Neurodevelopment Disorders
Basics of brain development
Neuron overproduction and selection
Synaptogensis
Dendrites starting to have synapses
Down Syndrome
Genetic disorder caused by a triplication of chromosome 21
Distinct physical attributes
30% born with heart defects
GI tract problems
10% have comorbid seizures
Delayed developmental milestones
Reduced cogntiive abilites
Explicit long-term memory deficits
Fragile X
>200 CGG repeats on the X chromosome
Reduces production of FMRP protein —- too much sunthesis of some proteins
mGluR5
Distinct physical attributes
Anxious, hypersensitive, impulsive, ADD-like symptoms
Delayed language development and intellectual disabilities
RETT Syndrome
X-linked mutation of gene for MeCP2 protein (this protein normally binds to chromosomes and either enhances or represses gene expression, playing a crucial role in the development and function of neurons in the brain). This mutation leads to the characteristic symptoms of Rett Syndrome, including loss of purposeful hand skills, gait abnormalities, and cognitive impairment.)
All cells have MeCP2 (it turns on/ off genes)
Almost 100% seen in females
Normal development until for 6-18 months
Then regress - lose language, lose purposeful movement, hand flopping
Develop autistic behaviors
Progressive motor problems
Underdeveloped cognitive abilities
Respiratory and GI problems
Prefrontal and frontal cortex fails to develop
Only one X chromosome is mutated, not both
If both X chromosomes are mutated = death
Treatments
How does gene therapy work?
Mutated gene (replace that gene with a normal gene)
Problems with Gene therapy (too much MeCP2 is just as bad as not enough)
All cells have MeCP2
Can cause liver failure and ultimately death
Gene Editing
Morally questionable
RNA editing
Re-activate X-chromosome