BG
Overview of Glutamate and its Role
Glutamate is identified as a stimulatory neurotransmitter.
Its action stimulates neurons within the striatum, leading to:
Increased excitatory postsynaptic potentials (EPSPs).
Increased action potentials traveling down the axons from the striatum to the globus pallidus externus.
Action of GABA in the Pathway
Neurons in the globus pallidus externus release GABA.
GABA is classified as an inhibitory neurotransmitter.
Consequences of increased action potentials:
Increased GABA release inhibits the following neuron:
Leads to hyperpolarization.
The inhibited neuron sends fewer action potentials from the globus pallidus externus to the subthalamic nucleus.
Resulting decrease in action potentials affects the following pathway.
Decreased action potentials result in:
Release of decreased amounts of GABA under the subthalamic nucleus.
GABA's inhibition is diminished, leading to disinhibition of the neuron.
Consequences of Disinhibition in the Indirect Pathway
Stimulation of subthalamic nucleus neurons occurs due to decreased GABA inhibition.
Increased stimulation results in:
Enhanced activation of neurons in globus pallidus internus.
Increased action potentials traveling down neurons from the globus pallidus internus to the thalamus.
Increased release of GABA onto thalamic nuclei from globus pallidus internus.
Increased GABA means stronger inhibition of the thalamic nuclei.
Resulting effect on the cortex:
Decreased action potentials from thalamus to motor cortex.
This ultimately reduces motor activity for specific body parts.
Modulation of Direct and Indirect Pathways by Basal Ganglia
The last function of the basal ganglia involves pathway modulation.
The nigrostriatal pathway plays a crucial role in this modulation.
Its function is described as amplifying motor activity.
Influence of Nigrostriatal Pathway on Direct and Indirect Pathways
Overview of Nigrostriatal Pathway Impact
The nigrostriatal pathway influences:
Direct pathway (amplifies motor activity).
Indirect pathway (modulates motor activity).
Influence on the Direct Pathway
The direct pathway leads to communication:
From cortex to striatum, then to globus pallidus internus, and finally to thalamus.
In this pathway:
Dopamine from substantia nigra influences striatum via D1 receptors (stimulatory).
Glutamate from the cortex enhances neuron activation in striatum, resulting in elevated action potentials toward globus pallidus internus.
Consequence of stimulation:
Increased GABA release from globus pallidus internus results in significant inhibition.
Underactive inhibition leads to increased action potentials back from thalamic nuclei to the cortex, thereby heightening motor activity.
Importance:
Damage to the direct pathway (especially involving substantia nigra) can lead to Parkinson's disease with symptoms including difficulty initiating and maintaining movement.
Influence on the Indirect Pathway
The indirect pathway connects:
From cortex to striatum, globus pallidus externus, subthalamic nucleus, and globus pallidus internus.
This pathway additionally involves D2 receptors (inhibitory) on striatum neurons:
Glutamate stimulates striatum.
Dopamine released onto D2 receptors inhibits the neurons in this pathway, modulating the thalamic nuclei’s activity.
Increased motor activity correlated to:
Suppression of unwanted motor movements via the indirect pathway.
Clinical Relevance of Basal Ganglia Disorders
Understanding the pathways contributes to understanding disorders:
Parkinson's Disease: Associated with damage to the direct pathway resulting in reduced motor activity initiation.
Huntington's disease: Damage to the indirect pathway can cause unwanted motor movements.
Wilson's disease: Effects from copper buildup leading to degeneration of the central nervous system affect movement.
Sydenham’s chorea: Resulting from antibodies attacking basal ganglia post-rheumatic fever.
Drug-induced movement disorders, including extrapyramidal symptoms (e.g., tardive dyskinesia, akathisia, dystonic reactions) often arising from first-generation antipsychotics affecting the D2 receptors.