Chapter 34: The Sensory System Vocabulary Flashcards
Fundamentals of the Sensory System
General Purpose of the Sensory System
Allows humans to perceive, interact with, and experience the surrounding environment.
Serves a critical biological role in protecting the body from environmental hazards and harm.
The Five Primary Senses
Sight (Vision)
Hearing (Audition)
Taste (Gustation)
Smell (Olfaction)
Touch (Somatosensation)
Somatosensation and Pain Perception
Mechanisms of Touch
Tactile sensory nerves are embedded throughout the layers of the skin.
Tactile sensitivity varies across different anatomical regions of the body.
Deep touch sensory nerves detect mechanical pressure and signal the body to alter physical positions to relieve pressure and prevent tissue ischemia.
Pathophysiology of Pain
Pain functions as the body's primary distress signal indicating potential or actual damage.
Referred (Radiating) Pain
Pain perceived in an anatomical location distant from the true origin or site of tissue injury.
Example: Radiating left arm pain experienced during a myocardial infarction (heart attack).
Phantom Pain
Neuropathic pain or sensory perception experienced in a limb or body part that has been surgically or traumatically amputated.
Chemical Senses: Taste and Smell
Gustatory System (Taste)
Taste receptors (tastebuds) are situated primarily on the surface of the tongue.
Five distinct taste modalities:
Sweet
Salty
Bitter
Sour
Umami (savory)
Olfactory System (Smell)
Sense organs and chemical receptors responsible for olfaction are located within the superior nasal cavity.
Age-Related Alterations and Geriatric Implications
The total number of operational chemoreceptors for taste and smell declines progressive with age.
Reduction in chemoreceptor density decreases the intensity of gustatory and olfactory sensations.
Diminished taste and smell intensity leads to a reduced appetite in elderly individuals.
Geriatric Risk: Decreased appetite predisposes geriatric clients to severe nutritional deficiencies, unintended weight loss, and dehydration.
Anatomy and Physiology of Vision
Primary Functions of the Eye
Detects light levels, spatial forms, visual colors, and physical shapes.
Structural Layers (Tunics) of the Ocular Globe
Sclera (Outer Layer)
Tough, fibrous protective outer coating, commonly referred to as the "white of the eye."
Cornea: The transparent anterior portion of the sclera that allows ambient light rays to enter the eye.
Choroid (Middle Layer)
Vascular layer containing blood vessels that supply oxygen and essential nutrients to eye tissues.
Ciliary Body: Tiny muscular structures connected directly to the lens; contracts and relaxes to alter lens shape and curvature, enabling visual accommodation and focusing.
Iris: Pigmented colored ring of the eye containing a central dynamic aperture called the pupil; regulates the volume of light entering the inner eye by adjusting pupil diameter.
Retina (Innermost Layer)
Sensory layer housing specialized photoreceptor cells:
Rods: Responsible for vision in dim light and black-and-white perception.
Cones: Responsible for high-acuity color vision and detail.
Transduces light energy into electrical nerve impulses.
Nerve impulses travel out of the eye along the optic nerve directly to the visual cortex of the brain for interpretation.
Internal Fluid Chambers
Anterior Chamber
Located in the space between the cornea and the lens.
Filled with clear, watery fluid termed aqueous humor.
Posterior Chamber
Located in the large space between the lens and the retina.
Filled with a clear, gelatinous substance termed vitreous humor, which maintains intraocular pressure and the structural shape of the eyeball.
Mechanics of Visual Perception
Visual mechanics function analogously to a camera:
The retina acts as the visual recording "film."
The iris and pupil function as the dynamic aperture control.
The cornea and lens act as optical lenses focusing light rays onto the retina to form a clear visual image, which the brain subsequently decodes.
Vision Impairments and Pathologies
Refractive Errors
Myopia: Nearsightedness (difficulty seeing distant objects clearly).
Hyperopia: Farsightedness (difficulty seeing close objects clearly).
Astigmatism: Visual distortion resulting from irregular curvature of the cornea or lens.
Age-Related Ocular Changes
Decrease in the operational photoreceptor cell count within the retina.
The crystalline lens becomes gradually opaque and cloudy.
The iris becomes rigid, impairing rapid pupil adjustment to altered lighting environments.
