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Cardiovascular System Overview
Rheumatic Heart Diseases
Definition of Rheumatic Fever
An immune-mediated inflammatory acute febrile illness following a pharyngeal infection (e.g., acute tonsillitis, scarlet fever) caused by beta-hemolytic group A streptococcus.
Post-Streptococcal Glomerulonephritis
Definition
A complication due to autoantibody enhancement by nephritogenic strains of group A streptococcal bacteria. Does not combine with the M protein antigen.
Common Sites Affected in Rheumatic Fever
Affected Endocardium Sites
Lines of contact between valve cusps when closed
Chordae tendineae
Clinical Complications
Fibrous union leads to stenosis, while thickened chordae cause valve incompetence (regurgitation). Repeated attacks can result in chronic heart failure.
Incidence of Cardiac Valve Involvement in RF Patients
Common Involvement Patterns
Mitral valve alone
Mitral and aortic valves simultaneously
Mitral, aortic, and tricuspid valves together
Rare: Aortic valve alone and very rare for tricuspid valve alone.
Morphology of Aschoff Bodies
Macroscopic
Scattered pale foci
Microscopic
Granulomatous lesions with eosinophilic hyaline fibrinous material encircled by lymphocytes, macrophages, and giant cells.
Clinical Importance
Pathognomonic for rheumatic myocarditis; they dissipate after the acute phase.
Heart Failure Types in Rheumatic Myocarditis
Type of Heart Failure
Acute heart failure
Explanation
Resulting from loss of myocardial function, leading to left ventricular dilation.
Heart Failure in Rheumatic Pericarditis
Type of Heart Failure
Acute heart failure
Explanation
Caused by serofibrinous effusion interfering with heart movement.
Other Body Parts Affected by Rheumatic Fever
Clinical Examples
Joints: Migratory polyarthritis with redness and swelling
Skin: Erythema marginatum, subcutaneous nodules
CNS: Mild encephalitis, Sydenham’s chorea (involuntary movements)
Important Serological Tests for RF
Tests
Antistreptolysin O (ASO) antibodies: Long half-life can cause false positives; should be <250 IU
DNAseB antibodies: May yield false negatives, not all Streptococcus A strains are positive.
Major and Minor Criteria for Clinical Diagnosis of RF
Major Criteria
Migratory polyarthritis
Myocarditis/pericarditis causing acute HF
Subcutaneous nodules
Erythema marginatum
Sydenham’s chorea
Minor Criteria
Fever (38.2–38.9 °C)
Arthralgia
Leukocytosis
Heart block on ECG
History of prior rheumatic fever
Atherosclerosis
Definition
Abnormal buildup of yellow plaques within the intima of arteries, consisting of fatty substances, cholesterol, cellular waste, leading to thickened and stiffened arterial walls.
Complications of Atherosclerosis
Stenosis causing impaired blood flow:
Angina (coronary)
Chronic ischemia (lower legs)
Ischemic bowel disease (mesenteric arteries)
Plaque rupture leading to thrombosis:
Myocardial infarction (coronary)
Stroke (middle cerebral artery)
Vessel weakening resulting in aneurysms (e.g., abdominal aorta).
Morphological Stages of Atheroma
Yellow Fatty Streaks: Lipid-laden macrophages forming macular yellow lesions.
Young Atheromatous Plaque: Lipid accumulation, smooth muscle cell proliferation, fibrous cap formation.
Mature Atheroma: Thick fibrous cap, lower rupture risk but causes lumen narrowing.
Risk Factors for Atherosclerosis
Age, Hyperlipidemia, Gender, Hypertension, Diabetes Mellitus, Genetic predisposition, Cigarette smoking, Stress, Obesity, Lack of exercise, Oral contraceptive use.
Definition and Complications of Aneurysm
Definition
Localized vascular dilatation due to vessel wall stretching.
Possible Complications
Rupture leading to massive bleeding
Mural thrombus leading to thrombotic embolism.
Types of Aneurysms
Atheromatous
Syphilitic
Infective
Cerebral (e.g., berry)
Cardiac
Capillary (e.g., diabetic retinopathy)
Benign Vascular Tumors
Hemangioma
Glomangioma
Multiple angiomatosis syndrome
Malignant Vascular Tumors
Hemangioendothelioma
Hemangiopericytoma
Angiosarcoma
Kaposi's sarcoma
Pulmonary Edema
Definition
Abnormal fluid accumulation in interstitium and alveoli of the lungs.
Clinical Classes
Cardiogenic and Non-Cardiogenic pulmonary edema.
Mechanism Classifications
Hydrostatic pressure edema
Permeability edema with/without diffuse alveolar damage (DAD)
Mixed pulmonary edema.
Causes of Pulmonary Edema
Hydrostatic Pressure PE
Left heart failure, Volume overload.
