L12 - Higher Order Cognition
Case study link: Graham Partridge with orthorexia nervosa
Cognitive content as a source of pathology is limiting - we should extend to functions of cognition (in a processing sense (not bio/lesion sense) -what brain is doing)
Look at transdiagnostic approaches to PP to move away from putting everything into one box but have some aspects of functioning that transcend this
Transdiagnostic Perspective and Classification Limitations
High comorbidity rates across psychiatric diagnoses (e.g., meeting criteria for one personality disorder typically confers a high probability of meeting criteria for four) indicate fundamental flaws in categorical classification systems.
Transdiagnostic approaches shift focus from disorder-specific cognitive content to generalized cognitive functions and processing mechanisms that operate across diagnostic boundaries.
Eating Disorders
Anorexia Nervosa:
Restriction of food
Intense fear of gaining weight
Perceptual biases in the way they see body weight and shape which affects self-worth
Bulimia Nervosa:
Recurrent binge eating episodes (eating a lot and lack of control during episode)
Inappropriate compensatory behaviours (exercise, purging) to make up for binge
Binge and compensatory behaviour occur at least once a weekf or 3 weeks
Self-evaluation is based on body shape and weight
Not exclusively happening during anorexia episodes
Orthorexia Nervosa: Righteous appetite, characterized by:
Extreme preoccupation with consuming healthy foods, and rigid, egosyntonic avoidance of foods perceived as toxic or unhealthy.
Belief of physical or spiritual purity through dietary adherence (for some)
Deriving self-worth, control, pride, or identity from adherence; failure triggers severe self-reproach, guilt, and punishing compensatory behaviors.
Substantial expenditure of time and financial resources, resulting in social/occupational impairment.
Core Shared Mechanisms: All three conditions share rigid self-evaluative belief systems and behavioral routines designed to ccognitive dissonance (the discomfort arising from discrepancies between internal beliefs and behaviors).
Shows shared features across diagnoses (leads to need for transdiagnostic approaches)
Beyond Cognitive Content
Psychological disorders may be better understood by cognitive processes/functions, rather than just specific thought content
Locus of pathology (where problem lies)
Behaviour theory: learning mechanisms are normal, what has been learned is abnormal
Beckian theory: schema activation process is normal, schema content (outcome) is not
Classification of disorders
Behaviour theory = transdiagnostic
The same learning principles can explain many disorders
Focuses on common processes across diagnoses
Beckian theory = schema content is disorder-specific
Can explain each disorder by having a particular behaviour of congitive content that accounts for it
Constrained approach
Metacognition: "Thinking about thinking" - critical for resource allocation towards thinking.
Meta-cognitive phenomena questionnaire where each fator is focused on some aspect of maladaptive meta-cognitive function
Assessed via five distinct domains:
Cognitive confidence (trust in one's own memory).
Positive beliefs about worry (perceived coping utility of worry).
Cognitive self-consciousness (habitual monitoring of thought processes).
Uncontrollability and danger of thoughts.
Need to control thoughts.
The Self-Regulatory Executive Function (SREF) Model
Doesn’t focus just on cognitive content - considers the cognitive process
Core features:
Normal and abnormal (what changes when there’s PP)
Content and process - now also considers:
Attention (the way attentional resources are allocated (automatic or controlled)
Memory
Meta-cognition
Instrumental transitory states
Called configurations (sometimes modes)
Temporary thinking state ppl enter bc they think it will achieve change/a goal (e.g. Worry to prepare for danger, worry to solve a problem)
When maladaptive: gets stuck in the configuration where it becomes persistent and unhelpful (no longer transient)
Formulated by Wells and Matthews, describing cognitive operations across three levels:
Low-Level Processing Units: Fast, automatic, resource-independent processing.
External, body, and cognitive info
Memory Systems (beliefs): Declarative self-beliefs (positive, negative or neutral) and beliefs about procedural plans/rules (how should I do this).
SREF Configuration: Controlled, resource-dependent processing responsible for executive control, and especially identification and resolution of discrepancy/cognitive dissonance
Processing continues until dissonance is solved and then SREF goes “offline”
SREF model proposal that PP disorders are maintained not by just the thought content - but by maladaptive self-regulation strategies that become activated when people perceive a discrepancy between their current state and their goals/beliefs
Memory and SREF configuration are at a controlled processing level (resource-dependent domain) and low-level processing units operate somewhat automatically (fast, crude output)
Maladaptive SREF Outcomes in Pathology:
When the SREF configuration fails to terminate, remaining perpetually active:
Belief elaboration breaks down bc they don’t expose selves to situations that show their belief was wrong
If they are exposed → discount that evidence
Attentional bias toward threat cues and chronic active worrying/rumination.
