Med 1
OVERVIEW
Chapter focus: Prehospital recognition, assessment and treatment of respiratory emergencies in adults and special populations (paediatric & geriatric)
National EMS Education Standards: Medicine – Respiratory content area; competency = apply knowledge to provide basic emergency care/transport based on assessment findings of an acutely-ill patient
19 major objectives ranging from terminology to special-population considerations
KEY TERMINOLOGY
Wheezing, rhonchi, crackles, dyspnoea, respiratory distress/failure/arrest, hypercarbia, hypoxaemia, hypoxia, bronchoconstriction, bronchodilator, SCAPE, spontaneous pneumothorax, etc.
Know page-reference table for first appearance in textbook
RESPIRATORY SYSTEM REVIEW
Major divisions
Upper airway
Lower airway
Lungs + accessory structures
Gas-exchange principles
Ventilation ⇌ Perfusion balance; disturbances on either side → cellular hypoxia & acidosis
Equation for pulse-ox target: SpO_2 \ge 94\% (except COPD baseline or pregnancy >20 weeks – target highest possible)
Respiratory control & receptors
Medulla + pons centres; stretch receptors; irritant receptors; juxta-capillary receptors (J-receptors) – sense engorgement → rapid shallow respirations & sensation of dyspnoea
NORMAL vs. ABNORMAL BREATHING
Normal adult RR 12!–!20\;min^{-1} with adequate tidal volume (TV)
Paediatric/geriatric norms differ; always correlate to age and baseline
Adequate breathing = open airway, bilateral breath sounds, minimal accessory-muscle use, normal mental status, SpO_2\ge94\%, skin warm/pink/dry
Inadequate signatures: altered mentation, cyanosis, brady- or tachy-cardia, laboured pattern, poor chest rise, use of accessory & abdominal muscles, retractions, pulsus paradoxus \ge10\;mmHg drop on inspiration
AUSCULTATION TECHNIQUE
Use diaphragm directly on bare skin; patient seated upright if possible
Listen full inspiratory/expiratory phases; mirror-sites comparison
Key locations:
2nd ICS mid-clavicular (trachea/large airways – stridor, rhonchi)
3rd ICS AAL or 4th ICS MAL (bronchioles – wheeze)
5th/6th ICS posterior mid-scapular (alveoli – crackles)
BREATH-SOUND PATHOPHYSIOLOGY
Wheeze = lower-airway narrowing (asthma, COPD, CHF); primarily expiratory unless severe
Rhonchi = larger-airway mucus; clears/changes after cough or position
Crackles = alveolar fluid opening; CHF or pneumonia; base-posterior first; not cleared by cough
RESPIRATORY DISTRESS CONTINUUM
Category | TV | RR | Intervention |
|---|---|---|---|
Distress | Adequate | Adequate | O₂ (NC 2 lpm titrate ≥94 %) ± CPAP |
Failure | ↓ or ↑/↓ extreme | Inadequate | PPV + O₂ (BVM) |
Arrest | Absent | Absent/agonal | PPV + O₂ immediately |
MAJOR CONDITIONS & PREHOSPITAL MANAGEMENT
1. Obstructive Pulmonary Diseases
Emphysema
Alveolar wall destruction ➔ ↓ surface area, air-trapping; pursed-lip “physiologic PEEP”
Pink, thin, barrel-chest; diminished sounds; SpO₂ ≥94 % unless in failure
Care: Titrate O₂ 88–92 % for chronic CO₂ retainers; cautious CPAP (5–10 cmH₂O) if moderate/severe distress; monitor for barotrauma
Chronic Bronchitis
Bronchiole inflammation, mucus & scarring; “blue bloater” (outdated)
Productive cough ≥3 mo/year ×2 yrs; coarse rhonchi/wheezes; cyanosis, JVD, pedal oedema
Same care as emphysema; consider CPAP if indications met
Asthma
Triad: bronchospasm, oedema, ↑mucus → expiratory wheeze, prolonged expiration, pulsus paradoxus
Severity guide (mild/mod/severe) by ability to speak, RR>30, HR>120, accessory use
Status asthmaticus = acute severe asthma unresponsive to SABA & O₂; high ALS priority
Management: O₂ ≥94 %; coach controlled breathing; administer SABA (albuterol) via SVN preferable; cautious CPAP; PPV 10–12 min⁻¹ if failure (allow full exhalation to avoid air-trapping)
