Chapter 1 & 2: Sense Of Pain and Located Sharp Pain
Chapter 1: Sense Of Pain
Section Overview
- Objectives of this section include:
- Describing the sense of pain and pain disorders.
- Describing kinesthesis and disorders of kinesthesis.
- Describing the vestibular sense and disorders of the vestibular system.
The Sense of Pain
- Pain is defined as the body's natural defense mechanism:
- Alerts the body to danger.
- Signals the presence of injury.
- Alerts the body to disease.
Perception of Pain
- The experience of pain is influenced by a person's perception or awareness of noxious stimuli.
- Noxious stimuli are defined as any stimuli that can cause damage or potential damage to the body and can be categorized as:
- Mechanical
- Chemical
- Thermal - Abundance of certain stimuli can also activate pain senses:
- For instance, the body can tolerate certain heat levels; exceeding this can result in pain.
Receptors for Pain
- Pain receptors consist of free nerve endings originating from the spinal cord:
- Located throughout the body, excluding the brain.
- Detect chemicals released from painful stimuli.
- Trigger neural signals in response to pain. - Nerve fibers are responsible for carrying these neural signals from pain receptors to the spinal cord:
- Different types of fibers are activated based on the type of pain experienced.
Chapter 2: Located Sharp Pain
Types of Pain Fibers
- A delta fibers:
- Associated with specifically located sharp pain. - C fibers:
- Associated with dull aches and pains.
Pathway to the Brain
- Pain signals follow a specific pathway:
- From nerve fibers, up the spinal cord to the thalamus.
- The thalamus then relays the signals to other areas of the brain for further processing.
Theories of Pain Processing
- Currently, no single theory fully explains all aspects of pain experience and modulation.
- Gate Control Theory:
- A theory that attempts to elucidate how pain sensation is modulated by the spinal cord.
- Suggests that the spinal cord contains a "gate" that can be opened or closed to regulate the pain signals sent to the brain, influencing the experience of pain directly correlating its intensity to various physical and environmental factors.