CVA
CVA and Increased Intracranial Pressure
Definition and Overview
- CVA (Cerebrovascular Accident) often results in increased Intracranial Pressure (ICP) due to fluid accumulation.
- Increased ICP leads to swelling and stress on the brain.
- This condition is considered a medical emergency that necessitates prompt intervention.
Signs and Symptoms of Increased ICP
- Decreased Level of Consciousness: Initial and crucial sign.
- Additional Symptoms:
- Restlessness
- Irritability
- Confusion
- Headache
- Nausea and Vomiting
- Changes in speech pattern
- Change in sensorimotor status
- Seizures
- Cushing's Triad:
- Severe hypertension
- Widened pulse pressure
- Bradycardia
- Posturing: Abnormal postures may be exhibited.
Other Complications Associated with Increased ICP
- Hydrocephalus (accumulation of cerebrospinal fluid)
- Vaso- and arterial spasms
- Occurrence of seizures
Blood Pressure Control
Ischemic Stroke Management
- Clients require slightly elevated blood pressure to facilitate circulation around obstructed vessels.
- Utilize a flat head of the bed to minimize cardiac workload.
- Use of Pressors:
- Medications such as Neo-Synephrine may be administered to raise blood pressure.
Hemorrhagic Stroke Management
- Clients necessitate a lowered blood pressure to mitigate further bleeding.
- Elevate the head of the bed to a position of 30-45° for adequate drainage.
- Antihypertensives:
- Nicardipine can be used to effectively reduce blood pressure.
Ongoing Therapy in Stroke Management
Post-Ischemic Stroke Therapy
- Patients are typically prescribed:
- Antithrombotic/Anticoagulant Medications:
- Aspirin
- Antiplatelet Agent: Plavix
- Warfarin: Used in cases of atrial fibrillation to maintain anticoagulation for cerebral circulation.
- Antihypertensives: Examples include ACE inhibitors and diuretics (e.g., Hydrochlorothiazide).
- Stool Softeners: Employed to prevent straining, which can increase ICP.
Post-Hemorrhagic Stroke Therapy
- Key medication includes Nimodipine, a calcium channel blocker that:
- Effectively crosses the blood-brain barrier.
- Prevents vaso- and arterial spasms.
- Stool softeners are also advised to prevent complications.
Types of Ischemic Stroke
Thrombotic Stroke:
- Occurs as a result of a stationary thrombus, typically due to atherosclerosis.
- Develops slowly, leading to a gradual reduction in oxygen supply.
Embolic Stroke:
- Resulting from a mobile clot that lodges into vessels quickly.
- Causes an abrupt interruption in cerebral perfusion, creating an immediate medical emergency.
Treatment Importance
- A crucial time-sensitive treatment window exists; "Time is brain tissue":
- The longer the delay in treatment, the greater the risk of permanent disability due to brain tissue loss.
Consequences of Stroke
- Often leads to contralateral injuries:
- Right-brain stroke results in left-side weakness or paralysis and vice versa.
- Brain Edema: Increased intracranial pressure may cause additional secondary injuries.
Tissue Plasminogen Activator (TPA) Administration Guidelines
Preparation for TPA Administration:
- Prior checks include coagulative values to assess the risk of bleeding.
- Measure the client’s blood pressure:
- If greater than 180 mmHg, administer IV antihypertensives and closely monitor blood pressure.
Administration Protocol for TPA:
- Do not perform IV push except for the initial 10% bolus given over one minute by an experienced critical care nurse.
- The remaining dosage is administered over 60 minutes.
- Transfer clients immediately to a critical care unit with staff trained in TPA management.
Monitoring During TPA Therapy:
- Conduct frequent neurological checks, ideally every 15 minutes to one hour.
- Discontinue TPA if neurological status declines, indicating a shift from ischemic to hemorrhagic stroke.
- Continue TPA if the patient's neurological condition improves.
- After the initiation of TPA, avoid any invasive lines for 24 hours.
- Refer to the textbook for comprehensive care practices for clients undergoing TPA therapy.