Recording-2025-09-03T14:55:17.337Z

Neuron: Basic Structure and Function

  • The brain contains around N8.5×1010N \approx 8.5 \times 10^{10} neurons (estimates vary). Neuron = a nerve cell and the primary functional unit of the nervous system.

  • Dendrites: tree-like branches on the left side of the neuron.

    • Dendrites are where neurons receive information from other neurons.

    • Receptors on dendrites pick up signals from neurotransmitters released by other neurons.

    • These signals cause electrical changes that are interpreted in the soma (cell body).

  • Soma (cell body): contains the nucleus with DNA; site of integration of information from dendrites.

    • Soma performs metabolic functions and houses the cell’s machinery.

    • The soma is the center of the neuron’s metabolism and contains mitochondria; glial cells support the soma.

  • Axon hillock: the integration site where inputs from dendrites are summed; acts as a gatekeeper.

    • If the signal is strong enough (surpasses a threshold), the neuron generates an action potential and sends it down the axon.

    • If not strong enough, the signal stops at the hillock.

  • Axon: conducts the electrical signal (action potential) away from the soma toward axon terminals.

    • Axon is insulated by myelin, which prevents signal degradation and speeds transmission.

    • The axon ends in axon terminals (synaptic buttons).

  • Myelin: an insulating white fatty layer around the axon that speeds conduction and protects the signal.

    • Myelin is formed by glial cells: Schwann cells in the peripheral nervous system (PNS) and oligodendrocytes in the central nervous system (CNS).

    • A single Schwann cell wraps around one axon segment to form one internode; one oligodendrocyte can form many internodes on multiple axons.

    • Myelinated axons appear white and constitute the brain’s white matter; unmyelinated axons contribute to gray matter.

    • Myelination increases conduction speed; disruptions in myelin (e.g., multiple sclerosis) impair neural communication and can cause physical/psychiatric symptoms.

  • Nodes of Ranvier and internodes:

    • Gaps in myelin are nodes of Ranvier; sections adjacent to nodes are internodes.

    • Nodes are rich in sodium channels; they help refresh the action potential as it travels along the axon.

    • The action potential appears to jump from node to node (saltatory conduction), increasing speed.

  • Axon terminals and synapses:

    • The axon terminals release neurotransmitters into the synaptic cleft when an action potential arrives.

    • The synaptic cleft is the gap to the next neuron’s dendrites; neurons do not touch.

    • Neurotransmitters bind to receptors on the postsynaptic membrane of the next neuron to propagate or modulate the signal.

  • Purkinje cell example (cerebellum):

    • Purkinje cells have highly arborized dendritic trees; the cerebellum’s wiring supports balance and reflexes.

    • The presence of a soma, dendrites, axon, and a complex dendritic arbor is typical of cerebellar circuitry.

  • Electrical and chemical signaling:

    • Neurons rely on electrical signals (action potentials) and chemical signaling (neurotransmitters) to communicate.

    • The two primary modalities coordinate to pass information rapidly and precisely.

  • The soma and cortex:

    • The soma sits in the cortex (gray matter) while the axon travels through white matter.

    • The cortex is about 40 Post-it notes thick in common analogies; it contains somas and dendrites involved in high-level processing.

  • Brief cat cerebellum note:

    • The cerebellum’s structure (including arborized dendrites) is associated with balance and reflexes; this is used to illustrate neural wiring in the brain.

  • Two-minute neuroscience snapshot on myelin:

    • Myelin is an insulating layer around axons; it is lipid-rich and prevents electrical current from leaking.

    • More myelin → faster and more efficient conduction of action potentials.

    • Myelin is not continuous; there are gaps (nodes of Ranvier) that help maintain signal strength.

  • Axon hillock and signal strength:

    • The axon hillock determines whether the incoming signal reaches threshold to trigger an action potential.

    • Signals that do not reach threshold are suppressed and do not propagate.

  • The synapse (revisited):

    • Neurotransmitters are released from presynaptic terminals, traverse the synaptic cleft, and bind to receptors on the postsynaptic neuron.

    • Neurotransmitters can be excitatory (increase likelihood of the postsynaptic action potential) or inhibitory (decrease likelihood).

    • Clear, precise communication depends on receptor binding, transmitter clearance, and receptor response.

  • Vesicles and neurotransmitters:

    • Neurotransmitters are packaged in vesicles in the presynaptic neuron.

    • When an action potential arrives, vesicles fuse with the presynaptic membrane and release their contents into the synaptic cleft.

    • Vesicles may contain thousands of neurotransmitter molecules.

  • Clearance from the synaptic cleft: