micro chapter 5
Chapter 5: Viruses
Lesson 5.1: General Structure and Classification
Methods of Classifying Viruses:
Two commonly used methods:
Morphological characteristics
Nucleic acid type
Definitions and Descriptions:
Capsid: The protein coat of a virus that encases the genetic material.
Capsomeres: The individual protein subunits that make up the capsid.
Nucleocapsid: The complex of the viral nucleic acid and the capsid, serving to protect the viral genome.
Virion: A complete, infectious virus particle consisting of nucleic acid, capsid, and optional envelope.
Envelope: A lipid membrane surrounding some viruses, derived from the host cell's membrane, containing viral glycoproteins.
Types of Viral Genomes:
Viral genomes can be classified as:
DNA
RNA
Types of nucleic acids:
Double-stranded (ds) or single-stranded (ss)
Can be linear or circular.
General Structure of Viruses
Viruses: Microscopic particles that infect host cells to replicate.
Composed of either DNA or RNA as genome.
Cannot reproduce independently; are obligate intracellular parasites.
Infect both eukaryotic and prokaryotic cells.
Bacteriophage: A type of virus that specifically infects bacteria.
Classification of Viruses
Challenge in Classification:
Taxonomic classification is complicated due to diversity.
Classification Criteria:
Viruses classified based on:
Morphology
Nucleic acid type
Mode of replication
Host organism
Associated diseases
International Classification:
Order: -virales
Family: -viridae
Subfamily: -virinae
Genus: -virus
Species: -virus
Baltimore Classification System
Overview:
Categorizes viruses into one of seven groups based on the relationship between genomic nucleic acid and messenger RNA:
Group I: dsDNA
Group II: ssDNA
Group III: dsRNA
Group IV: ssRNA+
Group V: ssRNA-
Group VI: ssRNA-RT
Group VII: dsDNA-RT
Morphology of Viruses
Size Variation:
Viruses range from approximately 20 nm (e.g., parvovirus) to up to 450 nm (e.g., poxvirus).
Most viruses cannot be visualized with light microscopy except for some larger viruses like mimivirus (750 nm) found in amoebas, which has about 1000 genes on double-stranded circular DNA.
Capsid Structure:
Composed of proteins encoded by viral genes; provides structural integrity and serves as basis for morphological classification.
Protomers aggregate to form capsomeres, which then assemble into the capsid.
Nucleoproteins:
Proteins that associate with nucleic acid; when capsid proteins complex with nucleic acid, they form the nucleocapsid.
Spikes:
Long projections extending from the nucleocapsid.
Virion:
The fully assembled and infectious virus particle.
Classification by Morphology
Helical Viruses:
Features rod-shaped capsomeres, encapsulating either ssRNA or ssDNA.
Can be naked (e.g., tobacco mosaic virus) or enveloped (e.g., influenza virus).
Icosahedral Viruses:
Form a three-dimensional geometric shape with 12 corners, 20 triangular faces, and 30 edges.
The arrangement of capsomeres varies among different viruses.
Examples include: Herpesviridae, Adenoviridae, Papovaviridae, and Parvoviridae.
Enveloped and Complex Viruses
Enveloped Viruses:
Consist of a viral envelope that surrounds the nucleocapsid.
Envelope is typically acquired by budding from a host membrane, which may include viral glycoproteins and some host membrane proteins replaced by viral proteins.
Complex Viruses:
Bacteriophages:
Comprise a head or capsid containing nucleic acid, variable in size and shape, made up of one or more proteins.
Include a tail—a hollow tube surrounded by a contractile sheath—with a base plate and fibers for attachment.
Poxviruses:
Lack a regular capsid structure.
Feature a nucleoid surrounded by a membrane with two lateral bodies present.
Genome Types
General Types:
All viruses possess either DNA or RNA, classified as:
dsDNA
ssDNA
dsRNA
ssRNA
Genetic material may be linear or circular, usually found in one form.
Exception: Cytomegalovirus (genus of herpesviruses) contains both DNA and RNA capacities.
DNA Viruses:
Most belong to Group I or II of the Baltimore classification.
Replicate using DNA-dependent DNA polymerase.
RNA Viruses:
Utilize RNA as genetic material and do not undergo DNA replication.
Typically group II, IV, or V in the Baltimore classification.
Reverse Transcribing Viruses:
Contain RNA genomes and replicate through a DNA intermediate.
Classified under group VI and VII in the Baltimore system.
Lesson 5.2: Viral Multiplication, Effects on Host Cells, and Subviral Agents
Overview of Viral Multiplication:
Viruses are acellular and lack the capacity for replication through mitosis or binary fission; they rely on the host cell's machinery to produce copies.
Outside of host cells, viruses behave as inert particles.
Multiplication of Bacteriophages
Phage Typing:
Bacteriophages (phages) are utilized in diagnostic laboratories to identify pathogenic bacteria due to their specificity to particular bacterial strains.
Steps in Phage Multiplication:
Adsorption:
Phage attaches to its bacterial host, mediated by tail fibers.
Phage receptors on bacterial surface include proteins on cell walls, pili, and flagella.
Penetration:
After binding, phage injects nucleic acid into the bacterium through contraction of the tail structure.
Usually, only the nucleic acid enters the cell, leaving the phage outside.
Replication:
Host cell metabolism shifts towards the expression of viral nucleic acid.
Production of viral components occurs.
Assembly:
Viral components are assembled into new phage particles.
Maturation:
Finalization of viral assembly into mature virions.
