Comprehensive notes on lymphocytes, thymus, Bursa of Fabricius, and secondary lymphoid tissues (vet physiology)

Hematopoiesis: lineages and the focus on lymphocytes

  • The bone marrow generates all white blood cells (WBCs) and red blood cells (RBCs) from pluripotent stem cells.

  • There are three main lineages of blood cells mentioned: erythroid (red blood cells and platelets), myeloid (basophils, eosinophils, etc.), and lymphoid (lymphocytes).

  • The lecture mainly focuses on lymphocytes (B cells and T cells) and briefly mentions natural killer (NK) cells as another lymphoid-associated population.

  • The immune system comes online shortly after birth and is most functional in young animals; by puberty, sites of lymphocyte proliferation begin to decrease, and in geriatric animals lymphocyte activity is limited though the basic function remains.

Key terms and concepts

  • Pluripotent stem cells: can give rise to multiple cell types, but are typically committed to a precursor destined for a specific lineage.

  • Lymphoid precursors: commit to the lymphoid lineage, which gives rise to B cells, T cells, and NK cells.

  • Innate vs adaptive immunity:

    • B and T cells are part of the adaptive (acquired) immune system and are highly specific.

    • NK cells are part of the innate lymphoid lineages and target infected or cancerous cells.

  • B cell activating factor (BAFF): a cytokine that stimulates B cell activation.

  • Intestinal microbiome: gut microbes that influence immune development and B cell activation.

  • Bursa of Fabricius: a specialized organ in birds adjacent to the cloaca where B cells are trained; its name helped define B cells.

  • Peyer’s patches: lymphoid tissue in the ileum (ileal Peyer’s patches) that is involved in intestinal immune responses and B cell development in mammals.

  • IgA, IgG, etc.: antibodies produced by B cells/ plasma cells in response to antigens (not spelled out in detail in this transcript, but relevant to B cell function).

B cells: development, training sites, and activation

  • Immature B cell development: B cells initially develop in the bone marrow and then migrate to peripheral lymphoid tissues for final maturation.

  • Two main factors controlling B cell development after leaving the bone marrow:

    • B cell activating factor (BAFF). This is a cytokine that stimulates B cell activation and maturation.

    • Intestinal microbiome: gut microbes influence B cell activation and the development of antibodies.

  • Birds (B cell training):

    • Bursa of Fabricius is the primary training site for B cells in birds, located next to the cloaca.

    • Bursaectomy (removal of the bursa at a young age) removes the ability to develop antibodies, showing the critical role of the bursa in B cell training.

    • Bursa reaches maximum size in young birds and undergoes involution (shrinking) after puberty, being replaced largely by fatty tissue.

  • Mammals (B cell training):

    • In mammals, B cells are trained primarily in primary lymphoid tissues (bone marrow and ileal Peyer’s patches) and then in secondary lymphoid tissues (spleen and lymph nodes).

    • Most B cell training in higher vertebrates happens in secondary lymphoid tissues (spleen and lymph nodes), providing efficient central hubs for antigen encounter.

  • Ileal Peyer’s patches (IPPs):

    • In some mammals (e.g., sheep, lambs), IPPs in the ileum are important for antibody production.

    • Removal of IPPs in young lambs reduces antibody production on challenge, but does not completely abolish it, illustrating redundancy and compensatory mechanisms.

  • Secondary lymphoid tissues for B cells: spleen and lymph nodes are major sites for B cell activation and antibody production in many mammals.

  • Birds vs mammals: in birds, B cells are trained in the Bursa of Fabricius; in mammals, B cells are trained largely in primary lymphoid tissue (bone marrow) with additional training in IPPs and other secondary tissues.

T cells: development, training, and selection

  • T cell development site: thymus, which is located in the upper thorax (near the sternum) across mammals and horses.

  • Thymus entry point for developing T cells: subcapsular region, where precursors enter the organ via blood-tissue interfaces.

  • Naive T cells: after initial development, T cells become naive and have not yet been tested for their specific antigen.

  • Migration within thymus: naive T cells migrate from the outer cortex toward the inner medulla during maturation.

