Emotion
Theories of Emotion
Integration of Physiological and Behavioral Processes:
Emotion is defined as a subjective mental state often accompanied by distinctive behaviors, cognition, and involuntary physiological changes (Watson & Breedlove).
Physiological sensations such as goosebumps, tingly feelings, and fluttering in the stomach are results of autonomic nervous system activation:
Sympathetic Nervous System: Known as the “fight or flight” system; it prepares the body for action.
Parasympathetic Nervous System: Prepares the body to relax and recuperate.
The Relationship Between Emotions and Bodily Changes
James–Lange Theory: Proposes that an autonomic reaction triggers the feeling of an emotion.
Cannon-Bard Theory: Suggests that autonomic reaction and feeling occur simultaneously; the perception of specific autonomic arousal equals a specific emotion.
Schachter’s Theory
Cognitive Appraisal of Context:
Emotional labels are attributed to the sensations stemming from physiological arousal.
The specific emotion experienced is dependent on cognitive systems that assess the context of the current situation.
The Schachter and Singer experiment illustrates that while autonomic responses may heighten emotions, cognitive appraisal is crucial in determining the specific emotion experienced.
Intensity vs. Specific Emotion: There is a distinction where intensity of emotion can be differentiated from the specific emotion felt.
Core Set of Emotions
Darwin's Proposition:
Charles Darwin suggested that expressions of emotions are universal among all humans and have evolved from those of other species (1872).
Similarities in facial expressions between humans and nonhuman primates suggest a common ancestor.
Emotions as Motivational Programs
Adaptive Problem Solving:
Emotions have evolved to serve as motivational programs that coordinate behaviors and responses to address adaptive problems in the environment:
Fear: Responds to aversive situations and threats, enhancing survival.
Appetitive Program: Engages in positive situations related to food and mating, also improving survival.
Motivational States:
Fear increases adaptive behaviors and decreases maladaptive behaviors.
Facial Expressions and Communication
Cross-Cultural Similarity:
A significant degree of similarity exists across cultures regarding the expressions of specific emotions; however, notable differences can arise in certain isolated groups, particularly for surprise and disgust.
Cultural and experiential factors shape the rules governing facial expressions, signifying the complexity of emotional communication.
Cultural Differences in Recognizing Facial Expressions of Emotion
Research Findings (After J.A. Russell, 1994):
A graph illustrates the percentage of agreement in recognizing various emotional expressions across cultural groups:
Expression Categories:
Happiness (Ha)
Surprise (Su)
Anger (An)
Sadness (Sa)
Fear (Fe)
Disgust (Di)
Groupings for assessment include (1) non-Western literate (11 groups), (2) isolated nonliterate (3 groups), and (3) Western literate (20 groups).
Emotions in Mice
Dolensek et al. Study:
Researchers employed a machine learning program to classify various stimuli-driven facial expressions in mice into emotional categories.
When coupled with neuron imaging, specific neural circuits were identified in the insular cortex that correspond with facial expressions associated with emotions.
Mechanisms of Facial Expressions
Mediated by Muscles and Nerves:
Facial expressions arise from muscle contractions controlled by cranial nerves and CNS pathways:
Superficial Facial Muscles: Attach between facial skin points.
Deep Facial Muscles: Attach to bone, enabling significant movements (e.g., chewing).
Cranial Nerves Involved:
Facial Nerve (VII) and Motor Branch of Trigeminal Nerve (V).
Facial Feedback Hypothesis
Influence of Facial Expression on Mood:
Sensory feedback from facial expressions can alter mood states, lending support to the James–Lange theory.
Individuals simulating emotions (happy or sad faces) report stronger feelings corresponding to those expressions.
Brain Circuits and Emotions
Distinct Brain Circuits for Emotions:
Electrical brain stimulation can elicit emotional reactions.
Brain Self-stimulation: Animals (including humans) will often work to receive self-administered electrical brain stimulation.
Key brain sites supporting self-stimulation have predominantly been identified as subcortical structures located in the medial forebrain bundle.
Medial Forebrain Bundle and Reward Circuitry
Function of the Medial Forebrain Bundle:
This tract ascends from the midbrain through the hypothalamus and includes multiple sites for self-stimulation.
Nucleus Accumbens:
Serves as an important target for medial forebrain axons and represents a major component of the reward system; dopamine release in this area induces pleasurable feelings.
Brain Lesions and Emotional Responses
Effects of Brain Lesions on Emotion:
Decorticate Rage: Observed in dogs post-cortex removal, suggesting the cortex generally inhibits rage responses.
James Papez: Proposed that destruction within interconnected brain regions of the limbic system would disrupt emotional processing.
Klüver-Bucy Syndrome: Characterized by a significant reduction in fear and anxiety resulting from bilateral amygdala damage.
Amygdala's Role in Emotional Learning
Classical Conditioning and Fear:
Classical conditioning involves associating a neutral stimulus (tone) with an aversive stimulus (shock), resulting in freezing behavior and increased blood pressure.
The amygdala, located in the temporal lobe, is crucial for mediating fear responses during these conditioning processes.
Circuitry of Fear
Fear Processing Circuitry:
Visual representations of the circuitry involve connections from the thalamus to the sensory cortex and through various pathways leading to the amygdala.
High Road Pathway: Involves the sensory cortex and hippocampus, indicating a more processed emotional response.
Low Road Pathway: Directly routes sensory information to the amygdala for expedited emotional reactions.
Amygdala Circuitry in Humans
Human Studies:
Studies demonstrate the same circuitry involved in human emotional response:
Temporal Lobe Seizures: Often precede intense fear when involving the amygdala.
Patient S.M. (Urbach-Wiethe Disease): Exhibited a lack of fear towards spiders or dangerous individuals; absence of startle response and only recognizes non-fear emotions in others.
Activation of Brain Regions and Emotions
Distinct Brain Responses:
Activation during the experience of emotions such as love results in:**
Increased activity in the insula and anterior cingulate cortex.
Decreased activity in the posterior cingulate and prefrontal cortices.
A comparative study showed distinct brain activation associated with emotions such as sadness, happiness, anger, and fear but indicated no straightforward linkage between a single emotion and particular brain regions.
Exception: The amygdala has a specific, significant role related to fear.