• Tissue injury or necrosis due to physical or chemical agents
• Adaptive immune reactions
• Chief functions:
• To mobilize and attract immune components to the site of injury
• To set in motion mechanisms to repair tissue damage and localize and clear away harmful substances
• Destroy microbes and block their further invasion
Stages of inflammation
1)vasodilation-
• Increased diameter of arterioles allows more blood flow through area bringing supplies and removing debris—redness and heat
2)increased permeability of blood vessels in area
• substances normally retained in the blood are permitted to pass out – antibodies and clotting factors—swelling and pain
• Fluid dilutes toxic substances
3)phagocyte migration: bacteria and dead cells are removed
4)repair
TERMS
• Pus-composed of cellular debris, dead neutrophils and fluid + bacteria
• Pyogenic: bacteria such as streptococci, staphylococci, gonococci, and meningococci that stimulate the formation of pus
• Abscess- excessive pus in an enclosed space
• Ulcer- inflamed tissue is shed many times at one site→open sore
• Exudates- tissue fluid and proteins collects in inflamed tissues
FEVER
• Abnormally elevated body temperature:
• Nearly universal symptom of infection
• Associated with certain allergies, cancers, and other organic illnesses
• If cause is unknown, it’s called a fever of unknown origin (FUO)
• Body temperature is maintained around 37°C (98.6°F) by the hypothalamus:
• Low-grade fever: 37.7 to 38.3°C or 100 to 101°F
• High-grade fever: 40.0 to 41.4°C or 104 to 106°F
Stages of fever response:
• pyrogens reset hypothalamus
• Exogenous pyrogens:products of infectious agents such as viruses, bacteria, protozoans, fungi, endotoxin, blood, blood products, vaccines, or injectable solutions coming from outside the body
• Endogenous pyrogens:chemicals liberated by monocytes, neutrophils, and macrophages during phagocytosis
• hypothalamus initiates fever response
• skin cold
• shivering
• stress is removed
• crisis stage (fever breaks)
• sweating, flushed skin
Function of fever:
• high temperature inhibits bacterial growth/multiplication
increases HR to deliver WBC’s to infected site
• phagocytes (monocytes and lymphocytes) attack bacteria more vigorously
• Increases metabolism and stimulates immune reactions and naturally protective physiological processes:
• Speeds up hematopoiesis, phagocytosis, and specific immune reactions
•
complications of a fever
• Tachycardia: rapid heart rate
• Tachypnea: elevated respiratory rate
• Lowering of seizure threshold
• dehydration, acidosis, brain damage
• fatal above 46 Celsius (114 degrees F)
ANTIMICROBIAL SUBSTANCES
• Interferon-hormone like peptides
• produced by lymphocytes, macrophages, and fibroblasts Infected with virus
• bind to receptors on nearby cells, cell is induced to synthesize antiviral chemicals (prevent viral replication)
• also mediate other immune responses (phagocytosis) and prevent tumor growth
• The complement system
• proteins in blood that are activated to enhance immune, allergic and inflammatory reactions.
