MS Ch65 Oncologic & Degenerative Neurologic Disorders – Comprehensive Review
Brain Tumors
- Classification
- Benign vs. Malignant
- Benign → slow-growing (e.g., meningioma) yet may compress tissue
- Malignant → rapid growth, infiltrative, require aggressive therapy
- Primary vs. Secondary
- Primary: arise in CNS, do NOT metastasize
- Secondary (metastatic): spread from lung, breast, kidney, melanoma, etc.
- Major Primary‐Tumor Types
- Gliomas – astrocytoma, oligodendroglioma, glioblastoma
- Meningioma – usually benign but invasive
- Acoustic neuroma (vestibular schwannoma) – CN VIII, ⇣hearing, tinnitus, vertigo
- Pituitary adenoma – hormonal syndromes (Cushing, acromegaly)
- Angioma – vascular malformation ↑ risk of hemorrhage (not true neoplasm)
- Clinical Manifestations
- Depend on size, location, compression
- ↑ICP signs: headache, vomiting, papilledema, \uparrow BP, \downarrow HR/RR (Cushing’s triad)
- LOC changes: drowsiness → coma, restlessness, purposeless movements
- Focal deficits: seizures, motor/sensory loss, visual changes, hormonal effects, CN VIII deficits
- Common summarized S/S: headaches, N/V, seizures, sensory loss, ataxia, cognitive changes
- Diagnostics
- Neuro exam, CT (screen/ER), MRI (gold standard), PET, EEG, CSF cytology
- Surgical biopsy → histology & treatment planning
- Medical/Surgical Management
- Goal: total resection w/o new deficits OR decompression
- Procedures: craniotomy, trans-sphenoidal, stereotactic (biopsy or radiosurgery)
- Radiation (external beam, stereotactic): DNA damage → cell death; watch for tissue deformation
- Chemotherapy: IV/systemic; pre-medicate with anti-emetics; alopecia common
- Adjunct drugs: analgesics, dexamethasone (edema), phenytoin (seizure prophylaxis)
- Nursing Focus
- Baseline neuro, ICP monitoring, seizure precautions
- Promote independence, cognitive orientation, family + hospice referrals
- Nutrition: oral care, pleasant meals, preferred foods, weight & intake records
- Encourage advance directives before cognitive decline
Spinal Cord Tumors
- Anatomy-Based Types
- Intramedullary – inside cord
- Extramedullary (intradural/extradural) – outside cord but in canal
- Manifestations
- Pain (worse at night/lying), weakness, \downarrow motor/reflexes, sensory loss, para- or tetraplegia (level dependent)
- Treatment
- Surgical removal if accessible
- Dexamethasone + radiation to reduce edema/compress.
Parkinson’s Disease (PD)
- Key Patho Steps
- Degeneration substantia nigra → \downarrow dopamine
- Dopamine depletion in nigrostriatal pathway
- Relative \uparrow acetylcholine → excitatory dominance
- Basal-ganglia circuit imbalance → impaired extrapyramidal tracts
- Four Cardinal Signs
- Resting tremor (pill-rolling; worse fatigue, better movement/sleep)
- Muscle rigidity (cogwheel; ↓ ROM; difficult rising—use raised seats)
- Bradykinesia/akinesia (slow/frozen; shuffling start-hesitation)
- Postural instability (stooped, propulsive gait, falls)
- Other Motor & Autonomic Features
- Mask-like facies, monotonous speech, dysphagia, drooling
- Sweating, flushing, orthostatic hypotension, gastric/urinary retention
- Psychiatric: depression, anxiety, dementia, hallucinations
- Assessment
- Observe speech, facial expression, tremor amplitude, swallowing, ROM, cognition, fall risk
- Goals
- Maintain mobility & ADLs, bowel regularity, nutrition, communication, coping
- Nursing Interventions
- Daily exercise, PT/OT, rocking to initiate movement, rest periods, safe footwear, assistive devices
- Adaptive utensils, thickened liquids, speech therapy referral
- Support groups, realistic goals, environmental mods
- Medical Management
- Levodopa + Carbidopa (Sinemet) ↑ brain dopamine
- Additional meds: dopamine agonists, MAO-B inhibitors, anticholinergics