Intensification or suppression of activity (more worrying, avoidance, thought suppression etc)
SREF Summary:
Trigger → discrepancy detected → beliefs activated → coping strategy (worry/rumination/etc.) → attempt to resolve discrepancy → resolution
Or, if the strategy becomes persistent:
Attempt to resolve discrepancy → more monitoring of thoughts/body/threats → more coping strategy → distress maintained (PP)
Treatment Implications using SREF
Therapeutic Targets:
Disrupting Rumination: Teaching individuals to identify and interrupt perseverative worry loops without removing exposure to worry-activating situations
Avoidance doesn’t help correct maladaptive behaviour - need to step back in meta-cog sense and realise rumination
Detached Mindfulness: A distancing that facilitates meta-cognitive reflection on what’s happening
Belief Modification: Directly restructuring both core self-beliefs and metacognitive beliefs (e.g., challenging the belief that worry is protective or uncontrollable).
Response Plan Alteration: Establishing alternative, adaptive procedures to resolve distress and allow SREF deactivation.
Attention Training: Training conscious reallocation of attention away from self-monitoring and to something more adaptive.
Exposure to Activators: Exposing individuals directly to feared stimuli without permitting compensatory neutralizing behaviors to reduce avoidance.
Textbook Readings:
1. Relational Frame Theory & ACT (Bennett, pp. 66–72)
Relational Frame Theory (RFT):
Humans naturally link words, thoughts, and real-world events together (relational framing).
Cognitive Fusion: Treating thoughts as literal truths rather than temporary mental events (e.g., believing "I am a failure" instead of noticing "I am having the thought that I am a failure").
Experiential Avoidance: Trying to suppress or run away from uncomfortable thoughts and feelings, which causes psychological rigidity and makes distress worse over time.
Acceptance and Commitment Therapy (ACT):
Aim: To build psychological flexibility rather than fighting or eliminating thoughts.
Acceptance: Allowing feelings and thoughts to exist without trying to control them.
Cognitive Defusion: Stepping back and seeing thoughts merely as words or ideas.
Contact with the Present Moment: Being mindful of the "here and now" without judgment.
Values: Clarifying what truly matters most in life.
Committed Action: Taking practical steps aligned with personal values.
Overlap with the S-REF Model:
Both ACT and Wells' Self-Regulatory Executive Function (S-REF) model agree that psychopathology is maintained by rigid coping habits and avoidance, rather than just negative thought content alone.
2. Eating Disorders: Anorexia and Bulimia (Bennett, pp. 312–321)
Anorexia Nervosa:
Severely restricted food intake resulting in significantly low body weight.
Intense fear of weight gain and distorted evaluation of body shape and size.
Subtypes: Restricting type (dieting, fasting, exercise) and Binge-eating/purging type.
Highest mortality rate among psychiatric disorders, frequently linked to cardiac issues and suicide.
Bulimia Nervosa:
Repeated episodes of out-of-control binge eating (eating large amounts of food in short periods).
Followed by compensatory behaviors (purging, misuse of laxatives, fasting, or extreme exercise) at least once a week for 3 months.
Body weight is usually in the normal range, but self-esteem is excessively tied to weight and appearance.
Causes and Contributing Factors:
Biochemical & Gut Microbiome:
Dysregulation in the hypothalamus and neurotransmitters (dopamine alters reward and pleasure; serotonin influences satiety and mood).
Starvation in anorexia reduces serotonin, temporarily relieving anxiety.
Gut bacteria imbalances affect appetite hormones and neurotransmitter production.
Socio-Cultural & Family:
Societal pressures idealizing thinness.
Families with high conflict avoidance, over-protectiveness, rigid standards, or parental perfectionism.
Cognitive Factors (Fairburn):
Rigid rules around food and weight-related self-worth.
In bulimia, breaking an unrealistic dietary rule triggers "all-or-nothing" thinking, leading to a binge.
Psychological Autonomy (Bruch):
Early parenting that fails to respond to a child's internal cues leads to poor emotional awareness and a desperate need to control body shape.
3. Māori Mental Health Transformations (Durie, pp. 35–47)
Historical Transformations:
De-institutionalisation (1970s–1990s): Psychiatric asylums were closed in favor of community care, but poor planning led to "revolving-door" hospital admissions and homelessness.
Indigenisation: Addressing the post-1970s surge in Māori psychiatric admission rates by incorporating Treaty of Waitangi principles (partnership, participation, protection).
Key Models and Milestones:
Te Whare Tapa Whā (1982): A holistic health model based on four cornerstones:
Taha wairua (spiritual)
Taha hinengaro (mental/emotional)
Taha tinana (physical)
Taha whānau (family/social)
Whaiora Unit (1983) & Hui Whakaoranga (1984): Pioneered culturally grounded clinical units and established national health strategies led by and for Māori.
Workforce Growth: Training community health workers and funding Māori mental health professionals.
Practising at the Interface:
Integrating clinical science with indigenous knowledge (tikanga and mātauranga).
Ensuring culturally sensitive engagement and assessment (e.g., the Hua Oranga outcome measure that assesses all four dimensions of health across patient, whānau, and clinician perspectives).
The Next Frontier — Whānau Ora & Mauri Ora:
Shifting from reactive clinical treatment to addressing root social and environmental determinants (housing, education, poverty) through primary care.
Whānau Ora: Placing the family at the center of health solutions using navigators.
Mauri Ora (Flourishing): Moving beyond recovery from illness (mauri noho / languishing) toward holistic vitality, wellness, and self-determination (mauri ora / flourishing).