bio- lecture 9 notes
- actin accessory proteins
- thymosin- binds subunits, prevents assembly
- gelsolin- severs filaments and binds to + end
- does not require extra energy input
- tropomyosin- stabilizes filament
- cofilin- binds ADP-actin filaments, accelerates disassembly
- stabilizes actin
- capping protein- prevents assembly and disassembly at + end
- slows rates of both filament growth and deplymerization by inactivating + end
- - end bound or unbound by ARP complex
- uncapped population of filaments- growth at + and - ends
- capped population of filaments- growth at - end only
- microtubule accessory proteins
- stathmin- binds subunits, prevents assembly
- binds two tubulin heterodimers
- enhances catastrophes
- phosphorylation inhibits binding to tubulin
- keeps heterodimers bent back like they’re in GDP state
- katanin- severs microtubules
- includes two subunits
- smaller subunit hydrolyzes ATP and severs
- larger subunit directs protein to centrosome
- MAPs- stabilize tubules by binding along sides
- like a splint or back brace
- GTP cap on + end
- stabilization- frequency of catastrophes suppressed and/or growth rate enhanced
- result is longer, less dynamic microtubules
- destabilization- frequency of catastrophes increased
- result is shorter, more dynamic microtubules
- catastrophe factors bind to microtubules ends and pry protofilaments apart
- can pull off heterodimers in T state
- bundling proteins- cross-link actin filaments into a parallel array
- gel-forming proteins- hold two actin filaments together at a large angle to each other to create a looser meshwork
- contractile bundle- loose packing allows myosin-II to enter bundle
- found in stress fiber
- binds actin filaments with distance in between
- space for myosin-II
- helps cell move
- uses a-actinin
- gel-like network found in cell cortex
- parallel bundle- tight packing prevents myosin-II from entering bundle
- found in filopodium
- uses fimbrin
- filamin
- contain a bent connection between the two binding domains to form filament webs
- important to extend thin, sheet-like membrane projections- lamellipodia
- help cell crawl across surfaces
- attaches to gel-like network
- gives actin structure/meshwork
- proteins need to be activated to interact with other proteins
- actin structures can stiffen and change the shape of the plasma membrane via the ERM protein family
- actin specific drugs
- phalloidin- binds and stabilizes filaments
- from fungus
- cytochalasin- caps filament + ends
- swinholide- severs filaments
- latrunculin- binds subunits and prevents their polymerization
- extracted from sea sponge
- microtubule specific drugs
- taxol- binds and stabilizes microtubules
- from bark of tree
- colchicine, colcemid- binds subunits and prevents their polymerization
- from flower
- vinblastine, vincristine- binds subunits and prevents their polymerization
- nocodazole- binds subunits and prevents their polymerization
- motor proteins use energy derived from ATP hydrolysis to move steadily along polarized filaments
- carry membrane-closed organelles
- motor domain (head) identifies track and direction
- tail region identifies cargo
- microtubule based motors
- kinesin
- most walk toward + end
- most carry binding sites for organelle or another microtubule
- role in spindle formation and chromosome separation
- dyneins
- - end directed microtubule motors
- cytoplasmic dyneins are important for vesicle trafficking and localization of golgi
- axonemal dyneins are sliding mirotubules that drive cilia and flagella beating