Neurocognitive Disorders Notes
Perspectives on Neurocognitive Disorders
- Affect multiple cognitive processes:
- Learning
- Memory
- Consciousness
- Most develop later in life.
- Three classes:
- Delirium
- Mild neurocognitive disorder
- Major neurocognitive disorder
- DSM changes:
- "Organic" vs. "Neurocognitive disorders"
- Broad cognitive impairments:
- Memory
- Attention
- Perception
- Thinking
- Profound changes:
Delirium
- Clinical description:
- Global impairments:
- Develops rapidly:
- Example: confusion, disorientation, attention, memory, and language deficits.
- Diagnostic Criteria for Delirium (DSM-5):
- A. Disturbance in attention and awareness.
- B. The disturbance develops over a short period of time and tends to fluctuate in severity during the course of a day.
- C. An additional disturbance in cognition.
- D. The disturbances are not better explained by another preexisting, established, or evolving neurocognitive disorder.
- E. Evidence from history, physical examination, or laboratory findings that the disturbance is a direct physiological consequence of another medical condition.
- Statistics:
- 20% of older adults in acute care (ER).
- Highest prevalence:
- Older adults
- AIDS patients
- Cancer patients
- Medical patients
- Full recovery = several weeks
- "Vital signs"
- Subtypes of delirium:
- Delirium due to a general medical condition
- Substance-induced delirium
- Delirium due to multiple etiologies
- Delirium not otherwise specified
- Causes:
- Drug intoxication:
- Medications
- Illicit drugs: Ecstasy (methylene-dioxymethamphetamine)
- Poisons
- Withdrawal from drugs
- Infections
- Head injury
- High fever
- 50% of people with dementia suffer at least one episode of delirium
- Age
- Infections
- Head injury/brain trauma
- Sleep deprivation
- Immobility
- Excessive stress
- fMRI scanning
- Ethical concerns
- Treatment:
- Treat underlying medical or withdrawal problems.
- Acute delirium:
- Haloperidol or olanzapine
- First line of treatment - Psychosocial interventions:
- Education
- Reassurance
- Coping strategies
- Prevention:
- Proper medical care
- Proper medication use
Major and Mild Neurocognitive Disorders
- Major neurocognitive disorder (previously labeled dementia) is a gradual deterioration of brain functioning that affects:
- memory
- Judgment
- Language
- Other advanced cognitive processes
- Mild neurocognitive disorder is a new DSM-5 disorder that was created to focus attention on the early stages of cognitive decline.
- Diagnostic Criteria for Mild Neurocognitive Disorder (DSM-5):
- A. Evidence of modest cognitive decline from a previous level of performance.
- Concern of the individual, a knowledgeable informant, or the clinician.
- A modest impairment in cognitive performance, preferably documented by standardized neuropsychological testing.
- B. The cognitive deficits do not interfere with capacity for independence in everyday activities.
- C. The cognitive deficits do not occur exclusively in the context of a delirium.
- D. The cognitive deficits are not better explained by another mental disorder.
