Applied Microbiology week 8: Acellular Microbes, Viruses and Prions
Learning Objectives
Describe basic structures: viruses, viroids, satellites, prions.
Understand virus classification.
Describe 5 steps of all viral life cycles related to infection and disease.
Differentiate between lysogenic and lytic cycles.
Explain virus diagnosis methods.
Describe prion infection mechanisms and related diseases.
Acellular Agents
Viruses: Nucleic acid (DNA/RNA) + protein coat (capsid). Some have lipid envelopes.
Viroids: Circular RNA molecules with no protein coats; infect plants.
Prions: Misfolded proteins without nucleic acids; propagate by inducing misfolding in normal proteins.
Satellites: Incomplete viral agents needing helper viruses for replication.
Virus Characteristics
Submicroscopic, obligate intracellular parasites.
Infect various hosts: animals, plants, bacteria (bacteriophages).
Exhibit some characteristics of life but lack independent metabolism and cellular structure.
Virus Significance
Major cause of infectious diseases in humans/animals.
Emerging viruses pose ongoing threats.
Serve therapeutic applications (gene therapy, cancer targeting).
Critical ecological roles in nutrient cycling and horizontal gene transfer.
Virus Taxonomy
Family: (-viridae) based on genome type, structure, replication strategy.
Genus: (-virus) more specific group within a family.
Species: common name for the individual virus.
Subtypes/strains: variants within a species.
Viral Structure
Nucleocapsid: Core structure with nucleic acid and capsid.
Capsid: Protects genome, formed from protein subunits called capsomeres.
Envelope: Lipid membrane from host cell, aids in attachment and entry.
Viral Life Cycle
Attachment: Virus binds to specific host cell receptors.
Entry: Membrane fusion/endocytosis; uncoating releases nucleic acid.
Biosynthesis: Host machinery produces viral components.
Assembly: New virions develop from genome and proteins.
Release: Through cell lysis (non-enveloped) or budding (enveloped).
Infections by Bacteriophages
Virulent (Lytic) Phages: Rapidly lyse host cells.
Temperate Phages: Can replicate lytically or enter dormant lysogenic state.
Prions and Related Diseases
Arise from misfolded normal proteins, leading to neurodegenerative diseases.
Examples: BSE, Scrapie, Creutzfeldt–Jakob disease.
Propagation via misfolded proteins inducing normal proteins to misfold, leading to brain damage and dysfunction.