Cell Cycle
Cell Division Notes
Mitosis and Cell DIvision produces two identical daughter cells
The goal of mitosis and cell division is growth, repair, and development.
Ratio of Surface Area to Volume
A ratio is a comparison
Surface area/Volume
Volume = l x w x h
6:1
3:1
2:1
We want a higher ratio of surface area to volume
The greater the SA/V ratio, the easier it is for the cell to exchange materials
Cell Size and Material Exchange
The larger a cell grows the surface area/volume ratio decreases, which makes cells less efficient at…
Moving enough nutrients in
Moving waste materials out
The Cell Cycle and Mitosis
The cell cycle is divided into two halves
Interphase: period of the cell cycle between cell divisions
M phase: Period of time when mitosis and cell division occurs
Parts of Interphase
G1 Phase (Gap 1)
Growing
Synthesize new proteins and organelles
Doing their jobs
Longest phase of cell cycle
S Phase (Synthesis)
Chromosomes (DNA) are replicated
Key proteins associated with replication are made (centromeres)
G2 Phase (Gap 2)
Shortest of the 3 phases of interphase
Organelles and molecules for cell division are produced (centrioles)
Check-up phase before mitosis
Chromatin
Chromatin: A complex of DNA and proteins in the cell nucleus that condenses to form chromosomes during cell division
Chromosomes: Condensed chromatin
Chromosomes Replication
During the cell cycle (before division can take place) chromosomes are replicated to form an identical copy of itself
Two identical copies of a chromosome are called
“Sister” chromatids” - a replicated chromosome
Centromere - the protein where the chromatids of a chromosome are attached
Chromosomes
Different organisms have different numbers of chromosomes
Human cells - 46
Fruit Fly Cells - 8
King Crab Cells - 206
Prokaryotes have one circular chromosome and replicate via binary fission
Mitosis
Prophase
Chromatin condenses into chromosomes
Nucleoli and nuclear envelope break down
Spindle fibers grow from centrioles
Centrioles migrate to opposite poles of cell
Metaphase
The chromosomes are lined up at the equator of the cell
The spindle fibers from each centriole are attached to the centromeres of the chromosomes
The nuclear membrane has disappeared
Anaphase
The centromeres split, and the sister chromatids separate as each is pulled to an opposite pole
Telophase
The chromosomes become longer, thinner, and less distinct
New nuclear membranes form
The nucleolus reappears
Cell division is nearly complete
Cytokinesis
Cytoplasmic division and other changes exclusive to nuclear division that are part of mitosis or meiosis
Regulating the Cell Cycle
Internal Regulation:
-Cyclins
CDK’s (Cyclin Dependent Kinases
External Regulation:
Growth Factors HGH
Platelet Driven Growth Factors
Produced by platelet blood cells
- Density-Dependent Inhibition
-Anchorage-Dependent Inhibition
Density-Dependent Inhibition: No more space to reproduce (stops when they contact other cells)
Anchorage Dependence: To divide they must be anchored to a substratum (Bones or other sorts of tissue)
Cancer Cells
Free of both density-dependent inhibition and anchorage dependence
Divide excessively and invade other tissues because they are free of the body’s control mechanisms
How do cells know when to divide or not?
Internal Regulators - the proteins that respond to events inside the cell
Cyclins - Control the activation of enzymes
CDK’s - Cyclin dependent kinases - enzymes that control the cell cycle
External Regulators - Proteins that respond to events outside of the cell are called external regulators
Growth factors are among the most important external regulators
Molecules found on the surfaces of neighboring cells often have an opposite effect, causing cells to slow down or stop their cell cycles
Cancer
Tumor - a mass of cancer cells
Benign tumor - a tumor that does not invade it's surrounding tissue or spread around the body
Malignant tumor - a tumor that may invade it's surrounding tissue or spread around the body
Cancer has many causes:
Smoking
Radiation
Viral infections
Genetics
The common threat in all cancers is that control over the cell cycle has broken down
Over 50 percent of cancer cells have a defect in the p53 gene (anti-oncogne)
Proto-Oncogene
When proto-oncogenes get mutated, cancer usually follows
Oncogenes:
Proto-oncogene = ras
Oncogene = mutated ras
Always activated
Always stimulating
Proliferation
P53 Antioncogene
P53 is a tumor suppressor gene found mutated in about half of human cancers
It encodes a gene regulatory protein that is activated by damaged DNA and is involved in blocking further progression of the cell cycle
Cancer Treatments
Surgery: Remove the tumor
Radiation: Kill the cancer through radiation
Hormone Therapy
Chemotherapy
Targeted Therapy
Cell Growth Vs Development
Growth and Repair
New daughter cells divide from existing cells to form identical cells
Development
Stem cells
New daughter cells divide from existing cells, but changes occur to form new types of cells
Blood
Bones
Skin
Nervous
Differentiation - process of stem cells changing into specific cell types
Stem Cells
Totipotent (omnipotent) - can become entire organism (first four divisions after fertilization)
Embryonic Stem Cells
Pluripotent - can differentiate into nearly all cells / not full organism
Inner cells mass from blastocyst (4-5 days after fertilization)
3. Adult Stem Cells
Multipotent - a number of cell types
Oligopotent - a few cell types
Unipotent - one cell type
4. Induced Pluripotent Stem Cells
Created in lab settings. Adult somatic cells (usually skin cells) are reverted back to pluripotent form via treatment
Asexual Reproduction - reproduction through mitosis or binary fission
Fast reproductive rate but little variation (2 identical daughter cells)
Sexual Reproduction
Reproduction through the production of gametes (sperm and eggs) by the process of Meiosis. Slower Reproductive rates, but increased variation