- Specify whether due to:
- Alzheimer's disease
- Frontotemporal lobar degeneration
- Lewy body disease
- Vascular disease
- Traumatic brain injury
- Substance/medication use
- HIV infection
- Prion disease
- Parkinson's disease
- Huntington's disease
- Another medical condition
- Multiple etiologies
- Unspecified
- Clinical description:
- Gradual deterioration of brain functioning
- Affects multiple domains:
- Judgment
- Memory
- Language
- Advanced cognitive processes
- Multiple etiologies
- Initial symptoms:
- Memory impairment
- Visuospatial skills deficits
- Agnosia
- Delusions
- Depression
- Agitation
- Aggression
- Apathy
- Later symptoms:
- Continued cognitive decline
- Assistance with activities of daily living
- Death = inactivity + other illnesses
- Statistics:
- Onset:
- Any age
- Most common in the elderly
- Prevalence:
- New case every seven seconds
- 5% older than 65
- 20-40% over age 85
- Rates double with every 5 years after the age of 75
- Alzheimer’s in centenarians = 100%
- Longer lifespan
- Mild neurocognitive disorder:
- 10% over 70
- Black men and women at higher risk
- Cost of caring for 1 year $$=$ $100 billion
DSM-5 Classes of Neurocognitive Disorder
- Etiology:
- Dementia of the Alzheimer’s type
- Vascular injury
- Frontotemporal degeneration
- Traumatic brain injury
- Lewy body disease
- Parkinson’s disease
- HIV infection
- Substance use
- Huntington’s disease
- Prion disease
- Another medical condition
Neurocognitive Disorder Due to Alzheimer’s Disease
- Clinical description:
- Multiple cognitive deficits:
- Memory
- Orientation
- Judgment
- Reasoning
- Develop gradually and steadily
- Confusion
- Agitation / combativeness
- Depression
- Anxious
- Sundowner syndrome
- Range of cognitive deficits:
- Aphasia
- Apraxia
- Agnosia
- Executive function
- Significant social and occupational impairments
- Definitive diagnosis = Autopsy
- Brain scans
- Spinal fluid
- Mental status exam
- Nature and progression of the disease:
- “Nun study”
- Deterioration:
- Early and later stages = slow
- During middle stages = rapid
- Post-diagnosis survival = 8 years
- Onset = 60s or 70s
- 50% of the cases of neurocognitive disorder are found to be the result of Alzheimer’s disease
- Prevalence:
- 5 million Americans, several million worldwide
- Higher:
- Lower:
- Higher education
- American Indians - lower
- Diagnostic Criteria for Major or Mild Neurocognitive Disorder due to Alzheimer's Disease (DSM-5):
- A. The criteria are met for major or mild neurocognitive disorder.
- B. There is insidious onset and gradual progression of impairment in one or more cognitive domains.
- C. Criteria are met for either probable or possible Alzheimer's disease.
- D. The disturbance is not better explained by cerebrovascular disease, another neurodegenerative disease, the effects of a substance, or another mental, neurological, or systemic disorder.
Vascular Neurocognitive Disorder
- Vascular neurocognitive disorder is a progressive brain disorder that is a common cause of neurocognitive deficits
- Clinical description:
- Progressive brain disorder
- Blockage or damage to blood vessels
- Onset is often sudden
- Variable impairments
- DSM-5 Criteria:
- Cognitive disturbances:
- Speed of information processing and executive functioning
- Greater motor problems
- Significant impairments
- Prevalence:
- 1.5% in age 70 to 75
- 15% in age 80 or older
- Men > Women
- Higher rates of cardiovascular disease
- Most will require formal nursing care
- Death from infection
- Pneumonia
- Weak immune system
Other Medical Conditions That Cause Neurocognitive Disorder
- Frontotemporal degeneration
- Traumatic brain injury
- Lewy body disease
- Parkinson’s disease
- HIV infection
- Substance use
- Huntington’s disease
- Prion disease
- Normal pressure hydrocephalus (excessive water in the cranium, resulting from brain shrinkage)
- Hypothyroidism (an underactive thyroid gland)
- Brain Tumor
- Vitamin B12 deficiency
- Head trauma
- Accidents are the most common cause
- Memory loss is the primary symptom
- Chronic Traumatic Encephalopathy (CTE)
Frontotemporal Neurocognitive Disorder
- Damage the frontal or temporal regions of the brain affecting:
- Personality
- Language
- Behavior
- Two types:
- Declines in appropriate behavior
- Declines language
- Diagnostic Criteria for Major or Mild Frontotemporal Neurocognitive Disorder (DSM-5):
- A. The criteria are met for major or mild neurocognitive disorder.
- B. The disturbance has insidious onset and gradual progression.
- C. Either (1) or (2):
- Behavioral variant:
- Three or more behavioral symptoms (disinhibition, apathy, loss of empathy, etc.)
- Prominent decline in social cognition and/or executive abilities.
- Language variant:
- Prominent decline in language ability.
- D. Relative sparing of learning and memory and perceptual-motor function.