Permeability PE with DAD
ARDS due to toxins, inflammatory mediators, infectious processes.
Permeability PE without DAD
“Crack-lung”, High-altitude PE, Cytokine administration.
Mixed PE Causes
Severe neurologic injury, Abrupt lung re-expansion, Lung transplant.
Cardiogenic PE Causes
Left ventricular dysfunction, Valvular dysfunction, Rhythm disorders.
Paroxysmal Nocturnal Dyspnea in PE
Sudden breathlessness at night, relieved by sitting, due to increased pulmonary venous pressure when supine.
Orthopnea in PE
Breathlessness while lying flat, worsened pulmonary edema, similar to PND.
Crackles on Auscultation in PE
Fluid in alveoli and interstitial spaces causes crackles during air passage.
Ischemic Heart Disease Classification
Angina Pectoris, Myocardial Infarction, Chronic Ischemia with CHF, Sudden Death.
Definition of Angina Pectoris
Intermittent chest pain from reversible myocardial ischemia, characterized by severe pressing pain lasting 15 secs to 15 mins.
Stable Angina: Caused by fixed atherosclerotic obstruction, induced by oxygen demand increases, relieved by rest/nitroglycerin.
Variant Angina (Prinzmetal): Occurs at rest due to artery spasm, not related to exertion.
Unstable Angina: Severe chest pain at rest due to plaque disruption, potential for MI.
Incidence of Thrombotic Occlusion in IHD by Artery
Arteries
LAD: Anterior left ventricle (40-50%)
RCA: Posterior left ventricle (30-40%)
LCX: Lateral left ventricle (15-20%).
Factors Affecting Severity of Regional MI
Coronary occlusion characteristics, Duration, Blood size, Metabolic needs, Collateral flow, Myocardial response.
Typical Presentation of MI
Severe retrosternal pain, possible nausea, dyspnea, diaphoresis, weakness.
Silent MI: No symptoms, common in diabetes, elderly.
Acute Complications of MI
Sudden death, Arrhythmias, Cardiogenic shock, Ventricular aneurysm, Cardiac rupture.
Chronic MI Complications
CHF, Recurrent arrhythmias, Chronic ischemic heart disease.
Sub-Endocardium Zone MI
Severe ischemia in inner myocardium, often from partial occlusion or global hypoperfusion.
Important Investigations for MI Diagnosis
ECG: Q waves and ST changes
Serum Biomarkers: Troponins, CK-MB.
Heart Failure Definition
HF is the heart's inability to meet blood flow demands, resulting from low output or exceeding body's supply.
Pathological Types of Heart Failure
Low Output HF (congestive)
High Output HF.
Renin System and Congestion in Congestive HF
Decreased renal perfusion activates Renin-Angiotensin system, leading to fluid retention and pressure increase.
Cardiac Changes Secondary to Increased Afterload Pressure
LVH develops initially, leading to ventricular dilation, reduced compliance, and diastolic dysfunction over time.
Left Ventricular Ejection Fraction (LVEF)
The percentage of blood ejected during systole; indicative of systolic function, reduced in systolic HF.
Causes of Left and Right Systolic Congestive HF
Left: MI, Myocarditis, Dilated cardiomyopathy, Hypertension.
Right: Pulmonary hypertension, PE, Myocardial infarction.
Causes of Diastolic Congestive HF
Left: Pericardial diseases, Hypertrophy, Restrictive cardiomyopathies.
Right: Tricuspid stenosis.
Tachypnea, Palpitation, and Gallop Rhythm in HF
Tachypnea: Increased breathing from low cardiac output.
Palpitations: Awareness of abnormal heartbeats from compensatory tachycardia.
Gallop Rhythm: Third heart sound due to rapid filling from increased atrial pressure.
Clinical Features of Right-Side Congestive HF
Jugular venous distension, Hepatomegaly, Lower limb edema, Ascites.
Causes of High Output HF
Severe anemia, Uncontrolled thyrotoxicosis, Beriberi (Vitamin B1 deficiency), Arteriovenous fistula.
Investigating Methods of HF
Chest X-ray: Detects cardiomegaly, pulmonary congestion.
ECG: Identifies MIs and arrhythmias.
Echocardiography: Determines type, etiology, LVEF.
Blood Tests: Evaluates cardiac enzymes and other dysfunctions.
Cardiomyopathies
Definition
Heterogeneous myocardial dysfunctions, often with genetic bases.
Main Classifications:
Dilated Cardiomyopathy (Most common), Hypertrophic Cardiomyopathy, Restrictive Cardiomyopathy (Least common).
Pathological Effect of Dilated Cardiomyopathy (DCM)
Thin, stretched heart walls lead to systolic failure.
