12967_2024_Article_5401
Abstract
Background:
IDH1-wildtype glioblastoma multiforme (IDHwt-GBM) is characterized by heterogeneity and poor prognosis.
The aim was to develop multiomics molecular subtypes for improving diagnosis and treatment.
Methods:
Analyzed data from 184 IDHwt-GBM patients using consensus clustering and machine learning algorithms combining mRNA and MRI data.
Validated models across five datasets and created an online interactive system.
Assessed clinical impact and molecular associations of identified subtypes.
Results:
Two molecular subtypes (class 1 & class 2) were identified with distinct survival rates (HR=1.68 for class 1).
Class 2 showed better treatment sensitivity to radiotherapy and temozolomide.
Notable differences in mutation patterns, pathways related to PCD, and the immune microenvironment between classes.
Conclusion:
Multiomics clustering effectively stratified IDHwt-GBM patients, providing insight for personalized treatment approaches.
Introduction
Glioblastoma Overview:
Most lethal primary brain tumor; 15-month median survival; only 5% survive beyond 5 years.
Aggressive nature complicates treatment due to genetic diversity and the blood-brain barrier.
Historical Context:
Classification of glioblastoma evolved from histology-based to molecular characteristics post-2021 WHO classification.
Introduces molecular GBM (molGBM) based on molecular abnormalities compared to histologically classified GBM (histGBM).
Research Gap:
Previous studies had inconsistent survival findings between histGBM and molGBM.
This study aims to analyze multiomics data for a thorough understanding of IDHwt-GBM classifications using ten integration strategies.
Materials and Methods
Data Collection:
IDH1wt-GBM data sourced from TCGA, CGGA, GLASS, CPTAC, UPenn, and UCSF-PDGM datasets.
Methods of Analysis:
Used consensus clustering to apply advanced algorithms to multiomics data.
Employed partition around medoids (PAM) for subtype prediction using external validation cohorts.
Bioinformatic analyses measured differences in gene expression, somatic mutations, and therapeutic responses.
Results
Patient Demographics
Discovery set:
184 IDH1wt-GBM patients (62% male, median age: 60 years).
Supporting cohorts had similar demographics with minor age discrepancies.
Molecular Subtype Discovery
Identification of Subtypes:
Two subtypes classified (class 1 & class 2) revealed significant differences in overall survival rates.
Class 1 showed better prognosis; class 2 more responsive to treatments.
Mutation and Pathway Analysis
Somatic Mutations:
Class 2 patients often had TP53 mutations and increased amplification at 4q12, correlating with worse survival.
Mutational Signatures:
Detected various mutational signatures linked to treatment responses, particularly relating to temozolomide efficiency.
Pathways:
Enrichment in specific pathways like PD-1 checkpoint and ferroptosis highlighted distinctions between subtypes.
Clinical Implications
Predictive Modeling:
Established survival prediction models based on molecular subtypes integrating age and sex, allowing tailored treatment strategies.
Online platform developed for real-time predictions of IDHwt-GBM subtypes.
Discussion
Clinical Relevance:
Findings contribute to the understanding of GBM tumors in the context of molecular subtyping, influencing future treatment approaches.
Emphasizes the need for continuing observation of treatment responses and survival rates in different GBM subtypes.
Limitations:
Acknowledged the retrospective nature, variation in detection equipment, and sample size limitations across studies.
Conclusion
Successfully identified two distinct molecular subtypes of IDHwt-GBM using a comprehensive multiomics approach that directs more personalized treatment options.
Online tools for real-time patient profiling could greatly enhance clinical decision-making efficiency.