shock recognition and management
circulation - consider shock, affects vital organs
neurogenic
septic
toxic
cardiogenic
hypovolemic
haemorrhagic
treat shock before tq replacement
shock - o2 demands of cells aren’t met, leading to hypoxia - most likely to present as hypotension
s + s
weak or absent radial pulse major sign
pulse assessment (thready, faint, irregular)
altered mental state (avpu ever 15 mins) major sign
control bleeding, confirm all indications are effective
better to prevent shock than treat it - deal with it early. don’t wait for s+s to occur
hypovolemic shock - not enough volume circulating body
haemorrhagic
d + v
diabetic ketoacidosis
burns
disruptive shock - not enough perfusion due to maldistribution
neurogenic shock
anaphylaxis
sepsis
obstructive shock - not enough cardiac output due to mechanical obstruction
pulmonary embolism
tptx
cardiac tamponade
acute ivc or svc obstruction
cardiogenic - not enough cardiac output due to cardiac failure
mi
late sepsis
overdose
heart block
hypothermia - cold, give blanket
coagulopathy - loss of clotting factors in platelets
acidosis - buildup of lactic acid, give fluids (to make up volume, not to replace blood)
refractory shock - fatal manifestation of cardiovascular failure, inadequate response to shock interventions leads to hypotension and organ failure
s+s of tptx
thoracic trauma
resp distress
absent breath sounds
spo2 <90
if not already treated for tptx, do needle decompression if not responding to fluids