Protein Metabolism and Metabolic States of the Body
- Proteins are continually broken down and replaced.
- Amino acids are recycled or converted to other compounds.
- Excess dietary proteins are oxidized for energy or converted to fat for storage.
- Amino acid degradation:
- Deamination: NH2 removal, converted to pyruvic acid or keto acid intermediates.
- Transamination: Amine group transfer to α-ketoglutaric acid, forming glutamic acid; reversible.
- Oxidative deamination: In liver, glutamic acid's amine group becomes ammonia (NH3), converted to urea and excreted.
- Keto acid modification: Keto acids become metabolites for the citric acid cycle, contributing to gluconeogenesis.
- Protein Synthesis
- Amino acids are anabolic nutrients for proteins.
- Synthesis is hormonally controlled, requiring a complete set of amino acids (essential ones from diet).
Metabolic States of the Body
- Catabolic-Anabolic Steady State
- Organic molecules are continuously broken down and rebuilt.
- Nutrient pools (amino acids, carbohydrates, fats) are interconvertible.
- Amino acid pool: source for protein resynthesis, derivatives, and gluconeogenesis.
- Carbohydrate and fat pools: easily interconverted; excess stored; oxidized for energy.
- Absorptive State (Fed State)
- Anabolism exceeds catabolism; lasts ~4 hours after eating.
- Excess nutrients stored as fats.
- Carbohydrates: Glucose is main fuel, converted to glycogen or fat.
- Triglycerides: Hydrolyzed to fatty acids and glycerol.
- Amino acids: Deaminated or used for protein synthesis.
- Hormonal control: Insulin promotes glucose uptake, glycogenesis, lipogenesis, and protein synthesis; inhibits gluconeogenesis.
- Postabsorptive State (Fasting State)
- Catabolism exceeds anabolism; energy from body reserves.
- Goal: Maintain blood glucose, promote fat use for energy.
- Sources of blood glucose: Glycogenolysis (liver, muscle), lipolysis, catabolism of cellular protein.
- Glucose sparing: Conserves glucose for brain by using noncarbohydrate sources.
- Hormonal control: Glucagon promotes glycogenolysis and gluconeogenesis; sympathetic nervous system triggers fat mobilization.