Complex diseases
Complex Disorders
Adult Onset: Diabetes Mellitus, Epilepsy, Glaucoma, Hypertension, Ischemic heart disease, Manic depression, Schizophrenia.
Congenital: Cleft lip/palate, Congenital dislocation of the hip, Congenital heart defects, Pyloric stenosis, Talipes.
Genetic and Environmental Contributions
Genetic Factors: Simple, unifactorial, high recurrence rate (e.g., Duchenne muscular dystrophy, Hemophilia).
Environmental Factors: Influences multifactorial diseases, which are common, complex, and have low recurrence rates (e.g., Peptic ulcer, Diabetes, Tuberculosis).
Most diseases with a genetic component display multifactorial inheritance rather than Mendelian inheritance.
Polygenic Inheritance
Definition: Involvement of many genes where each contributes a small amount to the final phenotype. ALso environment is a factor
Examples: Skin color.
Model: Multiple alleles at multiple loci contribute to a single trait.
Some diseases are defined clinically in terms of quantitative traits meaning it can be measured, example- hypertension
Other multifactorial disorders differ qualitatively from normal states e.g. cleft palate which means that normally there would not be a gap between the nose and lip, however, in cleft palate there is.
Multifactorial Inheritance
Definition: Controlled by many genes with small additive effects (polygenic) along with environmental factors.
Phenotype Determination: Dependent on genetic, environmental, and developmental factors, including protective and susceptibility factors.
Genetic Predisposition: Inherited from both parents, but the exact genes and environmental factors vary among individuals.
Model Characteristics:
Several loci are involved without dominance or recessively.
Additive effects of loci.
Interaction of genotype and environment to produce phenotype.
Normal Variation: Most phenotypic variations in normal individuals (e.g., skin colour, height, intelligence) are due to multifactorial traits.
If an individual liability exceeds a certain threshold value, he or she will have the disease.
Tools in the Study of Complex Diseases
Twin Studies: Assess genetic effects on variation. Monozygotic (identical) twins share 100% genes, and dizygotic (fraternal) twins share 50% genes.
Concordance Rates: High concordance in monozygotic twins for genetic traits indicates a stronger genetic influence.
Utility: Distinguish between genetic, non-genetic, and combined effects. Limitations include small sample sizes and biases.
Adoption Studies: Compare adopted children with their biological and adoptive parents.
Advantages: Isolate genetic factors from environmental influences.
Limitations: Selective placement and non-representative samples.
Migrant Studies: Compare disease patterns in immigrants with citizens and kin in the homeland.
Purpose: Assess the roles of genetic, environmental, and lifestyle factors in disease occurrence.
Linkage Studies: Used for identifying genes or genetic variants responsible for diseases by studying large families with affected individuals across generations.
Advantages: Localize disease risk areas across the genome and study multiple genetic markers.
Limitations: Require large numbers of affected families, and less helpful for complex traits.
Association Studies: Compare the DNA of people with a disease (cases) and without (controls) to find associated genetic variants.
Genome-Wide Association Studies (GWAS): Investigate the entire genome for disease-associated SNPs.
Factors Influencing Recurrence Risk
Higher Risk: More affected relatives, severe cases, and relatives of the less commonly affected sex.
Determination: Through family studies and empiric risk observation.
Multifactorial Threshold Model
Concept: Disease manifests only when a certain threshold of genetic and environmental risk factors is exceeded.
Examples: Spina bifida, and pyloric stenosis.
Family Studies
Proband: The affected individual from whom the study starts.
Recurrence Risk Calculation: Based on the proportion of affected siblings.