Problems Related to DCM
Mural thrombi, Secondary mitral regurgitation, Arrhythmias.
Etiology of DCM
Idiopathic, Genetic mutations, Viral myocarditis, Alcohol abuse, Medications, Pregnancy.
Morphological Changes in Hypertrophic Cardiomyopathy (HCM)
Massive hypertrophy of LV and interventricular septum, thick-walled, hypercontracting heart.
Problematic Issues Related to HCM
Mitral regurgitation (asymmetric hypertrophy), Functional aortic stenosis (subaortic hypertrophy).
Microscopic Features of HCM
Hypertrophied myocytes with interstitial fibrosis.
Pathogenesis of HCM
Autosomal dominant disorder caused by mutations in sarcomere protein genes leading to hypercontractility and hypertrophy.
Syncope and Sudden Death in Athletes with HCM
Syncope: Reduced output due to obstruction.
Sudden Death: Caused by ventricular arrhythmias.
Morphological Changes in Restrictive Cardiomyopathy
Ventricular wall rigidity disrupts diastolic filling, potentially affecting systolic function.
Etiology of Restrictive Cardiomyopathy
Infiltrative and systemic diseases (e.g., amyloidosis, hemochromatosis).
Infective Endocarditis (IE) Overview
Etiology
Commonly caused by gram-positive bacteria, particularly Streptococcus viridans, and occurs after transient bacteremia.
Predisposing Factors
Rheumatic valvular diseases, congenital lesions, prosthetic valves, immunosuppression, poor dental hygiene.
Pathological Changes in IE
Causes friable vegetations on valves, leading to necrosis, scarring, and potential embolization.
Differences Between Acute and Sub-Acute IE
Acute IE: Fast, severe course, high mortality (>50%). Caused by virulent bacteria.
Sub-Acute IE: Longer, less severe, caused by less virulent bacteria.
Microscopic Features of IE Vegetations
Composed of fibrin, platelets, microorganisms; underlying valves show necrosis.
Hematuria and Other Symptoms in IE
Hematuria: Renal infarcts or glomerulonephritis due to embolization.
Petechiae and Splinter Hemorrhages: Embolic phenomena from angitis causing skin and mucosal lesions.
Main Complications of IE
Acute heart failure, organ embolization, renal failure, chronic heart failure.
Myocarditis Causes
Viral, bacterial, parasitic infections, and non-infectious causes (e.g., rheumatic fever).
Gross and Microscopic Changes in Myocarditis
Flabby heart, four-chamber dilatation, patchy hemorrhage, interstitial infiltrates depending on the infective agent.
Local and Systemic Causes of Pericarditis
Local: MI, Surgery, Trauma, Radiation.
Systemic: Uremia, autoimmune diseases, infections.
Types of Acute and Chronic Pericarditis
Acute Types: Serous, fibrinous, purulent, hemorrhagic.
Chronic Types: Adhesive, constrictive, caseous.
Inflammatory Vascular Disorders
Local Complications: Thrombosis, rupture, aneurysm formation, chronic ischemia.
Infectious Vasculitis Examples
Localized arteritis, infected emboli, meningococcal vasculitis, syphilitic arteritis.
Thrombophlebitis and Phlebothrombosis
Thrombophlebitis: Inflammation with thrombosis.
Phlebothrombosis: Thrombosis without inflammation, migrans indicates cancer.
Immune-Mediated Vasculitis Examples
Polyarteritis nodosa, cranial arteritis, Buerger’s disease, Kawasaki disease.
Upper Respiratory Tract Disorders
Rhinitis: Inflammation of nasal mucosa, types include acute and chronic variants.
Acute Catarrhal Rhinitis
Caused by viral infections, predisposed by cold; secondary bacterial infections possible.
Chronic Non-specific Rhinitis Causes
Nasal obstruction due to various factors, chronic sinusitis, occupational exposure.
Rhinoscleroma
Granulomatous inflammation caused by Klebsiella; complications include nasal obstruction and infections.
Types of Sinusitis
Acute and chronic types; inflammation of sinus mucosa.
Nasal Polyps and Fungal Sinusitis Types
Edematous mucosal projections, fungal types can be invasive or noninvasive.
Epistaxis Causes
Nosebleeds caused by trauma and various local factors.
Tumors in Nose and Sinuses
Benign: Papillomas, angioma, osteoma
Malignant: Carcinomas, sarcomas.
Acute Tonsillitis Overview
Causes: Viral (common), bacterial; prevalent in ages 5-15.
Types of Acute Tonsillitis
Forms include catarrhal, follicular, and membranous.
Complications of Acute Tonsillitis
May lead to peritonsillar abscess, infections spreading, and systemic effects.
Chronic Non-Specific Tonsillitis Features
Hyperplasia of lymphoid tissues, chronic inflammation, and potential late-stage fibrosis.
