Sleep Apnias
Overview of Sleep-Related Diseases
Focus on Sleep Apnea
Differentiating between types of sleep apnea: obstructive sleep apnea (OSA) and central sleep apnea (CSA)
Obstructive Sleep Apnea (OSA)
Definition:
OSA is related to an excessive amount of soft tissue in the upper airway, leading to obstruction during sleep.
Causes:
Commonly associated with:
Obesity
Increased neck circumference
Excessive soft tissue leads to the airway being obstructed as it falls over the tracheal opening.
Symptoms:
Characterized by a breathing pattern:
Breathing occurs, then a period of apnea (cessation of airflow) where breathing stops for over 10 seconds.
Repeat breathing pattern may be observed: breathe-breathe-breathe-apnea.
Apnea Definition:
Defined as the cessation of airflow for greater than 10 seconds during sleep.
Physiological Response:
As the airway obstructs, blood oxygen levels (SpO2) drop, signaling the body to start breathing again.
Treatment:
Continuous Positive Airway Pressure (CPAP) is used to stent open the upper airways, preventing obstruction.
Apnea-Hypopnea Index (AHI):
Measures apneic episodes per hour:
0-4: Normal
5-14: Mild sleep apnea
15-29: Moderate sleep apnea
30 or greater: Severe sleep apnea
AHI aids in assessing treatment effectiveness. If AHI remains high while on CPAP, pressure may need to be increased.
Simplified AHI Reference:
Remember: 5 (Mild) - 15 (Moderate) - 30 (Severe)
Expect variable AHI numbers rather than specific clean digits like exactly 30.
Central Sleep Apnea (CSA)
Definition:
CSA occurs when no signal from the brain is sent to breathe during sleep, leading to the cessation of breathing effort.
Causes:
Often associated with congenital defects that affect the brain's signaling during sleep.
Symptoms:
Similar to OSA: breathing pattern is observed (breath-breath-breath-apnea), however, the emphasis is on the absence of breath, not the obstruction.
Physiological Response:
During apnea:
Blood oxygen levels (SpO2) drop.
Carbon dioxide (CO2) levels rise.
In certain cases, the brain may not initiate breathing again.
Treatment Options:
Does not utilize CPAP since it is not an obstructive issue:
Mechanical ventilation
Diaphragmatic pacemakers
Pharmacological agents like methylxanthines to stimulate the neural drive to breathe.
Distinguishing Between OSA and CSA
Assessment Tools:
Sleep studies analyze:
Airflow (can be measured as chest or abdominal excursion)
SpO2 levels.
Identifying OSA:
Look for signs of respiratory effort during the apnea periods:
If abdominal/chest excursion is present, OSA is likely due to an obstruction.
Identifying CSA:
If there is no effort or movement during apnea, it indicates CSA as the brain fails to signal.
Observational Patterns:
Airflow characteristics during apnea will show a clear distinction:
OSA: effort to breathe is noted despite obstruction.
CSA: no attempt to breathe is noted during apnea.
Conclusion
Clear understanding of OSA and CSA is crucial for effective treatment and patient care.
This knowledge serves as a foundation for further studies on related diseases, including Guillain Barre and Myasthenia Gravis.