The crystalline lens loses elasticity, impairing near-distance focusing—a condition known as presbyopia.
Lacrimal fluid production decreases, causing ocular dryness and chronic surface irritation.
Pathological Eye Conditions
Conjunctivitis ("Pink Eye")
Infection or acute inflammation of the conjunctival tissue, causing the underlying sclera to appear bright red.
Chief Complaints: Itching, burning, excessive tearing, and sticky white or yellow mucopurulent discharge.
Transmission: Highly contagious; easily spread by rubbing the infected eye and touching clean surfaces or the uninfected eye.
Treatment: Ophthalmic antibiotic drops or ointments.
Cataracts
Progressive yellowing, hardening, and opacification of the lens, preventing light transmission to the retina.
Leads to cloudy vision and eventual blindness if left untreated; highly prevalent in geriatric populations.
Treatment: Surgical extraction of the clouded lens and insertion of an intraocular lens implant.
Glaucoma
Pathological elevation of fluid pressure within the anterior chamber of the eye.
Occurs when aqueous humor fails to drain or be absorbed properly.
Progressive intraocular pressure crushes the retina and optic nerve, leading to irreversible destruction and blindness.
Risk Factors: Age over 40 years, family history of glaucoma, and individuals with dark irises.
Treatment: Early detection screenings, daily hypotensive ophthalmic drops, and surgical procedures.
Macular Degeneration
Pathology characterized by the build-up of proteinaceous deposits (drusen) or abnormal blood vessel growth over the macula (the central area of the retina).
Destroys photoreceptors, resulting in loss of central vision fields.
Leading cause of legal blindness in adults over 65 years of age.
Risk Factors: Tobacco smoking, excessive ultraviolet (UV) sunlight exposure, high-cholesterol diets, and genetic predisposition.
Ophthalmic Prosthetics and Assistive Device Care
Understanding Blindness
Legal blindness spans a spectrum: some individuals experience absolute darkness, while others retain partial vision (detecting light, movement, broad shapes, or colors without sufficient acuity to identify distinct objects).
Individuals with congenital blindness typically adapt effectively and maintain functional independence.
Individuals acquiring blindness later in life face significant adjustment and psychological challenges.
Eyeglass Maintenance Protocols
Cleaning: Clean exclusively with designated microfiber cloths and specialized cleaning solutions designed specifically for optical lenses to prevent stripping anti-reflective or protective coatings.
Drying: Dry lenses using soft, clean cloths.
Contraindications: Never use paper towels, tissues, or napkins to dry glasses, as their abrasive wood fibers cause permanent lens scratches.
Storage: Keep glasses protected inside a hard-shell case stored within easy physical reach of the client.
Contact Lens Care
Clean and store contact lenses exclusively using dedicated sterile contact lens solutions.
Always place individual lenses into their designated left ("L") and right ("R") case compartments to avoid cross-contamination or power mix-ups.
Prosthetic Eye Care
Prosthetic eyes are constructed of durable ceramic or specialized plastic materials.
Formed as a thin, curved shell disc (not a solid sphere) engineered to fit directly beneath the eyelids over an ocular implant.
Assist clients with routine removal, cleaning, and storage when required.
Prosthetics are extremely expensive; handle with extreme caution.
Risk: Improper handling causing physical scratches or nicks will severely injure or irritate the delicate mucosal tissue lining the interior eyelids.
Anatomy and Physiology of Hearing and Balance
The Outer Ear
Pinna (Auricle): External cartilaginous structure that collects environmental sound waves and funnels them into the auditory canal.
External Auditory Canal: Passageway lined with fine hairs and ceruminous glands that secrete cerumen (earwax) to trap foreign dirt and airborne particles.
Tympanic Membrane (Eardrum): Thin, membrane barrier separating the outer ear from the middle ear cavity; vibrates in direct response to arriving sound waves.
The Middle Ear
An air-filled space housing the auditory ossicles (three tiny bones):
Malleus (Hammer)
Incus (Anvil)
Stapes (Stirrup)
The ossicular chain creates a physical mechanical bridge between the vibrating eardrum and the inner ear.
Eustachian Tube: Channel connecting the middle ear cavity directly to the pharynx (throat).
