Syncope Notes
Syncope Overview
Objectives
Understand syncope.
Recognize differences in syncope between young and old.
Recognize the main causes of syncope:
Vasovagal response
Orthostatic hypotension
Cardiac disease
How to investigate and diagnose syncope.
How to treat the main causes of syncope.
Definition of Syncope
Derived from Greek words "Sūn" (together) and "kóptō" (to strike, cut off).
Transient loss of consciousness due to global cerebral hypoperfusion.
Characterized by rapid onset, short duration, and complete, spontaneous recovery.
Syncope Incidence
40-50% of the population experiences syncope over a lifetime.
Syncope in Young People
Over 90% of syncope in young people (<40 years) is vasovagal syncope (VVS).
VVS is the common faint.
VVS is a reflex, not an illness.
Involves the Bezold-Jarisch reflex.
Bezold-Jarisch Reflex
Sequence of events:
Decreased blood pressure (BP↓)
Decreased venous return (Venous return ↓)
Decreased end-diastolic volume (End diastolic volume ↓)
Increased heart rate (HR ↑)
Vigorous contraction of empty ventricle
Baroreceptors trigger
Vagus nerve stimulation
Medulla oblongata
Vasovagal reaction: HR ↓, BP ↓
Recognizing VVS - Presyncope
Light-headedness
Blackout (retinal ischaemia)
Yawning
Restlessness
Strong urge to lie down
Rising sensation
Intense heat
Nausea
Fatigue
Older people:
Very short, or even absent prodrome
Non-specific symptoms
"Just went down"
"I must have fallen"
Witness observations:
White/grey appearance
Sweating
VVS - Syncope Phase
Loss of consciousness
Loss of postural tone
Injury
Transient
Incontinence
Myoclonic jerks (more common in young people)
Older people:
Little/no myoclonus
Witness misinterpretations:
T-LOC lasting minutes to hours (mistaken for epilepsy)
Potential misdiagnosis as epilepsy
Rarely, mistaken for death (older people)
VVS - Post-Syncope
Spontaneous recovery
Fatigue (often profound)
Coat hanger headache
Older people:
Longer recovery
VVS - Triggers
Situational syncope:
Pain
Cough
Defecation/micturition
Deglutition (swallowing)
Gelastic (laughter-induced)
Shock/emotion
Standing, heat, dehydration, vasodilation (exercise, alcohol, sepsis, drugs)
VVS - Treatment
Water (+++)
Salt
Caffeine (↑/↓)
Compression stockings
Physical counter maneuvers
Trigger avoidance
Medication adjustments (stopping or starting medications)
Tilt-training
Pacemaker (in rare cases)
Syncope - Other Causes
VVS
Situational syncope
Carotid sinus syndrome
Carotid Sinus Syndrome
Affects older people
Little/no warning
Unexplained falls/syncope
Facial/head injuries
Types:
Vasodepressor (↓BP): Treat as VVS
Cardio-inhibitory (↓HR): Pacemaker
Syncope - Cardiac Causes
Cardiac structural disease
Arrhythmia
Orthostatic hypotension
Neurally mediated syncope
Orthostatic Hypotension (OH)
Pathophysiology of OH
Venous pooling
↓ venous return
↓ End-Diastolic Volume (EDV)
↓ Stroke Volume (SV)
↓ Cardiac Output (CO)
↓ Blood Pressure (BP)
↓ Baroreceptor stretch
Medulla response: Sympathetic (↑ activity), Parasympathetic (↓ activity)
↑ Heart Rate (HR)
Vasoconstriction
↑ Venous return
↑ EDV
↑ BP
↑ CO
Recognizing OH
Symptoms just after standing up
Worse in the morning
Nocturnal diuresis
Low volume intake
Low cortisol levels
Dizziness
Falls
Non-specific low BP symptoms
Aggravating factors:
Meals
Alcohol
Medications
Illness
Dehydration/heat
OH - Treatment
Water (+++)
Caffeine (↑/↓)
Compression stockings
Physical counter maneuvers
Trigger avoidance
Medication adjustments (stopping or starting medications)
VVS vs. OH
VVS: A reflex
OH: Abnormal response
Cardiac Syncope - Indicators
History of heart disease, especially heart failure
Chest pain, shortness of breath (SOB)
Palpitations
Little/no presyncope
Abnormal ECG
Family history of sudden cardiac death
Cardiac medications
Syncope while sitting/supine
ECG Clues
First-degree heart block: generally does not cause syncope.
Second-degree block - Mobitz 1: 2^{nd} degree block - Mobitz 1 – could cause syncope - further testing required
Second-degree AV block - Mobitz 2: causes syncope
Third-degree block: causes syncope
Other ECG Clues
Brady-arrhythmias:
Alternating left & right bundle branch block
Bifascicular block (LBBB or RBBB with LAFB)
Ventricular fibrillation/Ventricular tachycardia (VF/VT)
Bradycardia: HR <40 while awake or <50 while active
Sinus pause ≥3 seconds
Tachyarrthymias:
Supraventricular tachycardia (SVT)
Delta waves/pre-excitation
Long or short QT
Other (rare):
Epsilon waves (ARVD)
Brugada (T wave changes, RBBB pattern)
Investigations - Summary
VVS:
Clinical diagnosis
Tilt-test in cases of doubt
OH:
Lying & standing BP
Systolic BP (sBP) drop ≥20 mmHg or diastolic BP (dBP) drop ≥10 mmHg
Carotid Sinus Syndrome (CSS):
Carotid sinus massage (CSM) for 10s (supine then upright)
sBP drop >50 mmHg
Asystole >3s (>6s if pacemaker present)
Cardiogenic syncope:
12 lead ECG
Previous ischaemia
‘block’ or bradycardia
Tachyarrhythmias
External loop recorder
Internal loop recorder
Falls vs Syncope
When falls are actually syncope…
Various risk factors and incidents leading up to falls:
Balance, Gait, Culprit medication, Home hazard, Orthostatic hypotension, Vision, Carotid sinus hypersensitivity, Neuropathy, Depression, and Vasovagal are all factors that result in falls.
Falls vs. Syncope - Key Differentiators
Favors Falls:
Specifically recalls impact
Hand/wrist injury
No presyncope
Presence of environmental factor (e.g. ice/uneven pavement)
Favors Syncope:
"I must have…"
Facial injury
May or may not recall presyncope
Unexplained
Important Points - Older People
Less pre-syncope/warning
Often present with falls not syncope
Carotid sinus syndrome affects >40 year olds
Higher risk of cardiac causes
Important Points - VVS
Syncope: transient, global, rapid, spontaneous
Reflex not disease
VVS – really common
Non-drug treatments are important