Chapter 1 & 2: Sense Of Pain and Located Sharp Pain

Chapter 1: Sense Of Pain

Section Overview

  • Objectives of this section include:
      - Describing the sense of pain and pain disorders.
      - Describing kinesthesis and disorders of kinesthesis.
      - Describing the vestibular sense and disorders of the vestibular system.

The Sense of Pain

  • Pain is defined as the body's natural defense mechanism:
      - Alerts the body to danger.
      - Signals the presence of injury.
      - Alerts the body to disease.
Perception of Pain
  • The experience of pain is influenced by a person's perception or awareness of noxious stimuli.
  • Noxious stimuli are defined as any stimuli that can cause damage or potential damage to the body and can be categorized as:
      - Mechanical
      - Chemical
      - Thermal
  • Abundance of certain stimuli can also activate pain senses:
      - For instance, the body can tolerate certain heat levels; exceeding this can result in pain.
Receptors for Pain
  • Pain receptors consist of free nerve endings originating from the spinal cord:
      - Located throughout the body, excluding the brain.
      - Detect chemicals released from painful stimuli.
      - Trigger neural signals in response to pain.
  • Nerve fibers are responsible for carrying these neural signals from pain receptors to the spinal cord:
      - Different types of fibers are activated based on the type of pain experienced.

Chapter 2: Located Sharp Pain

Types of Pain Fibers

  • A delta fibers:
      - Associated with specifically located sharp pain.
  • C fibers:
      - Associated with dull aches and pains.

Pathway to the Brain

  • Pain signals follow a specific pathway:
      - From nerve fibers, up the spinal cord to the thalamus.
      - The thalamus then relays the signals to other areas of the brain for further processing.

Theories of Pain Processing

  • Currently, no single theory fully explains all aspects of pain experience and modulation.
  • Gate Control Theory:
      - A theory that attempts to elucidate how pain sensation is modulated by the spinal cord.
      - Suggests that the spinal cord contains a "gate" that can be opened or closed to regulate the pain signals sent to the brain, influencing the experience of pain directly correlating its intensity to various physical and environmental factors.