2. Pneumonia
Infectious (bacterial/viral/aspiration) alveolar fluid/pus; vent-perfusion mismatch
Fever/chills, sharp pleuritic CP, unilateral crackles or rhonchi, splinting, possible ALOC in elderly
Care: O₂ to ≥94 %; consider SABA if bronchospasm; transport
3. Pulmonary Embolism
Sudden perfusion disturbance; risk = DVT/immobility/estrogen/surgery
Presentation: sudden unexplained dyspnoea, sharp chest pain, tachypnoea, tachycardia, possible hemoptysis, clear breath sounds
Assess calves for redness/swelling; manage with high-flow O₂, rapid transport, ALS backup
4. Pulmonary Edema
Cardiogenic
LV failure → ↑hydrostatic pressure → fluid in interstitium/alveoli
Crackles, frothy pink sputum, orthopnoea/PND, HTN, JVD, pedal oedema
Tx: CPAP 5–10 cmH₂O; NTG per protocol; BVM if failure
SCAPE (Sympathetic Crashing Acute Pulmonary Edema)
Flash pulmonary oedema + severe HTN (>200 mmHg); treat with CPAP (up to 15–18 cmH₂O) + NTG (ALS)
Non-cardiogenic (ARDS) – similar care without NTG
5. Spontaneous Pneumothorax
Tall thin males, blebs, COPD; sudden SOB, unilateral decreased sounds, sub-Q emphysema
Contra-indication to CPAP; O₂; PPV cautiously – watch for tension conversion; ALS for decompression
6. Hyperventilation Syndrome
Anxiety trigger; hypocapnia → cerebral vasoconstriction → dizziness, carpopedal spasm
Coach breathing; remove stressor; no paper-bag rebreathing; O₂ only if hypoxic
7. Upper-Airway & Misc.
Epiglottitis: H. influenzae; sudden sore-throat, drooling, tripod, stridor; nothing in mouth, high-flow O₂, calm transport
Croup (peds): barking cough, stridor; cool mist/O₂
Pertussis: paroxysmal coughing, inspiratory “whoop”; PPE, humidified O₂
Cystic fibrosis: thick mucus, recurrent infections; saline SVN, humidified O₂
Poisonous inhalations: remove from source, high-flow O₂, consider CO/H₂S/cyanide
Viral respiratory infection (URI/bronchiolitis/RSV/COVID-19): supportive, monitor for distress; O₂ titrated as needed
MDI & SMALL-VOLUME NEBULISER ADMINISTRATION
Indications: respiratory distress with bronchoconstriction + Rx SABA + med-control authorisation
Contra: inability to follow commands, max-dose taken, non-Rx, med-dir denies
MDI steps: \text{Shake }30\;s → exhale → lips/seal → slow inhale 5 s & actuate → hold 10 s → exhale; spacer/VHC preferred
SVN: place liquid med, connect to 6–8 lpm O₂/compressor, slow deep breaths with occasional 2–3 s hold till mist stops (~5–10 min)
Reassess: RR, SpO₂, breath sounds, HR (β₁ side-effect)
ASSESSMENT-BASED APPROACH SUMMARY
Scene size-up: anticipate need for BVM/AED/CPAP; PPE
Primary: categorise breathing (2 adequate = distress → O₂; any inadequate = PPV+O₂)
Secondary: OPQRST + SAMPLE; focused lung exam, look/feel/auscultate; vital signs incl. SpO_2
Decision making: Distress vs Failure; choose O₂ device, CPAP, MDI/SVN, PPV
Reassessment: q5 min critical; monitor for deterioration (↓SpO₂, fatigue, silent chest, ALOC)
SPECIAL POPULATIONS
Paediatric
Early distress signs: nasal flaring, intercostal/subcostal retractions, tachypnoea >60\;min^{-1}, grunting, seesaw breathing
Failure: bradypnoea, cyanosis, ALOC, head-bobbing, poor tone, HR <60
Tx: Blow-by O₂/NC; BVM 20–25 min⁻¹ if failure; avoid aggressive suction near epiglottitis
Geriatric
Increased baseline RR, diminished airway reflexes; higher risk of infection & heart failure
Atypical presentations; careful titration of CPAP & NTG; watch for polypharmacy
DEVICE/PROCEDURE CONTRA-INDICATION HIGHLIGHTS
CPAP: AMS, vomiting, pneumothorax, facial trauma, SBP<90, no spontaneous drive
MDI/SVN: AMS, non-Rx, max dose, med-dir refuse
High-flow O₂ in ACS unless SpO_2<90\% or dyspnoea/hypoxia/SCAPE