• Function of COMPLEMENT:
• activation of inflammation
• immune adherence=binds to microbe so phagocyte can recognize it as a foreign body (attracts phagocytes)
• Enhances phagocytosis (cytolysis=attacks microbial membrane )
Adaptive Immunity
• Third line of defense:
• Acquired on an individual basis as each foreign substance is encountered by lymphocytes
• Adaptive immunity acquired only after an immunizing event such as an infection
• The reaction with each different microbe produces unique protective substances
• Provides long-term immunity
• B and T lymphocytes undergo a selective process that prepares them for reacting only to one specific antigen or immunogen
• Formation of T and B cells
• derived from stem cells in bone marrow-released in blood before they differentiate
• T cells mature in the thymus via thymosin, migrate to spleen, lymph node and nodules
• B cells mature in Bone marrow and migrate to spleen , lymph nodes , and nodules
• B and T cells constantly recirculate through the circulatory system and lymphatics, migrating into and out of the lymphoid organs
• Immunocompetence:the ability of the body to react with countless foreign substances
• Antigens: molecules that can be seen and identified by the immune system
• If they provoke a response, they are called immunogens
• Protein or polysaccharide molecules on or inside cells and viruses
• Any exposed or released protein or polysaccharide is potentially an antigen, even those on our own cells:
• Our own antigens usually do not evoke a response from our own immune system
• Specificity:antibodies produced against the chickenpox virus will not function against the measles virus
• Memory:lymphocytes have been programmed to “recall” their first engagement with the invader and rush to the attack once again
MECHANISM OF ADAPTIVE IMMUNITY
• When pathogens carrying antigens cross the first line of defense:
• Resident phagocytes migrate to the site
• Tissue macrophages ingest the pathogen and initiate an inflammatory response
• Phagocytic cells ingest the antigen and migrate to the nearest lymphoid organ, where they process and present antigen to T and B lymphocytes
• When challenged with antigen, both B and T cells proliferate and differentiate:
• A clone, or group of genetically identical cells, is created
Cell Mediated Immunity: How T cells respond to antigen
• The three functional types of T cells are as follows:
• Helper T cells (CD4):activate macrophages, assist B-cell processes, and help activate cytotoxic T cells
• Regulatory T cells:control the T-cell response by secreting anti-inflammatory cytokines or preventing proliferation
• Cytotoxic T cells (CD8):lead to the destruction of infected host cells and other “foreign” cells
MECHANISM
• T cells secrete cytokines to help destroy pathogens, but they do not produce antibodies
effective against intracellular pathogens-viruses, fungi, malignant cells, grafts
• ANTIGEN →stimulate helper T cells, which become activated and specific to the antigen and divide many times forming→
MEMORY T CELLS →remembers subsequent infection/faster the 2nd time; part of long term immunity
REGULATORY T CELLS-stop immune response once antigen is destroyed
CYTOTOXIC T CELLS →killer cells ; chemically destroy cell membrane and prevent virus from replicating and make cytokine, which attract macrophages.
Activated by Helper T cells or antigen/macrophage
Humoral Immunity: How B cells respond to Antigen
• When activated, B cells divide and give rise to plasma cells
• Plasma cells release antibodies into the tissue and the blood
• Antibodies attach to the antigen for which they are specific, and the antigen is marked for destruction or neutralization
• proteins; AKA-immunoglobins constitute the gamma globulin fraction of plasma
• specialized to react with specific parts of antigens
• types of immunoglobulins-IgA, IgM, IgG, IgE
• direct attachment results in agglutination, lysis, precipitation , or neutralization
MECHANISM
•sensitized helper T cells presents the foreign antigen to B cells, which provide a strong stimulus for the activation of B cells specific for this antigen
•the activated B cell divide many times into 3 types:
MEMORY B CELLS-will remember the specific antigen in future, part of long term immunity-faster response 2nd time
PLASMA CELLS-produce antigen – specific antibodies
REGULATORY CELLS- stop immune response once antigen is destroyed
Antibody Titer:levels of antibodies in the serum over time
• Measuring specific antibodies can help determine vaccination status against or previously infected by a certain pathogen.
• It can also help test integrity of immune system
RESPONSE TIME TO ANTIGEN
• Primary Response: the first exposure to an antigen the system undergoes
• Secondary Response: When the immune system is exposed again to the same immunogen within weeks, months, or even years
• Faster response
TYPES OF IMMUNITY
• Natural active-get sick; get well-results in memory B (antibodies) and T cells
• Natural passive-antibodies from another source-placental transmission of antibodies from mother to fetus or transmission of antibodies in colostrum
• Colostrum: first breast secretion, appears yellow and is high in immunoglobins (IgA)
• Protect infant against enteric pathogens (E. coli)
• Artificial passive-person is given an injection of preformed antibodies ( gamma globulins or immunoglobulins) after presumed exposure to particular pathogen
• Most useful in patients who have a diminished ability to mount their own immune response
• Artificial active-body is presented with antigen and immune system responds-a vaccine stimulates production of antibodies and memory cells