Exam 3 - Respiratory Drugs
Histamine
Chemical mediator that plays a role in the allergic response (along with others)
Actions:
Small vessel vasodilation
Increased capillary permeability
Bronchoconstriction
Sensory nerve stimulation
Allergic Rhinitis
Inflammatory disorder that affects the upper and lower airways and eyes
Treatment:
Antihistamines
Intranasal glucocorticoids
Sympathomimetic decongestants
Other—mast cell stabilizers, anticholinergics, leukotriene modifiers
Antihistamines “-ine”
Aka H1 Receptor antagonist relieves itching, sneezing, rhinorrhea (not congestion)
Generations:
Diphenhydramine (Benadryl – 1st)
Loratadine (Claritin – 2nd)
Azelastine (Astelin – 2nd generation, intranasal)
Use – mild allergy, motion sickness, insomnia
Adverse effects – sedation, nonsedative CNS effects – dizziness, fatigue, decreased coordination, anticholinergic effects, intranasal – headache, nosebleeds, unpleasant taste
All oral, some nasal and parenteral
**Not recommended for the common cold
Antihistamines— Patient Care
High risk - third trimester, nursing mothers
Caution: young children, elderly, patients with conditions aggravated by muscarinic blockade
Most effective when taken prophylactically – take throughout an allergy season – prevents histamine receptor activation
Sedative and anticholinergic effects more common with first generation than second
Sedative effects – caution driving, focused activities Nausea, vomiting – take with food
Don’t take with alcohol
Don’t crush or chew enteric coated preparations
In Summary
Histamine has a role in mild allergic symptoms (rhinitis, itching, local edema)
Mild allergic conditions respond well to antihistamines
Antihistamines are less effective than glucocorticoids for prevention and treatment of allergic rhinitis
DOES NOT play a major role in anaphylaxis (What is our drug of choice for anaphylaxis?)
Intranasal Glucocorticoids and Cromolyn
Intranasal Glucocorticoids
Fluticasone (Flonase – 2nd generation)
Anti-inflammatory Initial treatment of choice – reduce all symptoms
Adverse Effects – drying, burning, itching, sore throat, epistaxis, headache Intranasal
Intranasal Cromolyn
Cromolyn ( NasoCrom)
Stabilizes mast cells → suppresses release of inflammatory mediators
Prophylaxis – give before symptoms appear Adverse effects - minimal
Questions
Why should patients with glaucoma or BPH use first generation antihistamines with caution?
Name 4 anticholinergic effects that can occur when taking an antihistamine?
Which would be a better choice for allergic rhinitis – antihistamine or glucocorticoid
What is the action of cromolyn?
Sympathomimetics/ Decongestants
Uses – reduction of nasal congestion (allergic rhinitis, colds, sinusitis)
Action – stimulates alpha 1 receptors → vasoconstriction → shrinks swollen membranes (relieves stuffiness, congestion)
Examples: phenylephrine (Neo-synephrine) , pseudoephedrine (Sudafed)
Adverse effects: rebound congestion, CNS, caution in pts with cardiac disease
Administration: Topical (drops, sprays), oral
Sympathomimetics
PHENYLEPHRINE
Topicals
Act rapidly
Minimal systemic effects
Use no more than 5 days
Rebound congestion likely
PSEUDOEPHEDRINE
Orals
Act more slowly
More likely to have systemic effects
Cardiac and CNS effects more likely
Combat Methamphetamine
Epidemic Act - 2005
Abuse potential – amphetamine-like effects
Products with ephedrine and pseudoephedrine behind a counter Purchases limited—ID and signature required
Many products reformulated to contain phenylephrine
Other Respiratory
Antitussives - Cough
Opioids: Codeine
Non-opioids: Dextromethorphan (Robitussin), benzonatate (Tessalon)
Expectorants & Mucolytics
Guaifenesin (Mucinex) – expectorant – stimulates flow of respiratory secretions – increases/thins mucous
Acetylcysteine [acetaminophen reverse medication] (Mucomyst), hypertonic saline – mucolytics –direct action on mucous to make more watery.
