Reproduction and Sexual Behavior Summary
Why Sex
Sexual reproduction increases variation in the genetic pool.
Benefits of variation:
Enables quick evolutionary adaptations.
Corrects genetic errors.
Sex and Hormones
Hormones: Chemicals secreted by cells, traveling through the bloodstream to target tissues.
Prenatal sexual differentiation begins with chromosomes.
Early development: Both sexes have Müllerian and Wolffian ducts, plus undifferentiated gonads.
Wolffian and Müllerian Ducts
Wolffian ducts: Precursors to male reproductive organs.
Develop into vas deferens and seminal vesicles.
Müllerian ducts: Precursors to female oviducts, uterus, and upper vagina.
The SRY Gene
Male Y chromosome includes the SRY gene.
Causes primitive gonads to develop into testes.
Developing testes produce:
Androgens (increase testes growth).
Müllerian inhibiting hormone (MIH).
Females lack the SRY gene; gonads develop into ovaries.
Hormones in Males and Females
Testes produce more androgens; ovaries, more estrogens.
Androgens and estrogens are steroid hormones.
Steroid Hormones
Contain four carbon rings; exert effects in multiple ways:
Binding to membrane receptors (like neurotransmitters).
Binding to chromosomes (activating or inactivating genes).
Androgens and Estrogens
Categories of chemicals, not specific chemicals.
Androgens: Primarily male hormones; testosterone is most known.
Estrogens: Estradiol is most common.
Progesterone
Prepares the uterus for implantation of a fertilized ovum.
Promotes the maintenance of pregnancy.
Genes and Sex Differences
Hormones do not account for all biological sex differences.
Genes on X and Y chromosomes also produce sex differences.
Specific genes on Y are active in brain areas; one on X is active only in the female brain.
Effects of Sex Hormones
Organizing effects:
Occur at sensitive development periods.
Determine male or female characteristics of brain/body.
Activating effects:
Occur at any time; temporarily activate a particular response.
Sexual Differentiation
Depends mostly on testosterone levels during a sensitive period.
Human sensitive period for genital formation: first trimester of pregnancy.
Organizing Effects of Sex Hormones – Androgens
Female mammals exposed to testosterone during sensitive periods become partly masculinized.
Enlarged clitoris.
Masculinized sexual behavior.
Male rats lacking androgen receptors or castrated develop female-like anatomy and behavior.
Organizing Effects of Sex Hormones – Estrogens
Estrogens don't modify male anatomy but alter internal structures (e.g., prostate gland).
Absence of sex hormones generally leads to female-looking external genitalia.
Females lacking estradiol during early life do not develop normal sexual behavior.
Sex Differences in the Hypothalamus
Sex hormones early in life influence hypothalamus, amygdala, and other areas, producing anatomical and physiological differences.
Sexually dimorphic nucleus:
Area in the anterior hypothalamus.
Larger in males; contributes to control of male sexual behavior.
Sex Differences in the Hypothalamus – Hormone Release
Female hypothalamus generates a cyclic pattern of hormone release.
During early rodent development, testosterone is converted to estradiol within neurons.
Activating Effects of Sex Hormones
Sex hormones exert activating effects to temporarily modify behavior.
Behavior can influence hormone secretion.
Oxytocin: Important for reproductive behavior; stimulates uterine contractions and mammary glands.
Males – Sexual Arousal
Testosterone increases touch sensitivity in the penis.
Sex hormones bind to receptors in the hypothalamus.
Testosterone triggers dopamine release by the MPOA and other areas.
Males – Effects of Neurotransmitters
Dopamine stimulation of D1 and D5 receptors is associated with sexual arousal.
Higher dopamine concentrations stimulate D2 receptors and lead to orgasm.
Serotonin decreases sexual activity by blocking dopamine release.
Males – Testosterone Levels
Testosterone levels correlate positively with sexual arousal and drive to seek partners.
Married men have lower testosterone levels.
Males – Sexual Offenders
Testosterone reduction has been tried to control sex offenders.
Difficulties include medication discontinuation and side effects.
Females – The Menstrual Cycle
Hypothalamus and pituitary interact with ovaries to produce the menstrual cycle.
Menstrual cycle: Periodic variation in hormones and fertility over ~28 days.
Females – FSH
After menstruation, the anterior pituitary releases follicle-stimulating hormone (FSH).
FSH promotes follicle growth in the ovary.
The follicle nurtures the ovum and produces estrogen.
Follicle builds up receptors to FSH,
-> produces increasing amounts of estradiol.
Females – FSH and LH
Increased estradiol causes anterior pituitary to increase FSH and luteinizing hormone (LH) release.
FSH and LH cause the follicle to release an ovum.
Females – Pregnancy
If pregnant, estradiol and progesterone levels increase gradually.
If not pregnant, levels decline, and the uterine lining is cast off (menstruation).
High levels of estradiol and progesterone produce heightened activity in serotonin receptors, causing nausea.
Females – Birth Control Pills
Birth control pills prevent pregnancy by interfering with the feedback cycle between ovaries and pituitary.
Combination pills contain estrogen and progesterone.
Thickened cervical mucus makes it harder for sperm to reach the egg.
Females – The Periovulatory Period
Point of highest fertility (middle of menstrual cycle).
Women become more sexually responsive.
Show increased attention to sex-related stimuli and flirting.
