Dematiceous Molds
Introduction to Dematiaceous Fungi
The lecture focuses on melanized molds, also referred to as dematiaceous fungi.
Characteristics of dematiaceous fungi:
Produce dark pigmentation due to melanin in their cell walls.
Melanin is a pigment also present in human skin, contributing to skin color and offering protection from UV radiation.
Increased melanin results in darker skin, while reduced melanin results in lighter skin.
Melanin provides protection against host immune defenses and antifungal agents.
Morphological Characteristics
Dematiaceous molds are morphologically characterized by:
Dark brown to black color due to cell wall melanin.
Observed both macroscopically on cultures and microscopically through hyphae and conidia under a microscope.
This pigmentation acts as a virulence factor, assisting in resistance to oxidative killing by the host.
Growth Rates
The growth rates of dematiaceous molds are relevant in clinical contexts:
Molds are categorized into:
Slow Growers: Require 7-10 days or longer to grow.
Typically associated with chronic or subcutaneous infections (e.g., chromoblastomycosis, mycetoma).
Rapid Growers: Grow in less than 7 days.
Often opportunistic or contaminants, associated with sinonasal infections especially in immunocompromised patients.
Infections primarily occur in immunocompromised patients, not commonly seen in healthy individuals.
Important Diseases Caused by Dematiaceous Fungi
Three major disease terms associated with these fungi are:
Pheohyphomycosis: Presence of pigmented hyphae in infected tissue.
Chromoblastomycosis: Chronic skin infections with characteristic sclerotic bodies.
Mycetoma: Granulomatous infections with grains in pus.
Superficial Infections
Tinea Nigra:
Causative organism: Hortaea werneckii.
Often mistaken for melanoma or moles; manifests as a dark brown-black mole that is not raised.
Typically found on palms or soles of feet, can be scraped off revealing pigmented hyphae on KOH prep.
More commonly seen in tropical/subtropical climates.
Black Piedra:
Causative organism: Piedraia hortae.
Occurs in long untreated hair and wraps around hair shafts, creating gritty nodules; may feel like sand.
Common in regions like South America, Asia, and tropical areas.
Treatment options include topical antifungals or shaving the hair off.
Mycetoma
Characteristics:
Chronic granulomatous infections, primarily occurring on feet or legs.
Caused by traumatic implantation of environmental organisms through soil, thorns, etc.
Symptoms include immense swelling, purple discoloration, small wounds leading to chronic infections.
Diagnostic feature: Multiple sinuses draining pus containing grains, which are clumps of the causative agent.
Types of Mycetoma:
Actinomycetomas: Form yellow or white grains, usually caused by bacterial agents (e.g., Nocardia, Streptomyces).
Eumycetomas: Cause black or white grains and are primarily caused by dematiaceous fungi.
Organisms associated include:
White grains: can be from both bacteria and fungi (e.g., Fusarium, Acremonium).
Black grains: Almost always fungi, specifically dematiaceous molds; includes genera such as Curvularia, Madurella.
Chromoblastomycosis
Caused by melanized molds entering through trauma (thorns, splinters).
Common in rural warm climates like South America, Africa, Southeast Asia.
Often unnoticed initially; lesions grow and can be mistaken for warts or calluses.
Lesions develop into cauliflower-like crusted growths with black dots representing emerging fungal structures.
Histological hallmark: Sclerotic bodies (Medlar bodies), round, thick-walled structures that exhibit divisions in multiple planes, visible under microscopy.
Common causative organisms include Fonsecaea pedrosoi, Phialophora, and Cladophialophora.
Pheohyphomycosis
A catch-all term for infections caused by different types of melanized fungi, characterized histologically by pigmented septate hyphae.
May present with yeast-like forms; absence of sclerotic bodies or grains.
Common in immunocompromised patients but also seen in cases of trauma in immunocompetent individuals.
Notable serious cases include cerebral pheohyphomycosis.
Clinically Relevant Organisms
Pseudalsharia boydii:
Also known as Scedosporium boydii.
Associated with white grain mycetoma and emerging opportunist in immunocompromised patients.
Can lead to sino-pulmonary diseases, brain abscesses, or cutaneous infections.
Rapid grower (5-10 days) and changes from white to gray/brown with age.
Microscopically shows oval conidia in clusters or singly.
Acremonium:
Often viewed as a lab contaminant but can cause severe infections in patients with central lines/neutropenia.
Known to cause white grain mycetoma.
Forms powdery/velvety colonies, colors may vary.
Long slender conidia with irregular shapes.
Cladophialophora & Phialophora:
Both are slow growers, classic agents of chromoblastomycosis.
Diagnosis based on the presence of sclerotic bodies.
Cladophialophora: Forms long chains of smooth oval conidia without branching.
Phialophora: Distinctive flask-shaped conidiophores with a collarette.
Funsecchia:
The most common cause of chromoblastomycosis worldwide.
Exhibits a unique conidiation pattern, grows slowly, and produces dark colonies that can appear olive or black.
Alternaria:
Common environmental mold and recognized allergen.
Can cause opportunistic infections: sinusitis, cutaneous infections.
Fast-growing and exhibits suede-like texture and dark coloration.
Microscopically shows large, muriform conidia with transverse and longitudinal septa.
Bipolaris:
Clinically significant; can cause fungal sinusitis and subcutaneous infections.
Rapid-growing and morphologically dark.
Conidia are distoseptate with scythe-like or curved shapes, capable of germ tube formation at both ends.
Curvularia:
Recognizable by the dramatic curvature of its conidia.
Associated with fungal sinusitis or skin/nail infections.
Colonies are suede-like, while conidia display significant curvature resembling boomerangs.
Conclusion
Dematiaceous fungi represent a diverse group of pathogens primarily affecting immunocompromised individuals but can also infect healthy hosts through trauma.
Understanding the morphological traits, growth rates, and disease implications of these fungi is crucial for accurate diagnoses and effective treatments in a clinical setting.