  • Selection processes (to ensure self-tolerance and antigen specificity):

    • Positive selection: T cells must be able to recognize self-MHC molecules presenting antigens; otherwise they are eliminated.

    • Negative selection: T cells that react strongly to self-antigens are eliminated to prevent autoimmunity.

  • Outcome: functional naive T cells that are non-self reactive are released to the periphery.

  • Thymus anatomy and aging:

    • The thymus is encapsulated, with cortex (outer) and medulla (inner).

    • Hassall’s corpuscles (often called carpuscles in some texts) in the medulla release growth factors that promote ongoing T cell development.

    • The thymus is largest right after birth and undergoes age-related involution during puberty, progressively shrinking and often becoming difficult to detect in older animals.

  • Athymic (thymus-free) animals lack T cell–mediated (cell-mediated) immunity, illustrating the essential role of the thymus in T cell development.

Thymus histology and function

  • Encapsulated lymphoid tissue with lobulated structure; cortex to medulla flow is typical.

  • Immature T cells proliferate in the cortex and are tested with macrophages and dendritic cells throughout thymic tissue.

  • Antigen presentation within the thymus is supported by antigen-presenting cells (APCs): macrophages and dendritic cells process antigens and present them to developing T cells to drive selection.

  • Dendritic cells and macrophages are distributed throughout the thymus to present antigens and help educate T cells.

  • The thymus contains reticular fibroblasts that maintain the supportive stroma and contribute to the “sponge-like” architecture that permits cell movement and antigen sampling.

Lymphoid tissues: encapsulated vs unencapsulated

  • Encapsulated lymphoid tissues:

    • Lymph nodes and spleen are classic encapsulated secondary lymphoid organs with a capsule and trabeculae that organize tissue into functional zones.

    • The cortex/medulla in thymus is a special case of encapsulated lymphoid tissue; in lymph nodes, the analogous regions are the B cell follicles (often in cortex) and T cell zones (paracortex).

  • Unencapsulated lymphoid tissues (MALT):

    • MALT = mucosa-associated lymphoid tissue; includes tissues that line mucosal surfaces and are exposed to external environments.

    • Examples include tonsils (in horses these are often uncapsulated), Peyer’s patches in the small intestine, and other lymphoid tissues lining mucosal surfaces.

    • MALT serves as frontline immune tissue in areas exposed to external environment and pathogens.

  • Lymphoid tissue distribution in horses and other large mammals:

    • Tonsils in horses are uncapsulated in some regions.

    • Lymph nodes are widely distributed; many lymphoid centers exist in cervical, abdominal, and other regions.

    • The spleen is a major secondary lymphoid tissue with splenic cords and sinuses; the architecture supports filtering blood and mounting immune responses.

Lymph flow, lymph nodes, and antigen sampling

  • Lymphatic flow basics:

    • Lymph moves through lymphatic vessels via afferent ducts (toward lymph nodes) and efferent ducts (away from lymph nodes).

    • Lymphatic fluid enters the node via afferent lymphatics and percolates through subcapsular sinuses and cortical/medullary regions, allowing lymphocytes to sample antigens.

    • Lymph nodes have one-way flow with valves to prevent backflow, ensuring slow percolation for immune sampling.

  • Subcapsular sinus: entry region where initial antigen sampling occurs.

  • Chemotaxis: chemokine-guided migration helps B and T cells localize to their respective zones within lymph nodes.

  • Follicles and immune activation: B cell zones (follicles) and T cell zones (paracortex) separate, but antigens activate both populations to drive clonal expansion.

  • Plasma cells and antibody production:

    • B cells differentiate into plasma cells, which produce antibodies within the cords of lymph nodes.

    • Antibodies help other immune cells recognize and neutralize antigens and can be targeted by T cells for clearance.

Antigen, epitopes, and MHC interactions

  • Antigen: any substance that can provoke an immune response.

  • Epitope: the specific part of the antigen that interacts with B cell receptors or T cell receptors (via MHC presentation).

