Lecture 08_Hai_2025
Lecture 8: Yersinia Pestis and Plague
Overview of Yersinia Pestis
Type: Gram-negative coccobacillus.
Etiology: Known for causing plague, famously recognized for leading to the Black Death.
Evolution: Evolved from Yersinia pseudotuberculosis in the last 1500-2000 years.
Hosts: Affects humans, rodents, and arthropods (fleas).
Historical Discovery
Identified by: Alexandre Yersin and Kitasato Shibasaburo (1894).
Context: Emerged from an outbreak of pneumonic plague in China.
Forms of Plague in Humans
Bubonic Plague:
Symptoms: Hemorrhaging, inflamed lymph nodes (buboes).
Transmission: Bites from infected fleas.
Septicemic Plague:
Symptoms: Disseminated infection leading to skin hemorrhages.
Mortality: ~100% untreated.
Pneumonic Plague:
Symptoms: Lung infection, highly infectious.
Transmission: Primarily through aerosols; can spread from other infected tissues.
Severity of Plague
Bubonic Plague: Untreated fatality rate of 50-60%.
Septicemic Plague: Nearly 100% fatal without treatment.
Pneumonic Plague: Almost 100% fatal if untreated; very contagious.
Historical Pandemics
1st Pandemic (542 AD): >25 million deaths in initial outbreak.
2nd Pandemic (14th Century): Known as the Black Death; 200 million deaths across Europe.
3rd Pandemic (ended 1990s): 20 million deaths in India alone.
Plague Today
Last US Epidemic: 1924-25 in Los Angeles.
Current Status: Regarded as a re-emerging infectious disease.
Public Health: Modern medicine mitigates risks but requires vigilance.
Bioterrorism Concerns: Historical attempts to weaponize plague during sieges.
Treatment Options
Antibiotics: Must be administered promptly due to rapid disease progression.
Vaccine: Currently unavailable in the US.
Drug Resistance: Emergence of multiple-drug resistant strains (e.g., in Madagascar).
Animal Vectors and Transmission Cycles
Zoonotic Pathogen: Spread from rodents to humans via infected fleas.
Reservoirs:
Sylvatic Cycle: Includes wild rodents and their fleas.
Urban Cycle: Domestic black rat and oriental rat flea.
Transmission Pathways: Direct contact, flea bites, and secondary spread via pneumonic plague.
Flea Vector Dynamics
Primary Vector: Xenopsylla cheopis (oriental rat flea).
Mechanism: Y. pestis forms biofilms in flea gut, causing starvation and leading to regurgitation of bacteria during feeding.
Virulence Factors
Plasmids: Multiple virulence plasmids (pPCP1, pPMT1/pFra, pCD1) involved in pathogenicity.
Type III Secretion System (T3SS): Mediates host immune evasion and bacterial adhesion.
Key Effectors: Yop proteins disrupt host cell functions and can induce apoptosis in immune cells.