Venous Return
CIRCULATORY SYSTEM OVERVIEW
Topic Focus: Venous Return/Cardiac Output
Content Sections:
Venous Return
Cardiac Output
Factors Affecting Cardiac Output
Measurement of Cardiac Output
Presented by Dr. S B Phiri
VENOUS RETURN
Definition: Venous return (VR) is the flow of blood back to the heart.
Equilibrium: Under steady-state, venous return equals cardiac output (Q).
This is crucial as the cardiovascular system is a closed loop.
Prevents blood accumulation in systemic or pulmonary circulations.
Interdependence: Cardiac output and venous return may be regulated independently.
Factors Affecting Venous Return
Musculovenous Pump:
Rhythmic contraction of limb muscles during activities (e.g., walking) enhances VR through the muscle pump mechanism.
Decreased Venous Capacitance:
Sympathetic activation reduces venous compliance and increases venous return via:
Increased vasomotor tone
Increased central venous pressure
Augmented cardiac output (Frank-Starling mechanism).
Respiratory Pump:
During inspiration:
Negative intrathoracic pressure and positive abdominal pressure create a gradient, pulling blood towards the right atrium.
Vena Cava Compression:
Increased resistance in the vena cava (e.g., during Valsalva maneuver) reduces venous return.
Gravity:
Standing causes hydrostatic pressure changes:
Right atrial pressure decreases.
While venous pressure in dependent limbs increases, overall VR can decrease due to lowered cardiac output.
Pumping Action of the Heart:
Changes in right atrial pressure affect central venous pressure and, subsequently, venous return.
CARDIAC OUTPUT
Definition: Cardiac output (CO) is the volume of blood ejected by the heart per time unit (L/min).
Normal resting values: 5 to 6 L/min, about 8% of body weight per minute.
Cardiac Index: CO adjusted for body surface area; increases proportionally with body surface area.
Average cardiac index is ~3 L/min/m² for a normal 70 kg adult.
Calculation: CO = Heart Rate (HR) x Stroke Volume (SV).
Stroke Volume (SV)
Definition: Volume of blood ejected from one ventricle per beat (mL/beat).
Calculation: SV = End Diastolic Volume (EDV) - End Systolic Volume (ESV).
Healthy SV is typically approx. 70 mL for each ventricle.
Ejection Fraction (EF): Percentage of blood ejected per contraction; an important metric for heart function and heart failure assessment.
Left Ventricular Ejection Fraction (LVEF): Volume pumped by left ventricle per beat / Volume at end of diastolic filling.
Affected by overall heart activity; reductions indicate heart failure.
FACTORS AFFECTING CARDIAC OUTPUT
Heart Rate Influences:
Age: Newborns (HR ~ 120 bpm); rates decrease through youth then increase with age.
Exercise: Increases HR and SV during physical activity.
Pathological Rates:
Bradycardia: Reduced rate; can be intrinsic or extrinsic causes.
Tachycardia: Increased rate; often linked to stress or physiological conditions.
Autonomic Regulation:
Controlled by cardiovascular centers in the medulla oblongata (sympathetic and parasympathetic effects).
Broken Heart Syndrome:
Emotional events can weaken myocardium without oxygen lack.
Other Influences: Hormonal levels, ion concentrations, temperature, hypoxia, and pH.
Stroke Volume Influences:
Preload: Stretch on ventricles before contraction; increased EDV results in increased SV.
Contractility: Strength of ventricular contraction affected by:
Positive inotropic factors (e.g., catecholamines, some drugs).
Negative inotropic factors (e.g., certain drugs, hypoxia).
Afterload: Resistance against which ventricles must pump; increased afterload demands stronger contractions.
MEASUREMENTS OF CARDIAC OUTPUT
Direct/Invasive Methods:
Fick method
Indicator dilution method
Indirect/Non-invasive Methods:
Echocardiography
Computed Tomography
Radionuclide Imaging
Fick’s Principle: Blood flow equals difference in arterial and venous substance concentrations multiplied by blood flow rate.