Cervical Lesions: Carious and Non-Carious, Erosion, Abrasion, Treatment, and Sensitivity
DCD - Dr. Cube Dentistry
Fourth Stage: Operative
Cervical Lesions (Carious and Non-Carious)
Presented by: Dr. Meelad Joffery
Edition: 2025
Introduction to Cervical Lesions
Definition: Cervical lesions, also known as class V lesions, comprise both carious and non-carious defects located in the gingival third of the facial and lingual surfaces of teeth.
Class V Carious Lesions:
Result from bacterial plaque adherence to tooth surfaces.
Produce acids leading to demineralization of the enamel and dentin.
Non-Carious Cervical Lesions (NCCLs):
Develop from factors other than dental caries.
Increasingly recognized for their complex multifactorial etiology including stress, friction, and biocorrosion.
Detection of Cervical Lesions
Visual Detection
Class V Enamel Lesions:
Easily detected due to change in color seen visually.
Limitation of Color as a Predictor:
Tooth color does not reliably indicate the extent of root caries damage.
A discolored root surface can still have a hard sclerotic surface that does not require treatment unless acidity causes patient issues.
Conversely, root caries may appear healthy yet be soft when assessed by dental tools.
Challenges with Detection
Caries Disclosing Dyes:
May yield inconsistent results in identifying demineralized cementum and dentin on root surfaces.
Best Clinical Indicator:
The softness of the tooth surface tested with a dental instrument is currently the best indicator of carious lesions on root surfaces.
Non-Carious Cervical Lesions (NCCLs)
Prevalence:
Reported prevalence ranges from 5% to 85% in contemporary populations.
Most common in premolars and molars; severity increases with age.
Erosion
Definition: Loss of tooth structure resulting from chemical actions.
Common Causes:
Frequent consumption of acidic foods and beverages.
Activities such as swishing acidic drinks or exposure to gastric acids from gastrointestinal issues.
Early Stage Presentation:
Smooth, silk-like appearance of enamel surfaces.
Lesions occur coronal to CEJ with intact enamel along the gingival margin.
Intermediate Stage Erosion
Characteristics:
Development of shallow flat concavities with rounded edges.
Possible complete enamel removal resulting in dentin exposure.
Rounding of cusps and incisal edges noticeable.
Advanced Erosion
Biological Response:
Reactionary and reparative dentin formation may occur in response to continued loss of dentin, ultimately affecting dentinal tubules.
Factors Accelerating Dental Chemical Degradation
Box 5-2: Habits to Avoid
Avoid Frequent Intake of Acids:
Swishing acidic drinks before swallowing.
Consuming acidic beverages before sleep; reduces protective saliva effects.
Post-Acid Brushing:
Brushing immediately after acid exposure increases enamel wear due to abrasiveness of toothpaste on softened enamel.
Abrasion
Definition: Loss of tooth structure by mechanical or frictional forces.
Common Causes:
Excessive tooth brushing.
Damage from dental floss, toothpicks, or removable dental appliances.
Visual Presentation:
Abrasion lesions typically appear as V-shaped notches.
Factor Influencing Abrasion:
Brushing technique
Type of toothbrush
Abrasiveness of toothpaste
Duration and intensity of brushing
Abfraction
Definition: Loss of tooth structure due to flexural forces.
Theory:
Proposed to explain the development of V-shaped notches.
Stresses from occlusal loading are transmitted to the cervical area, causing fractures of cervical enamel rods.
Associated Conditions:
Most often seen in patients who exert heavy occlusal forces or exhibit signs of bruxism.
Access and Isolation for Treatment
Access Challenges
Supragingival vs. Subgingival Cervical Lesions:
Supragingival lesions are easier for access and treatment.
Subgingival lesions require careful isolation to remove carious material adequately and to prepare and finish restorations effectively.
Failure to obtain full access may result in retention of carious structure and, subsequently, restoration failure.
Isolation Techniques
Methods Used:
Cotton rolls, retraction cords for adequate access in supragingival lesions.
Rubber dams are preferable for subgingival lesions for better visibility and access.
Surgical Approach:
If rubber dam does not provide adequate access, may resort to gingivoplasty.
Surgical approaches should ideally occur 6 weeks before restorative treatment for optimal healing.
Treatment of Cervical Lesions
Active Caries
Required Action: Initiate treatment to control active disease and prevent further progression.
Non-Carious Cervical Lesions
Treatment Indications:
Protect remaining tooth structure if significant tooth loss exists.
Address cosmetic concerns if aesthetics are compromised.
Lower sensitivity issues.
Facilitate design for removable partial dentures.
Asymptomatic Non-Carious Lesions
Preferred Management: Prophylaxis through various measures:
Tooth wear management
Thorough diagnosis and causal therapy
Prophylactic adjustments in potential causative factors
Use of fluoride
Documenting and following up treatments
Restorative Procedures
Decision-Making for Restoration
Cavity Preparation:
Determine extent of restoration based on lesion size.
Remove all demineralized and unsupported enamel.
Materials Used in Class V Restoration
Composite Materials:
Resin composite (with dentin bonding systems)
Flowable resin composite
Composite with glass-ionomer base (sandwich technique)
Glass ionomer
Resin-modified glass ionomer
Compomer
Porcelain inlay (less common)
Non-Esthetic Materials:
Amalgam
Direct gold foil (less common)
Gold inlay (less common)
Esthetic Advancements:
Glass-ionomer to fill dentin, reduce microleakage, and improve retention.
Composite resin to enhance aesthetics and polishability.
Sandwich Technique (Laminated Technique)
Purpose: Combine advantages of glass ionomer cement and composite resin.
Techniques:
Open Technique: Glass ionomer at gingival margin is exposed.
Closed Technique: Glass ionomer entirely covered by resin composite.
Function: GIC serves as an intermediate layer between dentin and resin composite.
Dentinal Sensitivity
Association: Often related to gingival recession and NCCLs.
Cause of Sensitivity: Exposure of dentinal tubules transmits signals to the pulp, resulting in pain.
Influential Factors: Number and size of open tubules; fluid movement within the tubules changing in response to stimuli.
Treatment Options
Surgical Treatments:
Root coverage techniques like connective tissue grafts for gingival recessions.
Desensitizing Agents:
Stannous fluorides show promising results.
Potassium nitrate found in dentifrices or gels reduces sensory nerve activity rather than occluding tubules.
Other Treatment Options
Occlusion of Tubules:
Dentin adhesives provide temporary relief.
Oxalate solutions used alone or with electrophoresis show success in occlusion.
Conclusion
Final Note: If a practitioner knows the current condition and desired outcomes, executing effective treatment plans becomes more manageable, as underscored by the emphasis on informed clinical practices.
Acknowledgments: Presented by Dr. Cube Dentistry, a key educational platform in Iraq for medical students.