ETHICAL & PRACTICAL POINTS
Silent hypoxia in severe pulmonary disease – rely on SpO_2 and exam not dyspnoea alone
Do NOT use paper-bag rebreathing unless explicitly ordered by MD & no underlying organic cause
Respect pediatric/geriatric caretaker knowledge; incorporate home equipment settings (e.g., chronic O₂, CPAP/BiPAP machines)
NUMERICAL QUICK-REFERENCE (MEMORISE)
Adult normal RR 12–20; distress >24 or <10
Adult SpO₂ goal \ge94\% (COPD 88–92 %; Pregnant ≥ highest attainable)
Severe tachypnoea adult >40\;min^{-1} → likely failure
Paed red flag RR infant >60\;min^{-1}
CPAP typical pressure 5–10 cm H₂O (SCAPE up to 15–18)
Pulsus paradoxus diagnostic threshold \ge10\;mmHg drop SBP on inspiration
CLINICAL PEARLS
Diminished/absent wheeze in severe asthma = impending failure – air flow so low no audible sound
Crackles begin at lung bases; auscultate posterior lower lobes early
In PE, chest auscultation often clear! Dyspnoea + clear lungs + risk factors = suspect PE
COPD/asthma on CPAP – monitor for barotrauma: new unilateral silent hemithorax + sudden SpO₂ drop → tension pneumothorax
Hypoxic drive rare; never withhold O₂ if indicated
SAMPLE EXAM-STYLE EQUATIONS & FORMULAE
Alveolar ventilation V_A = (TV - Dead\;Space) \times RR – falls with ↑RR if TV shallow
Minute ventilation VE = TV \times RR – may look “normal” in distress yet VA inadequate
QUICK DRUG SHEET (EMT-Scope)
Albuterol SABA: 2.5 mg nebulised (SVN) or 4–6 puffs MDI
Levalbuterol: 1.25 mg SVN
Ipratropium (anticholinergic): 0.5 mg often in DuoNeb with albuterol
Epinephrine (racemic): 0.5 mL of 2.25% SVN for stridor/croup (per protocol)
O₂ titration: NC 2–6 lpm → NRB 10–15 lpm; CPAP/BVM as indicated
POST-TREATMENT REASSESSMENT CHECKLIST
Mental status improving?
RR & TV? Work of breathing decreased?
Breath-sound changes (wheezes ↓, crackles stable)?
SpO₂ rising & stable \ge94\% (or target range) with same O₂ device?
HR trending toward normal; BP stable; skin warm/dry
Side-effects of meds: tremor, tachycardia, headache
DOCUMENTATION POINTERS
Pre- & post-treatment vitals + SpO₂
OPQRST/SAMPLE details incl. triggers, home meds, prior episodes
Breath-sound locations & descriptions with time-stamps
Device settings: O₂ lpm, CPAP cmH₂O, BVM rate/VT estimate
Med admin: drug, dose, route, time, effect
REVIEW QUESTIONS
Differentiate bronchospasm vs. fluid crackles origins.
Explain why high RR with shallow TV → ↓VA despite ↑VE.
List CPAP contraindications.
Outline treatment algorithm for asthma that progresses to failure.
Calculate pulsus paradoxus from given SBP readings.
MAJOR CONDITIONS & PREHOSPITAL MANAGEMENT
1. Obstructive Pulmonary Diseases
Emphysema
Definition: Alveolar wall destruction leading to decreased surface area and air-trapping.
Signs & Symptoms: Pink skin, thin build, barrel-chest, diminished breath sounds, SpO₂ above 94% (unless in failure).
EMT Treatment: Titrate O₂ to 88–92% for chronic CO₂ retainers; cautious CPAP (5–10 cmH₂O) if moderate/severe distress; monitor for barotrauma.
Chronic Bronchitis
Definition: Bronchiole inflammation, mucus, and scarring.
Signs & Symptoms: Productive cough ≥3 months/year for 2 years, coarse rhonchi/wheezes, cyanosis, JVD, pedal oedema.
EMT Treatment: Same care as emphysema; consider CPAP if indications met.
Asthma
Definition: Triad of bronchospasm, oedema, and increased mucus, leading to expiratory wheeze, prolonged expiration, and pulsus paradoxus.
Signs & Symptoms: Expiratory wheeze, prolonged expiration, pulsus paradoxus. Severity indicated by ability to speak, RR >30, HR >120, accessory muscle use. Status asthmaticus involves acute severe asthma unresponsive to SABA and O₂.