- Stereotactic surgeries: thalamotomy (tremor), pallidotomy (rigidity), DBS
Huntington’s Disease (HD)
- Autosomal dominant, onset 25!–!35\,\text{yr} (±17)
- Premature death of cells in striatum & cortex
- Triad of Progression
- Chorea – jerky uncontrolled limb/trunk/face
- Cognitive decline – executive dysfunction → dementia
- Psychiatric – depression, anxiety, irritability, psychosis, suicidality
- Motor Complications
- Balance/falls (helmet), rigidity late, dysarthria, dysphagia (no milk), weight loss, fatigue
- Diagnostics
- Genetic test (CAG repeat); CT shows brain shrinkage (≈30% weight loss)
- Management (No cure)
- Goals: symptom relief, complication prevention, family support
- Meds
- Antipsychotics: haloperidol, chlorpromazine, olanzapine (avoid dystonia)
- Antidepressants: fluoxetine, sertraline, nortriptyline
- Anxiolytics/BZDs: diazepam, lorazepam, alprazolam
- Mood stabilizers: lithium, valproate, carbamazepine
- Botulinum toxin for dystonia
- Nutrition: ↑ calories, small frequent meals, thickened liquids, vitamin supplements, feeding tube PRN
- Exercise, social activity, protective environment, caregiver respite
Amyotrophic Lateral Sclerosis (ALS)
- Pathophysiology
- Loss of upper & lower motor neurons → muscle denervation
- "Amyotrophy" = wasting; "Lateral" = lateral corticospinal tract; "Sclerosis" = gliotic scarring
- Manifestations
- Progressive distal → proximal weakness & atrophy; fasciculations, cramps, spasticity, brisk DTRs
- Bulbar involvement: dysarthria, tongue atrophy, dysphagia, nasal speech
- Respiratory weakness → failure (major mortality cause)
- Etiologic Theories
- Viral, autoimmune, excess glutamate excitotoxicity
- Priority Problems / Interventions
- Respiratory: monitor ABGs, spirometry, BIPAP, suction, aspiration prevention
- Swallowing: thick liquids, PEG tube, speech therapy
- Musculoskeletal: ROM, preventing contractures, assistive devices
- Psychosocial: advance directives, support groups
- Pharmacology: Riluzole (glutamate inhibitor) slows progression; symptomatic meds
Muscular Dystrophy
- Inherited (e.g., Duchenne X-linked)
- Progressive skeletal muscle wasting, ↑ CK enzymes
- No cure; supportive care, PT, orthotics, cardiac/resp monitoring
Degenerative Disc Disease
- Age/trauma → disc dehydration & herniation
- Low-back pain; radiculopathy due to nerve root compression
- Conservative therapy: rest, NSAIDs, PT; surgery if refractory
Post-Polio Syndrome
- Occurs 30!–!40\,\text{yr} after initial infection
- New weakness, fatigue, pain; cause unclear (overuse, aging)
- No definitive treatment; energy conservation, PT, orthoses
Cervical Discectomy
- Indications: cervical herniation, stenosis, radiculopathy, myelopathy unresponsive to conservative care
- Pre-Op Assessment
- Pain onset, radiation, aggravating factors
- Neuro: paresthesia, motor deficits, bilateral vs unilateral symptoms
- Palpation → tone, tenderness; ROM limits
- Review comorbidities, prior spine surgeries; patient education & consent
- Potential Complications
- Hematoma → cord/airway compromise: monitor swelling, stridor, neuro changes, urgent evacuation
- Persistent/recurrent pain: NSAIDs, opioids (short-term), muscle relaxants, PT, possible epidural steroids or revision surgery
- Post-Op Goals & Nursing Care
- Pain relief: analgesics, positioning
- Mobility: brace if ordered, gradual PT
- Complication surveillance: dysphagia, hoarseness, infection
- Home care: activity limits (no lifting/twisting), wound care, follow-up
General Ethical / Advance-Directive Considerations
- Cognitive-decline disorders (brain tumors, PD, ALS, HD) → obtain advance directives early while patient A&O
- Discuss options: CPR, feeding tubes, ventilatory support, hospice