Questions
What adrenergic receptor is activated by pseudoephedrine?
Alpha 1
Why should clients with hypertension and cardiac disorders avoid sympathomimetics?
Can cause vasoconstriction
Which form of cough medication when taken as prescribed can have the adverse effect of suppressing respirations?
To avoid adverse effects, what advice should the nurse give to a client taking oral benzonatate?
Pharmacological Therapy: Combos
Nasal decongestant
Antitussive
Analgesic
Antihistamine
Caffeine
OTC Cold Remedies— Safety in Children
Recommendations:
should not be used for infants and children under 4-6
Use only products labeled for peds use
Consult a healthcare professional before giving to a child
READ all product safety information
Use measuring device provided by product
If condition worsens or doesn’t improve – discontinue use of med and consult provider
Don’t use antihistamine products for sedation
Drugs for Asthma and COPD
Anti–inflammatory
Glucocorticoids – beclomethasone , budesonide (inhaled), prednisone (oral), methylpredisolone
Others – Cromolyn, leukotriene modifiers
Bronchodilators
Beta 2 agonists – albuterol (inhaled, short acting), salmeterol (inhaled, long acting)
Methylxanthines – theophylline (Elixophyllin)
Anticholinergic – ipratropium (Atrovent)
Types of Devices Used to Deliver Inhalation
Meds Metered dose inhalers (MDI) – with or without spacers
Dry powder inhalers (DPI)
Respimat
Jet nebulizers
Most drugs given by inhalation - increases therapeutic effects, reduces systemic effects, rapid relief
Inhalation Devices
Asthma Treatment Approach
Reduce impairment
Reduce chronic and troublesome symptoms (coughing, shortness of breath )
Reduce use of short acting beta agonists (SABA) for symptom relief to 2 days a week or less
Maintain pulmonary function
Maintain normal activity levels
Reduce risk
Prevent exacerbations
Reduce ER visits, hospitalizations
Maximum benefit - minimize drug side effects
Prevent progressive loss of lung function
Accomplished through reduction of triggers, meds for quick relief (SABA) and long-term control (Anti-inflammatories, SABA)
COPD Management
Four classifications of COPD
Mild - group A – few symptoms, low risk
Moderate - group B – increased symp., low risk
Severe - group C – few symptoms, high risk
Very severe - group D – increased symp., high risk
Goals
Reduce symptoms
Improve health status
Increase exercise tolerance
Reduce risks and mortality
Prevent disease progression
Prevent and manage exacerbations
Accomplished through use of bronchodilators (LABA – control, SABA exacerbations), glucocorticoids and other drugs
Anti-Inflammatory: Glucocorticoids
Suppress inflammation
Decrease synthesis and release of inflammatory mediators
Decrease infiltration, activity of inflammatory cells
Decrease edema of the airway mucosa
Decrease airway mucus production
Increase number of beta 2 receptors
Oral/inhalation – can be given IV
Not used to abort an ongoing attack, given on a fixed schedule, not prn.
Anti-inflammatory: Glucocorticoids
Inhaled
Beclomethasone (Vanceril) – MDI
Budesonide (Pulmicort) – DPI
Fluticasone (Flovent) - MDI
Oral
Prednisone (Deltasone)
IV
Methylprednisolone (Solu-medrol)
Glucocorticoid Adverse Effects
Inhaled → dysphonia, oropharyngeal candidiasis, bone loss (long-term use), slows growth rate in children
Long term, high dose oral → adrenal suppression, osteoporosis, hyperglycemia, peptic ulcer disease, growth suppression in young
If taking oral, long-term glucocorticoids –during stress may need to give oral/IV supplements due to adrenal suppression
Patient Care: Anti-inflammatory Glucocorticoids
Inhaled – gargle after use to reduce dysphonia, candidiasis
Patient education
Teach use of MDI, DPI and spacers
Adequate calcium, vitamin D, weightbearing exercises to reduce bone loss
Educate about adverse effects
Not used to abort an acute exacerbation
Anti-inflammatory: Leukotriene Modifiers
Leukotrienes – inflammatory mediators, promote bronchoconstriction, increase capillary permeability and promote other inflammatory cell actions.