- E. The disturbance is not better explained by cerebrovascular disease, another neurodegenerative disease, the effects of a substance, or another mental, neurological, or systemic disorder.
- Probable vs. Possible frontotemporal neurocognitive disorder, based on genetic mutation evidence or neuroimaging.
Pick’s Disease
- Rare neurological condition
- 5% of all dementias
- Cortical impairment pattern
- Early onset = 40s or 50s
Traumatic Brain Injury
- Neurocognitive disorder due to traumatic brain injury - includes symptoms that persist for at least a week following the trauma, including executive dysfunction
- Diagnostic Criteria for Major or Mild Neurocognitive Disorder due to Traumatic Brain Injury (DSM-5):
- A. The criteria are met for major or mild neurocognitive disorder.
- B. Evidence of traumatic brain injury with one or more indicators (loss of consciousness, posttraumatic amnesia, etc.).
- C. The neurocognitive disorder presents immediately after the injury or recovery of consciousness and persists past the acute post-injury period.
Lewy Body
- Neurocognitive disorder due to Lewy body disease
- Lewy bodies are microscopic deposits of a protein that damage brain cells over time
- Diagnostic Criteria for Major or Mild Neurocognitive Disorder with Lewy Bodies (DSM-5):
- A. The criteria are met for major or mild neurocognitive disorder.
- B. The disorder has an insidious onset and gradual progression.
- C. Combination of core diagnostic features (fluctuating cognition, recurrent visual hallucinations, spontaneous parkinsonism) and suggestive diagnostic features (REM sleep behavior disorder, neuroleptic sensitivity).
- D. The disturbance is not better explained by cerebrovascular disease, another neurodegenerative disease, the effects of a substance, or another mental, neurological, or systemic disorder.
Parkinson’s Disease
- Degenerative brain disorder
- Dopamine pathway damage
- 1 out of 1,000 people worldwide
- Motor problems:
- Tremors
- Posture
- Walking
- Speech
- Sub-cortical impairment pattern
- Not all with PD will develop dementia
- Diagnostic Criteria for Neurocognitive Disorder due to Parkinson's Disease (DSM-5):
- A. The criteria are met for major or mild neurocognitive disorder.
- B. The disturbance occurs in the setting of established Parkinson's disease.
- C. There is insidious onset and gradual progression of impairment.
- D. The neurocognitive disorder is not attributable to another medical condition and is not better explained by another mental disorder.
- Criteria for probable vs. possible neurocognitive disorder.
Human Immunodeficiency Virus-Type 1 (HIV-1)
- Clinical description:
- HIV causes neurological impairments and dementia
- Cognitive slowness
- Impaired attention
- Forgetfulness
- Clumsiness
- Repetitive movements
- Tremors/leg weakness
- Apathy
- Social withdrawal
- Later stages of HIV infection
- Fewer with HAART medications
- 10% patients now experience neurocognitive disorder
- Sub-cortical dementia
- Motor skill impairments
- Slowing
- Anxiety
- Depression
- No aphasia
- Diagnostic Criteria for Major or Mild Neurocognitive Disorder due to HIV Infection (DSM-5):
- A. The criteria are met for major or mild neurocognitive disorder.
- B. There is documented infection with human immunodeficiency virus (HIV).
- C. The Neurocognitive Disorder is not better explained by non-HIV conditions.
- D. The neurocognitive disorder is not attributable to another medical condition and is not better explained by a mental disorder.
Huntington’s Disease
- Genetic autosomal dominant disorder
- Chromosome 4
- Early onset = 40s or 50s
- Motor symptoms:
- Sub-cortical dementia pattern
- Diagnostic Criteria for Major or Mild Neurocognitive Disorder due to Huntington's Disease (DSM-5):
- A. The criteria are met for major or mild neurocognitive disorder.
- B. There is insidious onset and gradual progression.
- C. There is clinically established Huntington's disease, or risk based on family history/genetics.
- D. The neurocognitive disorder is not attributable to another medical condition and is not better explained by another mental disorder.
