Hemorrhagic Disorders Summary
Hemorrhagic Disorders
- Hemorrhagic disorders result from defects in vessel walls, platelets, or coagulation factors.
- Abnormal bleeding presentations vary:
- Massive bleeds: Result from ruptures of large vessels (e.g., aorta, heart).
- Often fatal due to overwhelming hemostatic mechanisms.
- Examples: aortic dissection, abdominal aortic aneurysm (Chapter 11), myocardial infarction complicated by rupture (Chapter 12).
- Subtle defects: Evident under hemostatic stress (surgery, childbirth, dental procedures, menstruation, trauma).
- Common causes: inherited vWF defects (Chapter 14), aspirin consumption, uremia (renal failure).
- Uremia alters platelet function through unknown mechanisms.
- Coagulation factor deficiencies (hemophilias, Chapter 14):
- Usually inherited, lead to severe bleeding if untreated.
- General principles related to abnormal bleeding:
- Defects of primary hemostasis (platelet defects, von Willebrand disease):
- Present with small bleeds in skin or mucosal membranes.
- Petechiae: minute 1- to 2-mm hemorrhages.
- Purpura: slightly larger (≥3 mm) than petechiae.
- Capillaries of mucosa and skin are prone to rupture following minor trauma.
- Platelets normally seal these defects immediately.
- Mucosal bleeding: epistaxis (nosebleeds), gastrointestinal bleeding, menorrhagia (excessive menstruation).
- Thrombocytopenia (very low platelet counts) can lead to fatal intracerebral hemorrhage.
- Defects of secondary hemostasis (coagulation factor defects):
- Present with bleeds into soft tissues (muscle) or joints.
- Hemarthrosis (bleeding into joints) is characteristic of hemophilia (Chapter 14) following minor trauma.
- Severe platelet defects may also result in intracranial hemorrhage, sometimes fatal.
- Generalized small vessel defects:
- "Palpable purpura" and ecchymoses (bruises).
- Ecchymoses: hemorrhages of 1 to 2 cm.
- Hematoma: palpable mass of blood due to large extravasated blood volume in purpura and ecchymoses.
- Characteristic of systemic disorders disrupting small blood vessels (e.g., vasculitis, Chapter 11) or causing blood vessel fragility (e.g., amyloidosis, Chapter 6; scurvy, Chapter 9).
- Clinical significance of hemorrhage depends on:
- Volume of the bleed.
- Rate at which it occurs.
- Location.
- Rapid loss of up to 20% of blood volume may have little impact in healthy adults; greater losses can cause hemorrhagic (hypovolemic) shock.
- Trivial subcutaneous bleeding can be fatal if located in the brain; intracranial hemorrhage may increase intracranial pressure and cause brain stem herniation (Chapter 28).
- Chronic or recurrent external blood loss (e.g., peptic ulcer, menstrual bleeding) causes iron loss, leading to iron deficiency anemia.
- When red cells are retained (e.g., hemorrhage into body cavities/tissues), iron is recovered and recycled for hemoglobin synthesis.