EMT Treatment: O₂ to ≥94%; coach controlled breathing; administer SABA (albuterol) via SVN (preferred); cautious CPAP; PPV at 10–12 min⁻¹ if failure (allow full exhalation).
2. Pneumonia
Definition: Infectious (bacterial/viral/aspiration) alveolar fluid/pus causing ventilation-perfusion mismatch.
Signs & Symptoms: Fever/chills, sharp pleuritic chest pain, unilateral crackles or rhonchi, splinting, possible altered level of consciousness (ALOC) in elderly.
EMT Treatment: O₂ to ≥94%; consider SABA if bronchospasm; transport.
3. Pulmonary Embolism
Definition: Sudden disturbance of lung perfusion, often due to a blood clot.
Signs & Symptoms: Sudden unexplained dyspnoea, sharp chest pain, tachypnoea, tachycardia, possible hemoptysis, clear breath sounds. Assess calves for redness/swelling.
EMT Treatment: High-flow O₂; rapid transport; ALS backup.
4. Pulmonary Edema
Cardiogenic
Definition: Left ventricular failure leading to increased hydrostatic pressure and fluid in the interstitium/alveoli.
Signs & Symptoms: Crackles, frothy pink sputum, orthopnoea/paroxysmal nocturnal dyspnoea (PND), hypertension (HTN), JVD, pedal oedema.
EMT Treatment: CPAP 5–10 cmH₂O; NTG per protocol; BVM if failure.
SCAPE (Sympathetic Crashing Acute Pulmonary Edema)
Definition: Flash pulmonary oedema with severe hypertension (>200 mmHg).
Signs & Symptoms: Severe HTN (>200 mmHg), flash pulmonary oedema.
EMT Treatment: CPAP (up to 15–18 cmH₂O) + NTG (ALS).
Non-cardiogenic (ARDS)
Definition: Acute Respiratory Distress Syndrome, pulmonary oedema not primarily due to cardiac dysfunction.
Signs & Symptoms: Similar to cardiogenic pulmonary oedema, but without primary cardiac cause indicators.
EMT Treatment: Similar care to cardiogenic pulmonary oedema without NTG.
5. Spontaneous Pneumothorax
Definition: Presence of air in the pleural space, often in tall thin males, those with blebs, or COPD.
Signs & Symptoms: Sudden shortness of breath (SOB), unilateral decreased breath sounds, sub-Q emphysema.
EMT Treatment: O₂; PPV cautiously (watch for tension conversion); ALS for decompression. Contraindication to CPAP.
6. Hyperventilation Syndrome
Definition: Anxiety-triggered rapid breathing leading to hypocapnia.
Signs & Symptoms: Dizziness, carpopedal spasm due to cerebral vasoconstriction.
EMT Treatment: Coach breathing; remove stressor; O₂ only if hypoxic. No paper-bag rebreathing.
7. Upper-Airway & Misc.
Epiglottitis
Definition: Inflammation of the epiglottis, often caused by H. influenzae.
Signs & Symptoms: Sudden severe sore throat, drooling, tripod position, stridor.
EMT Treatment: Nothing in mouth, high-flow O₂, calm transport.
Croup (peds)
Definition: Viral infection causing upper airway swelling in children.
Signs & Symptoms: Barking cough, stridor.
EMT Treatment: Cool mist/O₂.
Pertussis
Definition: Whooping cough, a highly contagious bacterial infection.
Signs & Symptoms: Paroxysmal coughing, inspiratory “whoop”.
EMT Treatment: PPE, humidified O₂.
Cystic fibrosis
Definition: Genetic disorder causing production of thick, sticky mucus.
Signs & Symptoms: Thick mucus, recurrent infections.
EMT Treatment: Saline SVN, humidified O₂.
Poisonous inhalations
Definition: Exposure to toxic gases or fumes.
Signs & Symptoms: Varies by substance (e.g., carbon monoxide, hydrogen sulfide, cyanide).
EMT Treatment: Remove from source, high-flow O₂, consider specific treatments for CO/H₂S/cyanide.
Viral respiratory infection (URI/bronchiolitis/RSV/COVID-19)
Definition: Infections of the respiratory tract caused by various viruses.
Signs & Symptoms: Varies, but generally supportive, monitor for distress.
EMT Treatment: Supportive care, monitor for distress; O₂ titrated as needed.
