GENE221_LECTURES
Course Overview
Course: GENE221 2025 Molecular and Microbial Genetics
Instructor: Associate Professor Htin Lin Aung
Department: Microbiology and Immunology
Email: htin.aung@otago.ac.nz
Lecture Objectives
Understand the differences between:
Complementation
Recombination
Learn molecular mechanisms of recombination
Historical Context
1950s: Key developments in genetics
Key Researcher: Seymour Benzer
Experimental System
Model Organism: Bacteriophage T4
**Process:
Uninfected cell
Adsorption of phage to host cell
Entry of phage nucleic acid
Assembly of phages
Lysis of host cell
Rationale for Using Bacteriophage
Advantages:
Easily and rapidly grown
Genetically simple organisms
Probes for genetic mechanisms
Large-scale analyses possible
Bacteriophage Plaques
Clear areas in a bacterial lawn
Indicate bacteriophage activity
Opaque: Lawn of bacteria
Clear: Plaques
rII Genes of Bacteriophage T4
Wild-type T4 can infect two E. coli strains: K and B
rII Mutants:
Large plaques on strain B
No plaques on strain K
Complementation
Definition: Wild-type phenotype from two different mutant genomes
Benzer's experiment with rII mutants in E. coli K12
Outcome:
Complementation occurs when mutations are in different genes
No complementation if mutations are in the same gene
Recombination Analysis
Definition: Generates new gene combinations
Details of Benzer's recombination analysis
Infect E. coli B: Pairs of mutants used for recombination
Testing Recombination
Progeny phage infection on E. coli K
Wild-type can grow; mutant cannot
Molecular Mechanisms of Recombination
Key Features:
Exchange of genetic material between chromosomes
Involves breakage and rejoining of DNA
Holliday Junction Model: Proposed by Robin Holliday
Supports available genetic information with refinements
Key Concepts of Holliday Model
Alignment of DNA sequences
Breakage and rejoining of DNA
Branch Migration: Creates heteroduplex DNA
Resolution: Leaves DNA molecules with new allele combinations
Recombination in E. coli
Key Enzymes Involved:
RecBCD: Nicks, unwinds, degrades DNA
RecA: Catalyzes base-pairing with target DNA
RuvA, RuvB: Cause branch migration
RuvC: Resolves Holliday junctions
Summary of Key Takeaways
Complementation indicates if mutations are in the same or different genes
Recombination results in genetically different progeny
Lecture Road Map
Lecture 1: One-gene-one-enzyme hypothesis
Lecture 2: Spontaneous mutations and mutagens
Lecture 3: Mechanisms of spontaneous mutations
Lecture 4: Mechanisms of mutagens
Lecture 5: Complementation and Recombination
Contact Information
Building: Microbiology Building
Email: htin.aung@otago.ac.nz