EMG and Muscle Contraction
LAB 7: EMG AND MUSCLE CONTRACTION
TYPES OF MUSCLE TISSUE
Cardiac Muscle
Features:
1-2 nuclei in fibers.
Fibers are physically and electrically connected to each other.
Striated appearance.
Involuntary control.
Smooth Muscle
Features:
Single nucleus in fibers.
Non-striated appearance.
Involuntary control.
Skeletal Muscle
Features:
Multinucleated fibers.
Striated appearance.
Voluntary control.
HISTOLOGY OF SKELETAL MUSCLE
Longitudinal Section of Skeletal Muscle at 400x
Distinct striations should be visible.
Transverse Section of Skeletal Muscle at 400x
Peripheral Nuclei:
Numerous peripheral nuclei are visible (indicated by arrows).
Skeletal muscle cells possess hundreds of nuclei squeezed between the outer cell membrane (sarcolemma) and myofibrils (bundles of muscle cell proteins).
Example tissue from the tongue shows skeletal muscle cells cut in longitudinal and cross-section.
Peripheral location of nuclei is evident in cross-sectioned cells.
SKELETAL MUSCLE FIBER CHARACTERISTICS
Unique Properties of Skeletal Muscle Cells (Fibers):
Size: Much larger than other body cells.
Nuclei: Multinucleate (typically hundreds of nuclei).
Appearance: Striated (banded).
Components:
Sarcolemma = plasma membrane.
Sarcoplasm = cytoplasm containing myofibrils.
MYOFIBRIL FUNCTION
Role in Muscle Contraction:
Myofibrils cause muscle contraction through the process of shortening.
Structure: Myofibrils consist of bundles of protein filaments called myofilaments, organized into functional units known as sarcomeres.
SARCOMERE STRUCTURE
Definition:
A sarcomere is defined as the region between one Z-line and the next Z-line.
Components:
Thin filaments (actin) occupy the I band.
Thick filaments (myosin) occupy the A band.
Functional Unit:
The sarcomere is the functional unit of a skeletal muscle fiber.
SLIDING FILAMENT THEORY
Mechanism of Contraction:
During a contraction:
The A band remains the same width.
Z-lines move closer together.
I band decreases in width.
When myofibril ends are free to move, the sarcomeres shorten together, pulling towards the center of the myofibril.
MOTOR UNIT
Definition:
A motor unit consists of a motor neuron and the muscle fibers it innervates.
Muscle fibers are dispersed throughout the muscle.
Recruitment Process:
Small motor units are recruited first in response to weaker stimuli.
The force of muscle contraction is determined by the number of motor units activated and the force generated by individual muscle fibers.
EXCITATION-CONTRACTION COUPLING
Process Overview:
Excitation:
Generation of an action potential in the sarcolemma by the motor neuron.
Contraction Triggering:
Calcium release from the sarcoplasmic reticulum is triggered by the action potential, leading to muscle contraction.
CALCIUM AND MUSCLE CONTRACTION
**Calcium Role:
Calcium ions bind to troponin, causing a conformational change in troponin.
This change moves tropomyosin away from active sites on thin filaments (actin).
Myosin heads can then bind to these active sites, forming cross-bridges.
CROSS-BRIDGE CYCLE
Key Events in the Cycle:
Cross-Bridge Formation:
Myosin heads bind to actin to form a cross-bridge.
Power Stroke:
The splitting of ATP causes the myosin head to change shape and pull against actin.
Cross-Bridge Detachment:
A new ATP molecule binds to myosin, causing it to detach from actin.
Myosin Reactivation:
ATP is hydrolyzed, resetting the myosin to its high-energy state for another cycle.
MUSCLE TWITCH PHASES
Phases Defined:
Latent Period:
Occurs between the action potential in the muscle fiber and the onset of contraction.
Contraction Period:
Begins when the muscle generates force and continues until peak tension is reached.
Relaxation Period:
The longest phase during which Ca extsuperscript{2+} reuptake into the sarcoplasmic reticulum (ATP-dependent) exceeds its release.
Tension diminishes until the fiber returns to its resting state.
SUMMATION AND TETANUS
Concept of Recruitment:
Recruitment increases due to frequency of stimulation, known as summation.
If a second stimulus is applied before relaxation from the first, greater force production occurs due to residual calcium ions in the cytoplasm.
Incomplete Tetanus:
Individual twitches are observable, but tension is higher than a single twitch with frequent stimulation.
Complete Tetanus:
No relaxation occurs, leading to maximum tension.
TYPES OF MUSCLE FIBERS
Fast Glycolytic Fibers:
Last to be recruited.
Break down glucose anaerobically.
Generate the most force but fatigue quickly.
Fast cycling due to fast ATPase activity.
Fast Oxidative Fibers:
Second to be recruited.
Use oxygen to aerobically break down glucose, generating more ATP.
Most resistant to fatigue.
Fast cycling due to fast ATPase activity.
Slow Oxidative Fibers:
First to be recruited.
Use oxygen to aerobically break down glucose, generating more ATP.
Most enduring fibers, but generate the least force.
Slow cycling due to slow ATPase activity.
MUSCLE FATIGUE
Definition:
A decrease in tension production following frequent stimulation.
Potential Causes Include:
Depletion of calcium.
Depletion of ATP.
Depletion of electrolytes (e.g., sodium).
Loss of neuronal signaling.
Build-up of metabolites disrupting signaling.
High-Intensity Activities:
Recruit fast glycolytic fibers, which rely on glycolysis for ATP, producing lactic acid that alters pH and disrupts contractile function.
Neuromuscular Fatigue:
Occurs when somatic motor neurons deplete their neurotransmitter supply due to intense exercise.
Low-Intensity, Long-Duration Exercises:
Primarily fatigue slow oxidative and fast oxidative fibers due to fuel source depletion.
ELECTROMYOGRAPHY (EMG)
Definition:
EMG measures electrical activity of muscles.
A healthy muscle will exhibit no electrical activity during rest.
Contractile Response:
As muscle contraction increases in force, more muscle fibers are activated, resulting in increased action potentials.
The change in mV (ΔV) increases with increased motor unit activation.
MUSCLE FATIGUE AND CENTRAL DRIVE
Definition of Central Drive:
Central drive refers to the neural signal from the brain (central nervous system) to the muscles.
Counteracting Fatigue:
The brain can increase central drive through:
Increased Motor Unit Recruitment: Activating more motor units to generate increased force.
Increased Firing Rate: More frequent signals sent to the muscles from the brain.