BI-112 Virus-Viroid Wi25
VIROIDS
Origins of Life
First Cellular Life: ~3.5-3.8 billion years ago (bya)
Pre-cellular life molecules: ~4 billion years ago
Key players: RNA, lipids, proteins
Jack Szostak's contributions to proto-cell formation
RNA World Hypothesis
Video resources available on RNA world
Microscopy evidence of vesicle growth and division
Model protocell membranes consisting of fatty acid vesicles from various acids:
Oleic acid
Myristoleic acid
Capric acid
Characteristics of Viroids
Defined as "rogue" RNAs, first identified in 1971
Structure: naked circular RNA, 200-400 nucleotides long
Size: Approximately 10 times smaller than the smallest virus
Unique feature: Lack of genes (non-coding RNA)
Self-replication capabilities are partial
Pathogenic Nature of Viroids
Rogue RNAs are known to cause diseases in plants
99% of known viroids affect plant life
Transmission methods:
Insects that bite plants
Spread through cell-to-cell communication and vascular tissues
Examples of Plant Diseases Caused by Viroids
Notable diseases:
Citrus exocortis
Potato spindle tuber
Cadang-cadang in coconuts
Avocado sunblotch
Tomato planto macho
Hundreds of viroid varieties exist, with characterized images available.
Theories of Viroid Origin
Explanations for viroids' origin:
Considered relict or "re-invention" from the RNA world
Proposed endogenous origin: escaped RNA from plants or viruses
Genetic Complexity
Introns discussed as non-coding regions in host genes:
Sequence flow from gene DNA to mRNA, excluding introns before translation
Viroids in Humans
Unique instance of viroid-like RNA in humans:
Delta agent within Hepatitis B virus
Causes Hepatitis D through interaction with the Hepatitis B virus
VIRUSES
What is a Virus?
Historical Context
Early mentions of variolation and vaccine development:
Edward Jenner's work (utilized cowpox with subjects like Blossum and Sarah Nelms)
Size Comparison of Cells and Viruses
Eukaryotic Cell: 0.01 mm (10 μm)
Bacterium: 0.001 mm (1 μm)
Virus: 0.0001 mm (100 nm)
Virus Structure
Composed of nucleic acid (single or double-stranded DNA or RNA)
Surrounded by a protein coat known as a capsid (an essential feature of all viruses)
Possible additional structures:
Sometimes includes "stuffing" with other proteins packed around the nucleic acid core
Special enzymes may be present, important for infection and replication
Outer envelope that may be present:
Lipid bilayer derived from the host cell's membrane
Non-enveloped viruses have surface proteins, some from the virus itself.
Are Viruses Alive?
Arguments against viruses being classified as living:
Not composed of cells
Metabolically inert (no energy-producing reactions)
Cannot reproduce independently without a host
Can be crystallized like pure compounds
Synthetic creation of viruses is achievable (example: Polio, 2002)
Possible argument in favor of the viral existence:
May form independent genetic lineages with hereditary traits and exhibit adaptation/evolution.
Origin of Viruses
Lack of fossil evidence leads to three main theories:
Viruses as relics of the proto-cell world.
Evolution from parasitic bacteria that streamlined over time.
Origin from escaped genetic material (instead of introns).
Virus Infection & Replication
Hosts for viruses are ubiquitous; found in every known organism.
Transmission methods include:
Water, food, and body fluids
Airborne vectors like mosquitoes
Phases of viral infection:
Attachment to host cells
Entry through membrane fusion or endocytosis (Trojan horse method)
Attachment Mechanism
Lock & Key mechanism described:
Virus protein identifies and docks with a specific host receptor protein.
Some viruses may require two different receptors for successful binding.
Penetration Processes
Mechanisms for viral entry into cells:
Direct fusion with lipid membrane if present.
Endocytosis via binding to specific membrane receptors.
Plant Virus Entry
Plant cells possess a protective wall; entry facilitated via:
Injection from insects
Breakage in the wall, allowing flow through plasmodesmata.
Composition of Viruses
All viruses consist of:
DNA or RNA
Protein coat
An outer lipid envelope (if present)
Hence, potential answers to questions on virus composition include all available structures.
Vaccine Etymology
"Vacca" meaning cow refers to the origins of the word vaccine.
Virus and Bacterial Interactions
Statement about bacteria's capabilities concerning viruses:
Bacteria cannot "catch" a virus (False claim).
Cellular Machinery hijacking by Viruses
Viruses hijack host machinery to replicate via:
Strategies outlined in the Baltimore Virus Classification System:
Double-stranded DNA viruses (e.g., Herpes, Hep B) integrate into host genome.
Single-stranded RNA viruses (e.g., influenza, HIV) either function directly as mRNA or utilize reverse transcription for integration.
Assembly and Release of Viruses
The lifecycle of viral replication includes:
Assembly of viral components at sites like ER and Golgi within the host cell.
Release methods vary: Lytic infections lead to quick cell lysis while persistent and lysogenic forms incorporate an extended silent phase.
Example Viruses and Characteristics
Virus profiles include:
Influenza (ssRNA with fragmented genome)
Coronavirus (structure and replication details)
Certain cancer-causing viruses (associated viral connections)
HIV specifics regarding infection and replication pathways.