Release:
New phages cause cell lysis, releasing newly formed particles into the environment.
Types of Bacteriophages
Lytic or Virulent Phages:
Multiply and destroy the bacterium post-multiplication, causing lysis.
Phage Life Cycle Stages:
Eclipse period: No infectious blood particles observed.
Intracellular accumulation phase: Assembly occurs.
Lysis and release phase: New phages are released from the destroyed host.
Lysogenic or Temperate Phages:
Phage integrates its DNA into the host genome as a prophage, persisting in this dormant state.
Prophage can subsequently lead to lysis or continue to coexist within the host cell division.
Multiplication of Animal Viruses
Comparison to Bacteriophage Multiplication:
Similar stages in the multiplication cycle:
Adsorption
Penetration
Uncoating
Replication
Assembly
Release
The duration of the viral cycle can vary significantly between different viruses.
Viral Multiplication Steps in Detail
Adsorption:
Virus binds to a susceptible host via specific attachment sites, known as receptors, which may be located on either capsids or envelopes.
Host species can be limited in range as viruses can only infect species with available receptors.
Penetration:
The virus enters the cytoplasm of the host cell either through endocytosis or membrane fusion, where the nucleocapsid enters the cell.
Uncoating:
Viral nucleic acid is released from the viral capsid.
If the virus enters the host cell via endocytosis, it is enclosed in a vesicle, which is dissolved by cellular enzymes, releasing the viral nucleic acid.
Replication:
DNA Viruses: Viral DNA enters the host cell's nucleus for transcription into mRNA, which is translated to produce early viral proteins necessary for further replication.
RNA Viruses: Must provide their own polymerase for transcription; retroviruses have unique replication mechanisms.
Translation of mRNA into viral proteins occurs on host ribosomes, contributing to the final assembly.
Assembly and Release:
Mature viral particles are constructed from accumulating viral components.
Release Mechanisms:
Budding (exocytosis)
Lysis of the host cell
Types of Viral Infections
Classification of Viral Infections:
Abortive: Infections without production of viral particles.
Lytic or Cytocidal: Viral infections that cause host cell death.
Persistent Infections: Infections that are not lytic but can lead to chronic or latent states.
Chronic persistent: Ongoing replication without host cell death.
Latent: Limited synthesis with long asymptomatic periods; examples—cold sores and shingles.
Slow infections: Long incubation periods leading to eventual disease; damage may take years.
Transforming infections: Viral nucleic acids may integrate into host cell genomes, leading to oncogenic transformations.
Host Cell Damage from Viral Infections
Morphological Effects:
Cytopathic changes induced by viral infections include altered cell shape, surface detachment, lysis, membrane fusion, altered permeability, and inclusion body formation.
Physiological Effects:
Incorporation of viral proteins alters the behavior and characteristics of host cell membranes, affecting ion movement and cellular activity.
Biochemical Effects:
Viral presence can inhibit or alter the synthesis of host macromolecules, leading to dysfunction.
Genotoxic Effects:
Genotoxicity may result in host DNA damage and mutations, which can initiate processes leading to cancer.
Major Groups of Viruses in Vertebrates
Characteristics:
Most viruses cause disease upon breaching protective host barriers and are typically specific to certain tissue or cell types.
Viral Genomes:
Consists of either DNA or RNA, which can lead to various disease outcomes.
DNA Viruses
Overview:
Various DNA viruses with associated diseases:
Adenoviruses:
57 adenoviral serotypes identified in humans, resistant to environmental agents, with implications in gene therapy.
Associated with respiratory illness, gastrointestinal disease, conjunctivitis, and cystitis.
Hepadnaviruses:
Cause hepatitis, notably hepatitis B, capable of producing acute or chronic diseases.
Herpesviruses:
Examples include Herpes simplex 1 and 2; Varicella-zoster virus.
Papillomaviruses and Polyomaviruses:
Associated with warts and potential cervical carcinomas.
Parvoviruses:
Only B19 causes disease in humans; others affect animals.
Poxviruses:
The largest viruses known, including those causing smallpox.
RNA Viruses
Overview:
Diverse families with significant impacts on human health:
Bunyaviridae:
Transmitted by arthropods.
Coronaviruses:
Responsible for various respiratory and enteric diseases including SARS.
Orthomyxoviruses:
Include influenza A, B, and C viruses, with A being the most virulent to humans.
Paramyxoviruses:
Includes Morbillivirus (measles) and Nipah virus among others.
Picornaviruses:
A large group including enteroviruses, rhinoviruses, and various hepatoviruses, acknowledged as the smallest RNA viruses.
Rhabdoviruses:
Infect a broad spectrum of hosts; the rabies virus is a well-known member.
Reoviruses:
Not specifically disease-associated but includes rotavirus, the leading cause of gastroenteritis in children.
Retroviruses:
Groups including Oncovirinae, Lentivirinae (HIV) among others that replicate through reverse transcription.
Togaviruses:
Includes alphaviruses and rubella virus.
Flaviviruses:
Includes viruses causing yellow fever, dengue fever, and hepatitis C.
Subviral Agents
Viroids:
Consist of circular RNA molecules that cause diseases in plants without requiring helper viruses.
Virusoids:
RNA molecules that require the presence of a helper virus, often causing plant infections.
Example: Hepatitis D virus necessitates simultaneous infection with hepatitis B virus.
Prions:
Infectious particles derived from abnormally folded proteins, lacking nucleic acids.
Associated diseases known as transmissible spongiform encephalopathies (e.g., mad cow disease, kuru).