Ulcerated lesions 1

describing clinical presentation:

  1. size

  2. distribution

  3. color

  4. type

  5. location

INFECTIOUS/INFLAMMATORY AND NEOPLASTIC

Primary Herpetic gingivostomatitis

-differential: primary herpetic gingivostomatitis, herpetiform aphthous ulcerations, oral manifestations of Crohn’s disease

-affected areas: keratinized and nonkeratinized mucosae

-Causative organism: HSV-1 or HHV-1

-treatment: supportive care, hydration, pain control, systemic therapy with acyclovir, valacyclovir

Histoplasmosis

-differential: SCC, deep fungal infection, traumatic ulcer/TUGSE, major aphthous ulceration, autoimmune process

-affected areas: anywhere

-causative organism: histoplasma capsulatum

-treatment: IV liposomal amphotericin B and oral itraconazole

Shingles (Herpes Zoster)

-differential: Herpes zoster, intraoral herpes injection in an immunocompromised patient, allergic stomatitis, autoimmune process

-affected areas: anywhere-normally on one side of the body and will stop abruptly at a midline rather than crossing over

-causative organism: reactivation of latent varicella zoster virus

-treatment: supportive care, hydration, pain control, and systemic therapy with acyclovir/valacyclovir. Shingrix-shingles vaccine for adults 50+

Recurrent intraoral herpes

-differential: secondary/recurrent intraoral herpes, traumatic ulcerations, autoimmune process

-affected areas: ONLY keratinized or attached mucosae (gingiva/hard palate) in immunocompetent individuals

-causative organism: Reactivation of latent herpes simplex virus 1

-treatment: supportive care, hydration, pain control;acyclovir/valacyclovir (start within 48hr)

Squamous Cell Carcinoma

-differential: SCC, deep fungal infection, Kaposi sarcoma, traumatic ulcer, TUGSE

-affected areas: anywhere

-causative organism: lowkey anything but norm. really bad lesions/ulcerations

-treatment: refer to oral and maxillofacial/ENT surgeon for wide surgical excision with lean margins, +/- lymph node neck dissection, with chemo/radiation therapy based on TNM staging

ULCERATED LESIONS II-auto-immune/Immune related/Allergy or Traumatic/reactive

Aphthous stomatitis

-differential: recurremt minor aphthous ulceration, traumatic ulceration. crohn’s disease. behcet syndrome, nutritional defic

-affected areas: nonkeratinized mucosa

-etiology: t-ce;; mediated immunologic reaction with involvement of tumor necrosis factor alpha

-tx: mild- no tx or otc anestetics or protective bioadhesive

severe- topical Corticosteroid gels

Herpetiform aphthous ulcerations

-differential: recurrent aphthous ulcerations-herpetiform, Herpes virus infection. R/O Crohn’s disease, Behcet syndrome, nutritional deficiences

-affected areas: nonkeratinized mucosae

-etiology: T cell mediated immunologic reaction with involvement of tumor necrosis factor alpha destructive disease

-tx: mild- no tx or otc for pain or protective bioadhesive products

severe- corticosteroid rinse

Erythema multiforme-crusty lips

-differential: erythema multiforme, infectious ulcers, allergic rxn

-affected areas: na

-etiology: immune mediated abnormal t cell response to HSV, mycoplasma, NSAIDS, antibiotics, antifungals, and antivirals

-tx: symptomatic relief- IV rehydration with topical anesthetic agents to decrease discomfort. If due to meds-discontinue. If triggered by HSV, acyclovir

Erosive lichen planus

-differential: erosive lichen planus, lichenoid drug rxn, lichenoid foreign body rxn, graft vs host disease, lupus erythematosus, vesiculobullous disorders, dysplastic leukoplakia ulceration

-affected areas: na

-etiology: immune mediated abnormal t cell response- autoimmune condition but the target antigen hasn’t been identified

-tx: erosive- topical corticosteroids AFTER BIOPSY PROVEN CONFIRMED DIAGNOSIS

diprolene gel

Mucous membrane pemphigoid/pemphigus

-differential: erosive lichen planus, mucous membrane pemphigoid, pemphigus, allergic/contact stomatitis

-affected areas: na

-etiology: immune mediated response, autoimmune tissue bound autoantibodies are directed against proteins in the basement membrane/hemidesmosomes

-tx: topical corticosteroids AFTER BIOPSY PROVEN CONFIRMED DIAGNOSIS, referral ophthalmologist

PIGMENTED LESIONS

Oral Mucosal Melanoma-neoplastic

-etiology: malignant neoplasm of melanocytic origin

-clinical features: often nodular at diagnosis. 75% occur on the hard palate or maxillary alveolus. Lesion usually begins as a brown or black macule that extends laterally with the development of a lobulated exophytic mass. cervical lymph node metastasis is often present

-diagnosis: diagnostic biopsy

Treatment: radicular surgery often with radiation therapy; poor prognosis

Kaposi Sarcoma-neoplastic

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Amalgam tattoo-reactive

-etiology: contamination of pre-existing oral mucosal wound with amalgam particles

-clinical features: blue-black macules with well defined or irregular borders. Most common gingiva, alveolar mucosa, and buccal mucosa; rarely show up on x-rays

-diagnosis: radiographs, diagnostic biopsy if radiographs negative

Treatment: none

Smoker’s Melanosis

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Oral Melanoacanthoma

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Drug-related discolorations

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Neurofibromatosis type 1- genetic

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Addison Disease (hypoadrenocorticism)

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Peutz-Jeghers Syndrome

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McCune Albright Syndrome

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Oral melanotic macule- idiopathic

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Acquired Melanocytic nevus (mole)

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Blue Nevus

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EXTRAORAL LESIONS

Basal Cell Carcinoma

-etiology: most common malignancy in humans; hair follicles and eccrine sweat glands; UVB radiation

-clinical features: 5 clinical patters- nodular (most common), superficial, micronodular, infiltrative, morpheaform

-diagnosis:

-treatment:

Seborrheic Keratosis

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