10.2 The Innate Immune system
Overview
The non-specific immune system, also known as innate immunity, is the body’s first line of defence against pathogens. It provides immediate, non-specific protection against a broad range of invaders without the need for prior exposure
Anatomical barriers
Skin
The epidermis forms a physical barrier against pathogens
It consists of dead, keratinised cells that are impermeable and continuously shed to remove microbes.
Sebum secreted by sebaceous glands maintains a pH of 3-5, inhibiting microbial growth
Langerhans cells, macrophages, and intraepithelial lymphocytes are present to combat pathogens
Mucous membranes
Line all body cavities exposed to the external environment, including the respiratory, gastrointestinal, urogenital tracts and conjunctivae
Composed of:
Mucus: Produced by goblet cells, traps microbes with high viscosity
Cilia: Hair-like projections that propel mucus and trapped microbes out of the respiratory and gastrointestinal tracts.
Commensal microbes
These beneficial microorganisms outcompete pathogens for nutrients and space
They digest dietary fibres to produce substances required for a healthy colon such as metabolites, vitamins, short chain fatty acids.
They also release antimicrobial substances like lactic acid and bacteriocins
Soluble barriers
Antimicrobial peptides
Cationic proteins that disrupt microbial membranes
Produced by keratinocytes, mucosal epithelium, neutrophils, and macrophages
Example: Definsins that bind and disrupt bacterial membranes
Complement system
A cascade of proteins that enhances immune responses
Pathways:
Classical pathways: Recognises antibody-antigen complexes
Alternative pathway: Activates on microbial surfaces
Lectin pathway: Binds to sugar residue on bacterial surfaces
Functions:
Opsonisation: Coats pathogens to enhance phagocytosis
Inflammatory response: Triggers inflammation and recruits immune cells
Membrane attack complex (MAC): Punches holes in microbial membranes, leading to cell lysis
Opsonins
Soluble proteins that tag microbes, making them more recognisable for phagocytes
Example: C3b from the complement system binds to pathogens for enhanced phagocytosis
Cellular barriers
Macrophages
Professional phagocytes that engulf and digest pathogens
Reside in tissues and act as antigen-presenting cells (APCs)
Neutrophils
Detect and destroy virus-infected cells and tumor cells by inducing apoptosis
Recognise cells that lack MHC I markers, a sign of infection of abnormality
Basophils and mast cells
Involved in inflammatory responses and release histamine during allergic reactions
Phagocytosis and apoptosis mechanisms
Phagocytosis
The process of engulfing pathogens by microphages and neutrophils
Steps:
Recognition: Microbes are identified by pattern recognition receptors (PRRs) or opsonins like C3b.
Engulfment: The pathogen is internalised into a phagosome
Digestion: Phagosome fuses with lysosomes to form a phagolysosome, where enzymes break down the pathogen
Apoptosis
Controlled cell death induced by natural killer cells
Infected or damaged cells are triggered to self-destruct, preventing the spread of infection
Apoptotic bodies are formed and safely removed by phagocytes without causing inflammation