Respiratory sounds can indicate specific conditions in different parts of the airway:
Upper Airway Sounds:
Wheeze:
Description: A high-pitched, whistling sound, most commonly heard during expiration, but can also be inspiratory if severe. It indicates narrowing of the lower airways.
Associated Conditions:
Asthma:
Definition: Triad of bronchospasm, oedema, and increased mucus.
Signs & Symptoms: Expiratory wheeze, prolonged expiration, pulsus paradoxus. Severity indicated by ability to speak, RR >30, HR >120, accessory muscle use. Status asthmaticus involves acute severe asthma unresponsive to SABA and O₂.
EMT Treatment: O₂ to ≥94%≥94%; coach controlled breathing; administer SABA (albuterol) via SVN (preferred); cautious CPAP; PPV at 10–12 min−1min−1 if failure (allow full exhalation).
Chronic Bronchitis:
Definition: Bronchiole inflammation, mucus, and scarring.
Signs & Symptoms: Productive cough ≥≥3 months/year for 2 years, coarse rhonchi/wheezes, cyanosis, JVD, pedal oedema.
EMT Treatment: Titrate O₂ 88–92% for chronic CO₂ retainers; cautious CPAP (5–10 cmH₂O) if moderate/severe distress; monitor for barotrauma.
Emphysema:
Definition: Alveolar wall destruction leading to decreased surface area and air-trapping.
Signs & Symptoms: Pink skin, thin build, barrel-chest, diminished breath sounds. While wheezing is possible, diminished sounds are more typical.
EMT Treatment: Titrate O₂ 88–92% for chronic CO₂ retainers; cautious CPAP (5–10 cmH₂O) if moderate/severe distress; monitor for barotrauma.
Cardiogenic Pulmonary Edema:
Definition: Left ventricular failure leading to increased hydrostatic pressure and fluid in the interstitium/alveoli.
Signs & Symptoms: May present with wheezing (cardiac asthma) in addition to crackles, frothy pink sputum, orthopnoea/PND, HTN, JVD, pedal oedema.
EMT Treatment: CPAP 5–10 cmH₂O; NTG per protocol; BVM if failure.
Rhonchi:
Description: Coarse, low-pitched rattling sounds that resemble snoring. They are typically caused by mucus or secretions in the larger lower airways (e.g., bronchi) and may clear or change after a cough or position change.
Associated Conditions:
Chronic Bronchitis:
Definition: Bronchiole inflammation, mucus, and scarring.
Signs & Symptoms: Productive cough ≥≥3 months/year for 2 years, coarse rhonchi/wheezes, cyanosis, JVD, pedal oedema.
EMT Treatment: Same care as emphysema; consider CPAP if indications met.
Pneumonia:
Definition: Infectious (bacterial/viral/aspiration) alveolar fluid/pus causing ventilation-perfusion mismatch.
Signs & Symptoms: Fever/chills, sharp pleuritic chest pain, unilateral crackles or rhonchi, splinting, possible ALOC in elderly.
EMT Treatment: O₂ to ≥94%≥94%; consider SABA if bronchospasm; transport.
Crackles (formerly rales):
Description: Short, popping, crackling sounds heard primarily on inspiration. They result from the opening of collapsed alveoli or fluid within the alveoli. Crackles often begin in the lung bases and typically do not clear with coughing.
Associated Conditions:
Cardiogenic Pulmonary Edema:
Definition: Left ventricular failure leading to increased hydrostatic pressure and fluid in the interstitium/alveoli.
Signs & Symptoms: Crackles, frothy pink sputum, orthopnoea/PND, HTN, JVD, pedal oedema.
EMT Treatment: CPAP 5–10 cmH₂O; NTG per protocol; BVM if failure.
Pneumonia:
Definition: Infectious (bacterial/viral/aspiration) alveolar fluid/pus causing ventilation-perfusion mismatch.
Signs & Symptoms: Fever/chills, sharp pleuritic chest pain, unilateral crackles or rhonchi, splinting, possible ALOC in elderly.
EMT Treatment: O₂ to ≥94%≥94%; consider SABA if bronchospasm; transport.
Non-cardiogenic (ARDS):
Definition: Acute Respiratory Distress Syndrome, pulmonary oedema not primarily due to cardiac dysfunction.
Signs & Symptoms: Similar to cardiogenic pulmonary oedema, but without primary cardiac cause indicators.
EMT Treatment: Similar care to cardiogenic pulmonary oedema without NTG.