Principles of anesthesia
The goal of inhalation anesthesia is to maintain a partial pressure of the anesthetic agent in the alveoli that is sufficient to maintain the required partial pressure of the brain
Stages of anesthesia:
Stage 1: analgesia
Pa\\in-free
beginning of induction to loss of consciousness, voluntary movements are present
pre-med through part of the induction
stage 2: delirium
period of excitement followed by loss of consciousness, nystagmus, and sometimes resp. from induction agent
pre-med through induction
stage 3: Surgical
plane 1:
regular breathing, poor muscle relaxation, nystagmus gone, active palpebral reflex
plane 2:
medium anesthesia depth, sluggish palpebral, active corneal reflex, muscle relaxation, adequate analgesia
Plane 3:
deep surgical anesthesia, some abdominal support to breathing, uneven respiratory character, palpebral reflex gone, week corneal reflex
Plane 4:
patient is dying
The Brain and the Body
Cardiovascular
heart rate and strength vs. anesthetics
concept of MAC
uptake, distribution, and elimination
Respiratory
VQ mismatch
induction, maintenance, and recovery
from anesthetic inhalant to blood phrase
Cardiovascular need to remember!
SV
the volume of blood pumped in one contraction
HR
number of contractions in one minute
CO
volume of blood pumped in one minute
SV x HR
MAC
The alveolar concentration of anesthesia at which 50% of the anesthetized patients will not respond to a surgical stimulus
Minimum alveolar concentraion
Means some may need more and some may need less
Blood/ Gas Coefficient
the ratio of anesthetic gas in the blood phase to that in the gas phase
There is no inhalation anesthetic, when present in quantities sufficient to produce anesthesia, that will fail to reduce contractility in the mammalian heart
Minimum Alveolar Concentration
the alveolar concentration of anesthesia at which 50% of the anesthetized patients will not respond to a surgical stimulus
every patient will be different
every inhalant has a different value
if the inhalant is more soluble it acts less
isoflurane
sevoflurane
desflurane
halothane
though mostly obsolete
Blood Gas Coefficient: why every inhalant will need to be monitored and adjusted differently
blood/gas solubility = the amount of anesthetic dissolved in the blood
a high coefficient means there is a large quantity of dissolved
the anesthetic that is dissolved in the blood is not available to diffuse into the brain
Isoflurane = 1.46 sevoflurane = 0.68
more soluble will produce more anesthesia less fast
less soluble allows the body to collect more which will take longer
this is the characteristic that determines induction and recovery time
MAC and the effect of anesthesia
cardiac output and induction time are directly related
CO increases, IT increases
CO decreases, IT decreases
MAC of your inhalant doesn’t change the effect of anesthesia on your patient, but it does affect the technique of how we achieve it
Respiratory need to remember!
Tidal volume (TV)
amount of gas passed in and out of the lung in one respiratory cycle
Mechanical dead space
combined inhaled and exhaled air
physiological dead space
mouth, trachea, etc.
Atelectasis
collapsed or airless state of the lung
resistance pressure
how much pressure is pushing up against the diaphragm
Inspiration Capacity
TV + inspiratory reserve
deep breath = reserve cavity
Effects of anesthesia on ventilation
The tidal volume of the patient’s respiration decreases
focus on how well the patient is breathing, not just the numbers
IPPV= intermittent positive pressure ventilation (bagging)
Mechanical dead space is increased
hypoxic pulmonary vasoconstriction (HPV) is obtunded
Reflex of the body
Vasoconstriction: constriction of blood pressure, causing increased blood pressure
the safety net of the body that saves energy and produces more uptake of oxygen in the body
V/Q mismatching occurs
Amount of the lung field that will not uptake air as much as other
V/Q Mismatch
Under normal, awake conditions ventilation and perfusion are matched so that proper exchange occurs
atelectasis creates hypoxic alveoli
in patients of greater mass, this problem is more difficult to overcome
horses, bovine, bigger sheep or goats, obese dogs and cats
Inhalant induction and maintenance
moving from the gas phase to the blood phase takes place by simple diffusion
Drop perfume analogy
gasses diffuse down a pressure or concentration gradient
the larger the gradient the quicker the concentration rises on the lower side of the gradient
The anesthesia machine is how this is allowed to happen
intermittent dosing device
Two carrier processes of anesthesia
anesthesia machine flow meter
oxygen flow: carries anesthetic from the vaporizer to the breathing circuit
patient ventilation: carries anesthetic from the breathing circuit to the lungs
the anesthetic moves from the alveoli to the blood by diffusion
the reduced TV and respiration rate generated by the patient necessitates the need for IPPV
The Anesthesia Machine
Increase the alveolar concentration
increase respirations or gas concentration
has a little vaporizer that releases the gas
decrease alveolar concentration
decrease respirations given via IPPV or gas concentration
Affect a change in alveolar concentration
changes planes of anesthesia
Changing the gas concentration
1 TC (time constant) = volume/flow
1 TC = 5L/1L per min
1 TC = 5 min
3 TC = 15 mins
Recovery
decrease the blood-brain gradient
what has been inhaled must be exhaled
all forms of the drug must be metabolized
Every patient will be different
the patient retreats back through the stages of recovery
REMEMBER
there is no safe anesthetics, just safe anesthetists
the anesthetist always doses inhalation agents to effect
therefore attention to the patient is the most important duty of the anesthetist