Chapter 10
Biosynthesis of Nucleic Acids - Replication
DNA metabolism
structure of DNA is fluid
new copy of DNA synthesized with high fidelity before cell division
errors are constantly checked for and repaired
segments are rearranged within a chromosome or between two DNA molecules giving offspring to a novel DNA
set of enzyme catalyzed and tightly regulated processes
DNA is the substrate that encodes its own metabolism
Flow of genetic information in the cell
Central Dogma: mechanisms by which information is transferred in the cell
Retroviruses: RNA as genetic material, RNA directs its own and DNA synthesis
Replication of DNA
naturally occurring DNA is single or double stranded, linear or circular
conservative, semiconservative, and dispersive replication possibilities
Meselson-Stahl Experiment
cells grown on 15N isotope then switched to 14N isotope and allowed to divide once
confirmed semiconservative replication
Fundamental rules:
Semiconservative (Meselson-Stahl)
begins at origin of replication and is bidirectional
circular DNA of prokaryotes: one origin, two forks
linear DNA of eukaryotes: several origins, two forks at each
catalyzed by DNA polymerase
nucleophilic attack by the 3’-OH of the growing DNA chain at the 5’-a-phosphate of the incoming dNTP
choice of dNTP (AGCT) is governed by existing template strand
DNA synthesis is template directed
all DNA polymerases require a template and a primer
primer: segment with free 3’-OH (primer terminus)
processivity: rate of synthesis divided by rate of dissociation
proceeds in 5’-3’ direction, semidiscontinuous
Challenges in replication
circular double stranded DNA
achievement of continuous unwinding and separation of two strands
protection of unwound portions from attack by nucleases
synthesis of DNA template strand from a 5’-3’ and a 3’-5’ strand
efficient protection from errors in replication
synthesis of leading and lagging strands
5’-3’ direction on both strands but replication fork moves in one direction
leading strand synthesized continuously
lagging strand synthesized semi-discontinuously
okazaki fragments
DNA ligase: joins Okazaki fragments via covalent bonds
Enzymatic synthesis and degradation of DNA
1. synthesis: DNA polymerase
E. coli has 5 DNA polymerases
function has all four deoxyribonucleoside triphosphates (ATGC), Mg++, RNA primer, DNA pol I, II, III
DNA pol I: repair and patching of DNA
DNA pol III: polymerization of newly formed DNA strand
DNA pol II, IV, V: proofreading and repair
Topoisomerase: remove and introduce supercoils
Helicase: promotes unwinding at replication fork
single stranded binding protein: stabilizes single stranded regions
primase: synthesis of RNA primers
catalyzes copying of short stretch of DNA template to produce RNA primer sequence
DNA ligase: covalently link Okazaki fragments
synthesis and linking of new DNA strands is begun by DNA pol III
new DNA is linked to 3’-OH of RNA primer
replication fork moves away, RNA primer is removed by DNA pol I
2. synthesis: reverse transcriptase
3. degradation: nucleases
exonucleases: degrade DNA from one end, direction specific
endonucleases: degrade DNA from internal sites, non-specific or specific
DNA Replication
Initiation
begins at OriC (245 bp, highly conserved)
Elongation
occurs at replication fork with leading and lagging strands
Termination
Ter sites and Tus protein
bind together and block activity of helicase
traps one replication fork at Tus-Ter complex
two replication forks can collide
causes DNA damage
DNA replication in eukaryotes
chromosome is larger, more complex, and linear
regulation is more complex - tied to cell cycle
rate of replication is slower
multiple sites for origin of replication
Error rates of DNA replication
Errors = mutations
E. coli: errors are rarely made
template directed synthesis works very well
incorrect bases do not base pair properly with template and are rejected before bond is made
rare tautomers can hydrogen bond to wrong base
3’-5’ exonuclease activity of DNA polymerase removes mismatched base and fills in the correct base
proofreading: removal of incorrect nucleotides immediately after they are added to the growing DNA
DNA repair
cells usually have one or two copies of genomic DNA
genetic information inherent in nucleotide sequence must be maintained
RNA and proteins turnover in cell quickly
mistakes in RNA and proteins are often minor
Genomic DNA does not turnover, it is replicated
mistakes in DNA are permanent = mutations
mutation: permanent change in nucleotide sequence of the genome
types of mutations
substitution mutation: replacement of one base with another
insertion or deletion mutation: addition or loss of one or more bases
silent mutation: mutation that has no effect on gene function
DNA lesions produced by chemistry of nucleotides and environmental damage
Non-enzymatic reactions of nucleobases
deamination or loss of exocyclic amino groups
hydrolysis of glycosidic bond
more common for purines
loss of purine = apurinic site
loss of pyrimidine = apyrimidinic site
depurination
light induced DNA damage