Neurogenetics 520 -- Ataxia Telangciestca
Overview of Study
Focus on understanding the relationship between the STRING gene and ATM protein in neuron behavior.
Investigated the effects of 650 newtons of a specific condition on eye development in mutant Drosophila flies.
Key Concepts
Neuron Development and Mutations
Lemmatidia Rows: Observed fewer nerve degeneration signs (degenerated photoreceptor neurons) with specific size comparisons in eyes.
Mutants: Identified six different mutants, including suppressors and STRING (cd c15) gene affecting cell cycle regulation.
Role of STRING in Cell Cycle
Cell Cycle Phases: G1 phase (diploid, 2n), S phase (DNA replication, 4n), G2 phase, mitosis.
STRING promotes transition from G1 to S phase, preventing excessive cell division in neurons, which are typically post-mitotic.
Loss of ATM inhibits STRING's ability to facilitate the G1 to S transition, hence preventing unwanted neuron proliferation.
Experimental Observations
DNA Content Measurement
Employ FAC (Fluorescent Activated Cell Sorting) to quantify DNA levels in cells from the eye imaginal disc.
Assuming neurons should only maintain 2n DNA content unless undergoing replication.
Genetic Experiments
Used heterozygous mutations for ATM to study cell cycle re-entry in neurons.
Predicted and confirmed STRING mutations reduced cell cycle re-entry phenotype associated with reduced ATM function.
Conclusions and Future Directions
Loss of ATM may lead to inappropriate cell cycle re-entry and has implications for DNA damage responses in neurons.
Examined connections between cell cycle regulation and innate immune responses in the brain, with some speculation towards potential therapeutic applications of ATM mutations in disease contexts.
Further research needed on the relationship between immune responses and cell cycle behaviors in neurons.