Clinical outcomes and emergence of resistance of Pseudomonas aeruginosa infections treated with ceftolozane-tazobactam versus ceftazidime-avibactam
Abstract
Few studies compare outcomes of patients with difficult-to-treat resistant (DTR) Pseudomonas aeruginosa infections treated with ceftolozane-tazobactam versus ceftazidime-avibactam.
A multicenter prospective study was conducted from 2018 to 2023.
Patients received >72 hours of ceftolozane-tazobactam or ceftazidime-avibactam, confirmed susceptible by broth microdilution.
Inverse probability weighting (IPW) was used for balanced baseline characteristics.
The study assessed 30-day mortality and emergence of resistance (≥4-fold increase in MIC).
Among 186 patients, 102 (55%) received ceftolozane-tazobactam, and 84 (45%) received ceftazidime-avibactam.
Thirty-day mortality was similar between groups (21% vs. 17%).
Emergence of resistance was higher with ceftolozane-tazobactam (38% vs. 25%) but did not reach statistical significance.
Durations of longer treatment and use of CRRT were linked to increased emergence of resistance.
Both agents appear equally effective for survival, but ceftolozane-tazobactam may have a greater concern for resistance emergence.
Introduction to Pseudomonas aeruginosa
Pseudomonas aeruginosa poses significant morbidity and mortality risks, especially for complex medical conditions (e.g., cystic fibrosis, burns, prosthetics).
It accumulates resistance through mechanisms like decreased outer membrane porin expression, increased efflux pump expression, and penicillin-binding protein modifications.
Defined as DTR, isolates exhibit non-susceptibility to traditional β-lactams and fluoroquinolones.
New β-lactam agents (e.g., ceftolozane-tazobactam, ceftazidime-avibactam) for DTR P. aeruginosa were approved, yet comparative outcome studies are limited.
Study Design and Methods
Study Design
Conducted a prospective observational study within the Johns Hopkins Health System from January 2018 to December 2023.
Eligibility Criteria
Inclusion: Clinical P. aeruginosa isolate meeting DTR definition, infection confirmation, susceptibility to administered β-lactam, treatment for ≥72 hours.
Exclusion: Patients dying within 72 hours post treatment initiation.
Antibiotic Stewardship
Both antibiotics require prior approval by the Antibiotic Stewardship Program before administration.
Treatment guidelines favor ceftolozane-tazobactam and ceftazidime-avibactam for DTR P. aeruginosa.
Dose adjustments based on renal function are outlined, but specific guidelines for CRRT are lacking.
Outcomes and Data Collection
Outcomes
Primary outcomes: 30-day all-cause mortality and emergence of resistance defined as a ≥4-fold rise in MIC.
Data collected included demographics, medical histories, renal function, and outcomes via chart review.
Statistical Analysis
Baseline characteristics were analyzed using t-tests and Fisher’s exact test.
Inverse probability weighting (IPW) was applied; balance was checked before and after weighting.
Results
Cohort Overview
186 patients analyzed: 102 (55%) on ceftolozane-tazobactam, 84 (45%) on ceftazidime-avibactam.
30-day mortality was close (21% vs 17%).
Resistance emergence was higher for ceftolozane-tazobactam (38% vs 25%), nearing significance (P = 0.09).
Baseline Characteristics Comparison
Both groups compared pre-and post-IPW showed similar baseline characteristics.
Annotations of severe immunocompromise were higher in the ceftolozane-tazobactam group prior to adjustments.
Common infection sources included pneumonia, bloodstream, skin & soft tissue.
Risk Factors for Emergence of Resistance
Continuous renal replacement therapy (CRRT) linked with increased resistance emergence (P = 0.04).
Longer treatment duration also associated with increased subsequent resistance likelihood.
Discussion and Conclusion
Although survival rates are similar between treatments, ceftolozane-tazobactam has a concerning higher rate of resistance emergence.
This implies careful selection in empiric antibiotic therapy could benefit patient outcomes, especially in cases needing renal replacement therapy.
Limitations include the observational nature of the study and potential bias in sample selection and outcomes.
Acknowledgments
Support received from various NIH grants focused on antibacterial resistance leadership.
The content represents the authors' views not necessarily reflective of the NIH.
Funding and Contributions
Detailed grants and author contributions provided, highlighting collaborative efforts across multiple departments.