Mood Disorders: Treatments and Psychological Therapies (Summary)
Electroconvulsive Therapy (ECT)
First-line meds are common, but ECT is used for severe, persistent mood disorders and treatment resistance.
History: introduced early 20th century; stigmatized in 1950s–1970s; resurged in the 1980s; now widely used in hospitals worldwide.
Typical procedure:
Seizure induced by passing current through the brain; anesthesia and muscle relaxants used.
Electrodes placed on the head; current of 70\text{ to }150\,\text{V} for about 0.5\text{ to }1\,\text{s}.
Seizure lasts about 1\,\text{minute}.
Full course: typically 6\text{ to }12\text{ sessions} .
ECT effectiveness:
Remission achieved in 50\% \text{ to } 80\% of clients; higher than antidepressants in some cases.
More likely to reduce symptoms than antidepressants for treatment-resistant depression.
Most effective as a first-line option in severe depression or during psychotic features; strong anti-suicidal effects.
Brain mechanism (still unclear):
May cause structural changes in mood-related regions: prefrontal cortex, hippocampus, angular gyrus, insula, among others.
Likely involves brain networks rather than a single area; may alter neurotransmitters (serotonin, dopamine), reduce inflammation, promote neuroplasticity and neurogenesis in regions like the hippocampus, basal ganglia, anterior cingulate, and prefrontal cortex.
Neurogenesis is promoted; ECT may rewire brain networks important for mood regulation.
Side effects & memory:
ECT can cause memory loss and learning difficulties, especially days after treatment.
Traditional bilateral ECT had greater cognitive effects; modern practice often uses unilateral ECT (usually right-sided) to reduce memory impairment, though bilateral ECT is still used when needed.
Relapse & maintenance:
Relapse can be high, up to 85\%; >30\% relapse within the first 6 months post-ECT.
Continuation/maintenance ECT can reduce relapse for some, but guidelines are not clear; gradual discontinuation may help reduce early relapse.
Access & perception:
Stigma and limited access in some regions impact perceived right to effective treatment.
Newer Methods of Brain Stimulation
Repetitive transcranial magnetic stimulation (rTMS)
Noninvasive, outpatient treatment using high-intensity magnetic pulses.
Target: left prefrontal cortex (often hypoactive in depression).
Few side effects; typically mild headaches; no anesthesia required.
Shown to improve depressive symptoms in treatment-resistant cases; promising alternative for those who cannot tolerate ECT.
Vagus nerve stimulation (VNS)
Invasive: electrodes implanted on the vagus nerve with a pulse generator under the skin (left chest wall).
Longitudinal data (≈5 years) show superior outcomes in effectiveness and mortality for chronic, severe, treatment-resistant depression, but coverage is limited by insurers.
Deep brain stimulation (DBS)
Electrodes implanted in specific brain regions; connected to an implanted pulse generator.
Small trials show promise for intractable depression; mechanism and optimal targets remain under study.
Light Therapy
applicable to Seasonal Affective Disorder (SAD), a winter-specifier of major depressive disorder.
SAD biology: may involve deficient retinal sensitivity to light and circadian rhythm disruption.
Efficacy:
Light therapy alone produced remission in 57\% of SAD patients in one trial.
Remission rose to 79\% when combined with cognitive therapy; control group remission was 23\%.
Mechanisms:
Resetting circadian rhythms; reducing melatonin production; increasing norepinephrine/serotonin; bright light may directly boost serotonin.
Psychological Treatments for Mood Disorders
Theoretical basis: behavioral, cognitive, and behavioral–cognitive approaches.
Goals across therapies: reduce negative thoughts, increase adaptive behaviors, and improve functioning.
Behavioral Therapy (short-term, ~12 weeks)
Focuses on increasing positive reinforcers and reducing aversive experiences.
Functional analysis to identify situations that worsen symptoms and to modify environmental interactions.
Teach relaxation and social/role skills; modify activities to increase pleasurable experiences and reduce isolation.
Case study highlight: client learned to schedule social/recreational activities and relaxation techniques; improved mood through increased control.
Cognitive-Behavioral Therapy (CBT)
Integrates cognitive and behavioral theories; two general goals:
Change negative, hopeless thinking patterns.
Solve real-life problems and increase effective reinforcers.
Structure: brief, time-limited (typically 6\text{ to }12\text{ weeks}) with client-set goals.
Process:
Identify negative automatic thoughts and link to mood.
Homework: track mood and thoughts (e.g., using thought records similar to Figure 7 in the text).
Case study highlight: Susan challenged beliefs about maternal criticism; learned to attribute criticism to her mother’s issues, not her own worth; gained greater emotional control.
Notes on Major Concepts
ECT remains a highly effective option for treatment-resistant depression and certain severe mood episodes, with rapid improvement in some cases.
Memory side effects are a consideration; unilateral ECT is used to minimize cognitive impact, though bilateral ECT may be more effective in some instances.
Newer brain stimulation methods (rTMS, VNS, DBS) offer alternatives with varying levels of invasiveness and evidence; ongoing research into mechanisms and optimal use.
Light therapy is a nonpharmacological option for SAD, with mechanisms linked to circadian biology and neurotransmitter regulation.
Psychological treatments (Behavioral Therapy, CBT) focus on altering behavior-environment interactions and cognitive processes to relieve depressive symptoms; often time-limited and goal-focused.
Key Formulas and Numbers
ECT current/duration: 70\le V \le 150, \quad \text{duration} = 0.5\text{ to }1\ \text{s}
Seizure duration: \approx 1\ \text{minute}
Treatment course: 6\text{ to }12\ \text{sessions}
Remission rates: 50\% \le \text{remission} \le 80\%
Relapse rates: up to 85\%; >30\% relapse in first 6 months
CBT/Behavioral therapy duration: typically 6\text{ to }12\ \text{weeks}