Effects of Sex Hormones on Nonsexual Characteristics
Prenatal androgens and estrogens influence brain development.
Stimulate brain areas important for memory.
Effects of Sex Hormones on Nonsexual Characteristics Part 2
Ability to recognize facial expressions.
Women receiving testosterone injections became less accurate at recognizing facial expressions of anger.
Parental Behavior – Hormonal Changes
Hormonal changes prepare female mammals to provide milk and care for young.
Prolactin is necessary for milk production.
Females change pattern of hormone receptors during pregnancy.
Parental Behavior – Hormonal Changes (cont’d.)
Vasopressin, made by the hypothalamus and secreted by the posterior pituitary, is associated with social behavior.
Mice can adopt maternal behavior without hormonal changes.
Parental Behavior – Hormones and the Quality of Care
Hormones play an initial role in maternal care quality.
Experience replaces hormonal influences as hormone levels decline.
Correlation exists between father’s hormones and level of interaction with their child.
Evolutionary Interpretations of Mating Behavior
Evolutionary Theory: Genes that help survival lead to more offspring.
Sexual selection: Genes that make an individual more appealing increase reproduction probability.
Evolutionary Differences: Men
Men seek multiple sex partners (especially short-term).
Evolutionary explanation: Spread genes through multiple partners or be loyal to one woman.
Evolutionary Differences: Women
Women can gain from multiple sexual partners (increased resources).
No direct evidence suggests specific genes influence preference for single or multiple mates.
What Men and Women Seek in a Mate
Men prefer a young partner (longer fertility).
Women select for long-term traits.
Status, ambition, generosity, kindness, emotional openness and willingness to devote resources to offspring.
Both prefer healthy, intelligent, honest, and physically attractive partners.
Differences in Jealousy
Men are more jealous at indications of a wife’s infidelity.
Evolutionary explanation: Ensure children are their own.
Cultural attitudes toward infidelity vary.
Sexuality is Cultural
Scientific data is not conclusive on the degree sexual behavior is evolutionary-guided or learned.
Cultural differences are influential.
Gender Identity and Gender-Differentiated Behaviors
Gender identity is how we identify sexually; sex differences are biological differences.
Gender differences result from thoughts about oneself as male or female.
Gender Identity and Gender-Differentiated Behaviors (cont’d.)
Most have a gender identity that matches their external appearance.
Some are dissatisfied with assigned gender.
Current evidence strongly suggests that biological factors are important, especially prenatal hormones.
Intersexes
People with anatomies intermediate between male and female.
Causes: Woman released two ova, each fertilized by different sperm or atypical hormone pattern occurs before birth.
Testicular Feminization
Androgen insensitivity: Individuals with XY chromosome pattern have smaller than average penis or female genital appearance.
Normal androgen production, but lack the receptor.
Congenital Adrenal Hyperplasia (CAH)
Overdevelopment of adrenal glands from birth.
Most common cause of intersex condition because cortisol production leads to extra testosterone.
Females become partly masculinized.
Genital Ambiguity
An estimated 1 out of 100 children is born with some degree of genital ambiguity.
1 in 2,000 has enough genital ambiguity to make the sex uncertain.
It's difficult for intersex people to connect due to hospital confidentiality.
Interests and Preferences of CAH Girls
Brains of genetic females with CAH were exposed to higher levels of testosterone during prenatal/early postnatal life.
Toy preference: intermediate between preferences of non-CAH boys and girls.
More physical aggression in adulthood.
Issues of Gender Assignment and Rearing
Past recommendations: rear intersex children as girls; perform surgery to make them look more feminine.
Current guidelines: Be honest with the person/family and identify gender based on predominant external genital appearance.
Discrepancies of Sexual Appearance
Some genetic males fail to produce an enzyme that converts testosterone to dihydrotestosterone.
Most look female at birth but a penis develops during adolescence and puberty.
Brain exposed to testosterone during early development.
Sexual Orientation
Homosexual behavior occurs in many animal species.
Most men discover sexual orientation early; process is slower for women.
Genetics
One contributing factor in homosexuality.
If one twin is homosexual, probability that other twin is homosexual is fairly high (for monozygotic twins).
No single particular gene has been identified.
An Evolutionary Question
Average homosexual man has one-fifth the number of children of average heterosexual man.
Hypotheses as to why genes have not been selected out:
Genes may be maintained by kin selection or related to epigenetics.
Prenatal Influences
Sexual orientation is not related to adult hormone levels.
Homosexual men and women tend to have normal hormonal levels for their biological sex.
Prenatal Stress
Mother’s immune system may exert prenatal effects.
Stress releases endorphins > Antagonizes the effects of testosterone on the hypothalamus.
Brain Anatomy
Differences in brain anatomy between homosexuals and heterosexuals exist.
On average, the homosexual brain is shifted towards the opposite sex in some ways.
Difficult to decipher why these differences exist.
Do Fetal Hormones Masculinize Human Behaviors in Adulthood?
Fetal androgen exposure is not as well understood in homosexual men.
Fraternal birth order effect: the more older brothers a boy has, the more likely he is to be homosexual.
Techniques of Modern Behavioral Endocrinology
Autoradiography shows where radioactively labeled chemicals accumulate in tissue.
Immunocytochemistry (ICC) uses antibodies to locate receptors and chemicals to make them visible.
In situ hybridization locates neurons that make mRNA for a receptor.