Prion Disease
- Always fatal
- Not contagious in humans
- Cannibalism
- Blood transfusions
- Linked to mad cow disease
- Diagnostic Criteria for Neurocognitive Disorder due to Prion Disease (DSM-5):
- A. The criteria are met for major or mild neurocognitive disorder.
- B. There is insidious onset, and rapid progression is common.
- C. Motor features of prion disease or biomarker evidence.
- D. The neurocognitive disorder is not attributable to another medical condition and is not better explained by another mental disorder.
Creutzfeldt-Jakob Disease
- Type of Prion disease
- 1 in 1,000,000
- Linked to mad cow disease
- 10 new cases of variant Creutzfeldt-Jakob Disease - United Kingdom
Substance/ Medication-Induced Neurocognitive Disorder
- Substance-induced persisting dementia
- Drug use plus poor diet
- Alcohol, inhalants, sedatives, hypnotics, anxiolytics
- Brain damage may be permanent
- Symptoms similar to other neurocognitive disorders:
- Aphasia
- Apraxia
- Agnosia
- Executive function impairments
- Diagnostic Criteria for Substance/Medication-Induced Major or Mild Neurocognitive Disorder (DSM-5):
- A. The criteria are met for major or mild neurocognitive disorder.
- B. The neurocognitive impairments do not occur exclusively during delirium and persist beyond usual intoxication/withdrawal.
- C. The substance and duration/extent of use are capable of producing the impairment.
- D. The temporal course is consistent with the timing of substance use and abstinence.
- E. The neurocognitive disorder is not attributable to another medical condition and is not better explained by another mental disorder.
Causes of Neurocognitive Disorder
- Early, unsupported views:
- Alzheimer’s disease is the most common cause of neurocognitive disorder
- Neurobiological influences:
- Neurofibrillary tangles
- Amyloid plaques
- Neuritic or senile plaques
- Spinal fluid
- Cortical atrophy
- Genetic influences:
- Polygenetic
- Chromosomes 21, 19, 14, 12, 1
- Chromosome 14—early onset
- Chromosome 19—late onset
- Deterministic genes:
- Preseniln-1, Preseniln-2
- β-amyloid precursor protein
- APP gene on chromosome 21
- APP produces amyloid in plaques
- Down syndrome and Alzheimer incidence
- Two mechanisms that may account for amyloid protein buildup:
- Amyloid precursor protein (APP)
- Apolipoprotein E (apo E)
- Susceptibility genes:
- apoE2
- apoE3
- apoE4
- Chromosome 19
- Late-onset Alzheimer’s
- Higher prevalence in families with Alzheimer’s
- Interaction with head injuries
Psychosocial and Social Influences
- Not direct causes
- Influence onset and course
- Lifestyle factors:
- Drug use
- Diet
- Exercise
- Stress
- Cultural factors:
- Variable risk for diseases and accidents
- Ethnicity
- Economic conditions
- Psychosocial factors
Treatment
- Early intervention is critical
- Three areas of focus:
- Prevent certain conditions
- Delaying onset
- Cope with the advancing deterioration
- Multidimensional treatment
- Focus on slowing the progression
- Caregivers
- Anxiety disorders
- Psychotropic medications use
- Biological treatment:
- Neuronal preservation and restoration
- Glial cell-derived neurotrophic factor
- Transplanting fetal brain tissue
- Cholinesterase-inhibitors
- New drugs that target the beta amyloid (plaques)
- Ginkgo biloba
- Antioxidants
- Vitamin E dangerous in high dose
- Antidepressants
- Antipsychotics
- All are only modestly effective for short periods
- Immune system
- Psychosocial treatments
- Focus on enhancement
- Dementia patients
- Families
- Caregivers
- Cognitive stimulation
- Teach adaptive skills
- Memory enhancement prosthetic devices
- Main emphasis:
- Coping strategies
- Behavioral interventions
- Implementing technology
- Stress reduction
- Dealing with depression
- Preventing elder abuse
- Supportive counseling for family
Prevention
- Reducing risk of dementia
- Controlling blood pressure
- Do not smoke
- Staying socially active
- Staying physically active
- New and hopeful research continues