Modifying response: reduces inflammatory response, prevents bronchoconstriction and eosinophil infiltration, reduces mucus production and edema
Maintenance (long-term) therapy, not quick relief
Drugs – Zafirlukast (Accolate), montelukast (Singulair), Zileuton (Zyflo)
Oral administration
Adverse effects: Zileuton – liver injury, dyspepsia, zafirukast – liver injury, GI disturbance, headache, montelukast – no liver injury
Anti-inflammatory: Cromolyn (Intal)
Suppress bronchial inflammation
Stabilizes cytoplasmic membrane of mast cells → suppresses release of inflammatory mediators Use only for prophylaxis – not acute problem
Must be taken before allergen exposure
Route: inhalation, fixed schedule
Safe, few adverse effects
Questions
What are three examples of drugs used to treat inflammation in patients with asthma?
A patient has been on long-term oral glucocorticoid therapy. Oral administration is changed to inhaled.
Why should long term glucocorticoid therapy not be abruptly stopped?
Why might supplemental glucocorticoids be needed?
increased stress, infections
Which anti-inflammatory acts by stabilizing mast cells?
chromolyn
Anti-inflammatory drugs to prevent asthma or COPD exacerbations are taken on what schedule (fixed or prn)?
fixed schedule
Bronchodilation — Beta 2 Adrenergic Agonist (respiratory effects)
Drugs:
albuterol (Proventil) - short-acting, inhaled, oral
salmeterol (Serevent) - long acting inhaled, terbutaline
(Brethine) - oral
Action
Activate Beta 2 receptors in smooth muscle
Bronchodilation → relieve of bronchospasm
Block release of histamine in lung
Increase ciliary motility
Use – acute bronchospasm
Route – inhaled, oral
Types
Inhaled short acting – prn relief, “rescue inhaler”
Inhaled long acting – long term control, not first line, not used alone
Oral – long term control, not first line, not used alone
Bronchodilation: Beta 2 Adrenergic Agonist
Adverse effects:
Short acting – tachycardia, angina, tremor
Long acting – increase risk of severe asthma and asthma-related death
Oral – beta 1 effects – angina, tachycardia, beta 2 effects on skeletal muscle - tremor
SA- albuterol (ProAir HFA), LA – salmeterol (Serevent), Oral –terbutaline (Brethine)
Questions
What two classes of drugs are most frequently used in the control of asthma?
What is the advantage of administering medications via inhalation, rather than oral or parenteral?
Can glucocorticoids be used to abort an ongoing asthma or COPD exacerbation?
How does a beta 2 agonist relieve symptoms of an acute exacerbation?
Bronchodilators — Actions
ANTICHOLINERGICS
Blocks muscarinic cholinergic receptors in bronchi
Promotes bronchodilation
Ipratropium (Atrovent) - inhaled
METHYLXANTHINES
Bronchodilation – exact mechanism uncertain
Narrow therapeutic range – 5-15 mcg/ml
>30 mcg/ml - seizures, dysrhythmias
Theophylline (Elixophyllin) – oral or IV
Avoid use with caffeine
Summary — Bronchodilators
Beta2-Adrenergic Agonists
Inhaled: short-acting albuterol (proventil)
Inhaled: long-acting salmeterol (Serevent)
Oral
albuterol (Proventil)
terbutaline(Brethine)
Methylxanthines
Theophylline
Anticholinergics
Ipratropium (Atrovent)
Questions
What is a non-pharmacologic treatment for asthma resulting from allergies?
What is the treatment for an acute asthma exacerbation?
A client uses a rescue inhaler consistently more than twice a week, what might be needed?
What is the treatment for a patient with stable COPD? Severe chronic COPD? COPD exacerbation?