Lecture6_AxonGuidance&GrowthCones_Notes
Axon Guidance & Growth Cones Overview
Date: February 9, 2014
Course: NRS 401
Quiz: Due tomorrow at midnight covering Lectures 4 & 6.
Review of Key Concepts
Proliferation
First Step in Development: Proliferation is essential for increasing the pool of progenitors in developing organisms.
Types of Division:
Symmetric Division: Increases progenitor pool.
Asymmetric Division: Leads to the bias towards neuron production influenced by factors:
Neurons influenced by Brain-Derived Neurotrophic Factor (BDNF) and Wnt.
Glial cells influenced by CNTF, BMP, and Sonic Hedgehog (Shh).
Migration along Radial Glia
Immature Neurons: Migrate on radial glial cells using:
Cell Adhesion Molecules: Essential for migration.
Rab Proteins: Enhance migration when influenced by environmental factors, particularly BDNF via TrkB.
Mechanisms of Migration:
Increased Rab5 & Rab11.
Reduced levels of Reelin impart regulation on migration.
Role of Reelin
Function: Acts as a stop and detach signal in neuronal migration.
Impact of Reelin Reduction: Disrupts the inside-out pattern of neuronal development where newer neurons pass older neurons.
Restoration of Reeler Phenotype: Can be improved by increasing Reelin levels or enhancing Rab5 & Rab7 levels.
Differentiation of Cells
Initiation of Neuronal and Glial Fates
Cell Characteristics: Differentiation features distinct physical characteristics that lead to neuron or glial fate.
Factors influencing fate:
Inherent Influences: Asymmetric synthesis and epigenetic factors.
Environmental Influences:
BDNF & Wnt promote neuronal differentiation.
BMP, Shh, CNTF promote glial differentiation.
Axonal Development
Differentiation Aspect: Axonal development occurs alongside dendritic growth.
Key Points:**
Growth of processes happens simultaneously.
Observations on what occurs to cell bodies and processes during development.
Neuronal Cytoskeleton Overview
Structure in Neurons: Critical proteins provide structural support distributed specific patterns.
Neurofilaments: Stable and provide rigidity.
Actin Networks: Flexible and modifiable to allow for plasticity.
Microtubules: Rigid yet modifiable, functioning as transport tracks.
Microtubule Dynamics
Microtubule Changes During Differentiation
Stage 1: Microtubules radiate from centromere forming star-like patterns; crucial for structural integrity.
Formation of Web-Like Structures: As dendrites extend, a web-like pattern forms in the soma leading to stability and structure for transport.
Axonal and Dendritic Development
Unidirectional Assembly in Axons: Microtubules exhibit unidirectional growth during axon development.
Bidirectional Dynamics in Dendrites: Dendritic assembly is characterized by bidirectional growth.
Growth Cone Structure and Function
Regions of Growth Cone
**Domains: **Three distinct regions identify growth dynamics:
C-Domain: Houses microtubules providing stability.
P-Domain: Actin-rich region, dynamic and responsive to signals.
T-Domain: Serves as a transition between structures.
Role of Actin in P-Domain
Actin Dynamics: Filopodia extending and retracting to survey environmental signals like BDNF and FGF-2, which serve as chemoattractants.
The Clutch Hypothesis
Mechanism of Growth Cone Movement: As actin extends and links with membranes, it generates a force for growth direction. Myosin contracts to:
Retract the actin cytoskeleton.
Pull along microtubules, facilitating movement.
Neural Guidance Through Chemical Signals
Effects of Chemoattractants and Chemorepellents
Chemotactic Responses: Filopodia express receptors that respond to signals enabling navigation and directional growth:
Attractants (e.g., BDNF, Netrin) promote microtubule stability and actin growth.
Repellents lead to microtubule destabilization and actin depolymerization.
Pauses in Growth Cone Development
Significance of Pauses: Enable reorganization and branching through microtubules.
Most common in larger axons that cross midline, undergoing fracturing produces branch points.
Midline Dynamics in Axon Guidance
Challenges at Midline Crossing
Attractant Signals Necessary: For effective crossing, attractants signal movement while responsive signals dictate left/right turns post-crossing.
Neuronal Signaling Modulation: Protein expression shifts post-midline, emphasizing sensitivity to repulsive and attractive signals, governed by receptor presence (e.g., Robo3 variants).
Case Study: Horizontal Gaze Palsy
Symptoms: Inability to coordinate eye movements and asynchronous blinking linked to structural disruptions in axon guidance, leading to neurological diseases.
Outcome of Axon Growth: Initial slow growth accelerates with complexity as guidance cues become established.
Conclusion
Axonal Tracking: A coordinated process facilitated by connections, resulting in robust networks in the nervous system.