Equalizes atmospheric pressure on both sides of the tympanic membrane to prevent membrane rupture.
The Inner Ear
Contains sensory receptor structures dedicated to hearing and spatial balance.
Structures for Hearing:
Cochlea: Fluid-filled bony organ shaped like a snail shell.
Structures for Balance (Equilibrium):
Vestibular Apparatus: Composed of the vestibule and three semicircular canals.
Physiology of Auditory Processing
Sound waves are collected by the pinna and funneled down the external auditory canal to strike the tympanic membrane.
Tympanic membrane vibrations cause physical movement of the three auditory ossicles (malleus, incus, stapes).
The stapes transfers mechanical vibrations into fluid displacement waves inside the cochlea.
Fluid movement stimulates internal cochlear hair cell micro-receptors.
Receptors generate electrical nerve impulses that travel along the cochlear nerve directly to the auditory cortex of the brain for decoding into recognized sound.
Physiology of Balance and Equilibrium
Changes in body or head position move internal fluid, activating specialized mechanoreceptors within the vestibular apparatus (vestibule and semicircular canals).
Receptors transmit positional nerve signals along the vestibular nerve to the brain, detailing spatial orientation relative to gravity to maintain physical equilibrium.
Pathophysiology of Motion Sickness:
Occurs when sensory signals transmitted by the eyes conflict directly with positional signals transmitted by the vestibular inner ear apparatus.
Sensory mismatch confuses the central nervous system, triggering systemic symptoms of nausea, severe dizziness, and vomiting.
Otic Pathologies and Auditory Disorders
Presbycusis
Age-related progressive hearing loss caused by stiffening of the tympanic membrane and ossicles, coupled with a decline in inner ear sensory hair cells.
Results in a selective inability to perceive high-pitched acoustic frequencies.
Causes specific difficulty distinguishing high-frequency consonant sounds like "th" and "s".
Significantly impairs the ability to follow spoken conversations when ambient background noise is present.
Cerumen (Earwax) Impaction
Excessive accumulation and hardening of earwax within the external auditory canal.
Manifestations: Conductive hearing loss, sensation of fullness in the ear, persistent ringing (tinnitus), localized itching, and otalgia (ear pain).
Risk Factor: Insertion of foreign objects into the canal (such as hearing aids) significantly elevates impaction risks.
Treatment: Professional canal flushing/irrigation and administration of prescribed cerumenolytic ear drops performed by nursing staff.
Ear Infections
Otitis Media
Infection of the air-filled middle ear space.
Symptoms: Acute ear pain, fever (pyrexia), and diminished hearing capability.
Treatment: Pharmacological oral antibiotics or surgical insertion of tympanostomy tubes.
Otitis Externa ("Swimmer’s Ear")
Infection or inflammation of the cutaneous lining of the external auditory canal.
Symptoms: Canal redness and extreme localized pain when the outer ear is touched or manipulated.
Treatment: Topical antibiotic ear drops.
Ménière’s Disease
Chronic, progressive inner ear disorder characterized by excessive accumulation of endolymph fluid.
Occurs in sudden periodic episodes or paroxysmal attacks featuring a classic symptom tetrad:
Severe vertigo (dizziness)
Tinnitus (ringing in ears)
Temporary sensorineural hearing loss
Intense feeling of ear fullness or pressure
Systemic attack manifestations include severe motion nausea and violent vomiting.
Immediate Clinical Intervention: Instruct the patient to immediately lie completely flat (supine) and maintain visual focus on a single non-moving, stationary object.
Auditory Assistive Devices and Communication Strategies
Communication Guidelines for Hearing Impairments
Position yourself directly in front of the individual, facing them fully when speaking.
Avoid chewing gum, covering your mouth, or speaking at an unusually fast rate.
Utilize written communication notes or visual pads when necessary.
Actively confirm that the individual has accurately understood the message being communicated.
Hearing Aid Maintenance and Troubleshooting
Operational Checks:
Verify that the hearing aid unit is switched to the ON position.
Verify that the physical volume control wheel is set sufficiently high.
Troubleshooting Non-Functional Units:
Check battery charge levels and replace depleted batteries.
Inspect the opening of the ear mold and external ear canal for blockage secondary to impacted cerumen.