  • Antigen presentation and APCs: antigen-presenting cells process antigens and present peptide fragments via MHC molecules to T cells; B cells can recognize intact antigens via B cell receptors.

  • T cell recognition specifics:

    • T cells typically recognize peptide fragments presented by MHC molecules on APCs.

    • B cells recognize native, unprocessed antigens on their surface or soluble antigens.

Spleen and systemic immunity

  • Spleen as a secondary lymphoid organ: filters blood rather than lymph.

  • Primary function in humans and many animals: immune surveillance and clearance of blood-borne pathogens.

  • In horses, the spleen has a specialized role as a reservoir for blood to support prolonged aerobic activity; it can release stored blood to sustain high activity and oxygen delivery to muscles during extended exertion.

  • Lymph flow in the spleen parallels other secondary lymphoid tissues but involves blood-borne antigens rather than lymphatic antigens.

Vaccination and immune response concepts

  • Vaccination types:

    • Live (attenuated) vaccines: may elicit strong, long-lasting immunity with fewer boosters because they mimic natural infection (involves active lytic phase in some cases).

    • Dead (inactivated) vaccines: often require boosters to maintain immunity because they do not replicate.

  • Rhinovirus example:

    • Rhinoviruses are classic pathogens studied for respiratory infections; immunity developed after exposure or vaccination can wane, necessitating boosters for some vaccines.

  • Lytic phase and viral vaccines: the lytic phase is a viral replication step that triggers host cell responses; vaccines can be designed to remove or inactivate this phase to reduce disease while still provoking an immune response.

Practical and ethical considerations in immune function research

  • Animal models: athymic (thymus-less) animals lack T cell–mediated immunity, illustrating the essential role of the thymus in adaptive immunity.

  • Immunosuppression and transplantation:

    • Cyclosporine is an immunosuppressive drug used to reduce graft rejection; it’s effective but expensive and has limitations.

    • Transplantation raises ethical and practical concerns about immune compatibility and long-term management.

  • Critical thinking about immune development:

    • Even when a primary organ (e.g., IP patches or bursa) is removed or damaged, other tissues often compensate, leading to partial rather than complete loss of function.

    • This highlights redundancy and plasticity in the immune system and has real-world implications for breeding, management, and disease resistance in animals.

  • Age-related decline in immune function:

    • In aging animals, lymphoid organs undergo involution and reduce output and activity, contributing to decreased immune responsiveness.

Summary of key relationships and concepts

  • Lymphocytes (B and T cells) are central to adaptive immunity; NK cells participate in innate lymphoid responses.

  • B cells mature and are activated in bone marrow and peripheral/secondary lymphoid tissues; birds rely on Bursa of Fabricius for B cell training, whereas mammals rely more on bone marrow and other primary/secondary tissues (e.g., IP patches, spleen, lymph nodes).

  • T cells mature in the thymus and undergo rigorous selection (positive and negative) to ensure self-tolerance and appropriate antigen specificity.

  • Lymphoid tissue organization (encapsulated vs unencapsulated) and the architecture of lymph nodes and spleen enable efficient sampling and mounting of immune responses.

  • Antigen interactions involve epitopes, MHC presentation, and APCs; B and T cells recognize antigens through different yet complementary mechanisms.

  • Vaccination strategies exploit differences between live and dead vaccines and leverage the immune system’s capacity to generate memory responses, with boosters often required for non-replicating vaccines.

  • The spleen’s role as a blood filter and reservoir supports systemic immunity and, in some species like horses, contributes to rapid physiological responses during exercise.

  • Ethical considerations in immune research include the use of animal models, transplantation challenges, and balancing immune competence with welfare.

22 main types of peripheral lymphoid tissue: encapsulated and unencapsulated (MALT)
33 primary lymphoid tissues involved in lymphocyte development (bone marrow for B cells, thymus for T cells)

  • Key terms to review: BAFF, Bursa of Fabricius, Peyer’s patches, Hassall’s corpuscles, subscapular region, germinal centers, afferent/efferent lymphatics, sinuses, plasma cells, and APCs (macrophages and dendritic cells).