Diphtheria Overview
Caused by Corynebacterium, leading to pseudomembranous inflammation and severe complications.
Nasopharyngeal Carcinoma
Linked to EBV, dietary factors, characterized by diverse clinical features.
Chronic Nonspecific Laryngitis and Singer's Nodes
Results from voice strain; nodes develop on vocal cords due to chronic stress.
Carcinoma of the Larynx
Subtypes include supraglottis, glottis, subglottis; early detection is crucial for prognosis.
Pneumonia Overview
Definition: Infectious inflammation of lung tissue causing pus- or liquid-filled air sacs.
Pneumonia Classifications
Based on anatomical site, causative agent, clinical course, and transmission route.
Complications of Pneumonia
Include abscess formation, pleural effusion, ARDS, respiratory failure.
Morphology of Bronchopneumonia
Patchy consolidation in lung lobes, often bilateral, with elevated lesions.
Lobar Pneumonia Characteristics
Involves entire lobes; primarily caused by Streptococcus pneumoniae.
Etiology of Atypical Pneumonia
Caused by intracellular organisms like Mycoplasma; morphological changes differ from typical pneumonia.
SARS Overview
Caused by a coronavirus, leading to pulmonary damage and ARDS.
Aspiration Pneumonia Definition
Results from inhalation of contaminated material leading to inflammation and infection.
Lung Abscess Definition
Suppurative necrosis in lung tissue with potential for formation through various mechanisms.
Chronic Pneumonias Examples
Histoplasmosis, blastomycosis, coccidiomycosis.
Bronchial Asthma Definition
Chronic inflammatory airway disorder characterized by episodic dyspnea and cough, often with reversible obstruction.
Main Mechanisms of Asthma
Include airway hyper-responsiveness, bronchospasm, smooth muscle hypertrophy, and excessive mucus secretion.
Classification of Asthma
Atopic: IgE driven, more common in children; Non-allergic: In adults, triggered by external factors.
Inflammatory Cells in Asthma
Mast cells, eosinophils, lymphocytes, and other immune cells contribute to airway inflammation.
Status Asthmaticus Definition
Severe asthma attack unresponsive to treatment, requiring intensive intervention.
Sputum Cytology in Asthma Diagnosis
Reveals eosinophils, Curschmann's spirals, and Charcot-Leyden crystals indicative of inflammatory activity.
Emphysema Definition
Permanent airway dilatation due to lung tissue damage, resulting in fewer but larger air pockets.
Classification of Emphysema
Centri-acinar: Associated with smoking, Pan-acinar: Linked to alpha-1 antitrypsin deficiency, Distal: Near pleura but less obstructive.
Complications of Emphysema
Include respiratory failure and pneumothorax among others.
Pathogenesis of Emphysema
Imbalance of protease-antiprotease activity leading to alveolar damage.
Morphological Features of Emphysema
Hyper-distended lungs with visible enlarged air spaces; damaged alveolar structures.
Chronic Bronchitis Definition
Persistent bronchial inflammation indicated by a chronic productive cough.
Pathogenesis of Chronic Bronchitis
Caused by inhalation of irritants leading to mucus hypersecretion and secondary infections.
Morphological Features of Chronic Bronchitis
Edematous membranes, mucous casts; chronic inflammatory cell infiltration.
Bronchiectasis Definition
Long-term abnormal airway dilation due to chronic inflammation and mucus accumulation.
Causes of Bronchiectasis
Result from necrotizing infections, bronchial obstructions, or hereditary conditions like cystic fibrosis.
Pulmonary Embolism Types
Includes saddle embolus, occlusions in major pulmonary arteries, and multiple small emboli.
Saddle Embolus Definition
Large thrombus at the bifurcation, causing severe chest pain, dyspnea, and rapid death risk.
Multiple Small Pulmonary Emboli Details
Can lead to acute or chronic respiratory issues or pulmonary arterial hypertension.
Inflammatory Disorders of Pulmonary Vessels
Includes Goodpasture’s syndrome and Wegener’s granulomatosis, both involve severe vascular inflammation.
Cor Pulmonale Definition
Right heart failure stemming from pulmonary hypertension, leading to structural and functional heart changes.
Pulmonary Hypertension Classification
Pre-capillary, Capillary, Post-capillary, each with distinct causes.
Types of Pleural Effusion
Hydrothorax: Due to pressure changes, Hemothorax: Blood leakage, Chylothorax: Lymph fluid accumulation, Inflammatory Effusion: Infection-related.
Empyema Definition
Collection of pus in thoracic cavity due to infection.
Pneumothorax Definition
Presence of air in pleural space; can be traumatic or spontaneous.
Mesothelioma Definition
Rare malignant tumor linked with asbestos exposure; aggressive with poor prognosis.