Lecture 6_ Chapter_006 (1)
Chapter 6: Rh Blood Group System
Overview
The Rh blood group system is a significant blood group system focused on the D antigen.
Understanding the Rh system is crucial for blood transfusions and preventing Hemolytic Disease of the Newborn (HDN).
Objectives of the Lecture
Recognize differences between ABO/Rh systems concerning immunoglobulin class, optimal temperature, and HDN.
Discuss five major alleles of the Rh system and inheritance theories.
Define classifications for weakened expressions of D antigen and their significance.
Understand implications for weak D antigen positive donors and recipients.
Compare theories of Rh null inheritance and their effects.
Identify actions of major types of Rh antisera and controls.
Interpret D and weak D results and corrective actions for false results.
Significance of D Antigen
The D antigen is highly immunogenic and is the most significant antigen after ABO.
Minimal exposure to the D antigen can prompt antibody production.
History of Rh System
In 1940, Levine and Stetson linked the Rh system with HDN.
Landsteiner and Wiener identified anti-Rh antibodies that agglutinate 85% of human RBCs.
Immunoglobulin Class Comparison
ABO Antibodies
Class: IgM
Nature: Naturally occurring; exposed through environmental antigens.
Optimal Temperature: Room temperature (20-25°C).
Rh Antibodies
Class: IgG
Nature: Produced through immunization during pregnancy or transfusions.
Optimal Temperature: 37°C.
Antigens of Rh System
D Positive/D Negative: Presence/absence of D antigen on RBCs.
Identifies C, c, E, and e antigens based on Fisher's naming convention.
Major alleles include C/c and E/e with recognized variations.
Genetics and Biochemistry of Rh Antigens
Involves genes closely linked on chromosome one:
RHD Gene: Determines D antigen expression.
RHCE Gene: Controls C, c, E, and e antigens.
RHAG Gene: Contributes to expression on chromosome 6.
Alleles show codominant inheritance.
Theories of Inheritance: Wiener vs. Fisher-Race
Fisher-Race Theory: Controlled by three sets of linked genes; phenotype based on D, C, E, c, e antigens.
Wiener Theory: A single gene at Rh locus produces a series of factors/antigens.
Rosenfield Terminology
Simplifies data entry, indicating antigens by number:
Rh1: D,
Rh2: C,
Rh3: E,
Rh4: c,
Rh5: e.
Example: D+, C+, E–, c+, e+ is denoted as Rh:1,2,-3,4,5.
Genotype vs. Phenotype
Genotype: Actual genetic makeup regarding Rh antigens.
Phenotype: Observable characteristics (e.g., blood type).
Variation in genotype frequency exists among ethnic groups.
Weak Expression of D
Not all D positive cells agglutinate reliably with anti-D.
Weak D testing involves incubation with anti-D at 37°C and follow-up AHG.
Positive result indicates weak D positive; negative indicates D negative.
Significance of Weak D
Recognizes weak D cells' potential to induce immune responses in transfusions.
Standard practice dictates transfusing D negative blood.
Weak D mothers do not receive RhIG.
Rh Null Phenotypes
Rh null red cells show no antigen sites, affecting red cell membrane structure.
Associated with stomatocytosis, hemolytic anemia, and decreased RBC survival.
Testing for Weak D Antigens
Uses indirect antiglobulin test (IAT) and monoclonal reagents.
Controls must validate weak D tests to avoid false positives.
False Positive and False Negative Results
False Positive Causes: Positive DAT, autoantibodies, rouleaux, reagent contamination.
False Negative Causes: Reagent issues, neutralization, improper testing procedure.
Rh Antibodies
Develop through immunization from transfusions/pregnancy.
Characteristics: IgG (low IgM), primarily detectable at 37°C.
Immunogenicity order: D > c > E > C > e.
Rarer Rh Alleles
Cw: observed in only 2% of whites.
V and VS antigens linked to f antigen on c/e haplotypes present in American blacks.
Additional Resources
Media Lab: Overview Of Major Antigens of the Rh Blood Group System.
Course: Clear as Glass Blood Banking: RhD Type Determination.